Paediatric Resp
Download
Report
Transcript Paediatric Resp
The Respiratory System
Paediatrics OSCE Revision
Elizabeth Evans
Plan
What could come up in the OSCE
Respiratory examination
Respiratory conditions
Key Points
Likely to be one examination from: CV/Resp/GI
May have a station with images or recordings
May have a history
Mostly normal children
Know a little about everything: Vivas are v. short
Easiest way to get marks: Be nice to the child!
What could come up?
Most respiratory disease in children presents acutely, thus
unlikely to appear in the OSCE.
OSCE:
History:
-Normal
-Asthma
-CF
-CF
-Hyperexpanded chest
from asthma
-Infection
Respiratory Examination
Inspection- increased work of breathing
Hands- cyanosis, clubbing (CF or CHD)
RR and HR – remember changes with age
Face- central cyanosis
Avoid trachea in children – indicate to examiner
Chest shape
Palpation- apex beat and chest expansion
Percussion- do not go straight for this on the chest!
Auscultate
Dyspnoea in a child
Nasal flaring
Subcostal and intercostal recession
Expiratory grunting
Difficulty feeding
Added sounds eg expiratory wheeze
CONDITIONS
Respiratory infections
URTI: common cold
LRTI: pneumonia
sore throat
bronchiolitis
otitis media
whooping cough
croup
diptheria
acute epiglottitis
Croup
Viral laryngotracheobronchitis (parainfluenza virus)
6 months – 6 years (peak in 2nd year)
URTI symptoms (coryza/fever) for 2 days before onset of a characteristic
barking cough and stridor (subglottic oedema) lasting around 3 days
Most improve spontaneously within 24h
1 in 10 require hospitalisation: under 12months, severe illness or signs of
respiratory failure
Single dose of dexamethasone or nebulised budesonide is beneficial
Nebulised adrenaline provides transient improvement (reduces oedema)
and is used in more severe cases to enable time for transfer to ITU
Acute epiglottitis
Life-threatening emergency
Haemophilus influenza type B (Hib immunisation has caused reduction)
1- 6 years
Rapid onset, intensely painful throat, febrile child, unable to swallow and
reluctant to speak. Sat upright with open mouth drooling saliva.
Resus room, call ENT and anaesthetist
Do not examine the throat!
Secure airway, then take bloods for culture and commence IV cefuroxime
Rifampicin for household contacts
Diptheria
Cornyebacteria diptheriae
Sore throat, fever, lymphadenopathy and stridor
Hallmark sign= thick grey material on back of throat
Potentially fatal, highly infectious
Eliminated by immunisation programme
Bronchiolitis
Commonest serious respiratory infection of infancy
1-9 months
Viral infection (mainly RSV)
Coryzal symptoms followed by dry cough with increasing SOB
Tachypnoea, hyperinflation, bilateral fine crackles and wheeze
CXR: chest hyperinflation
Supportive management to address hypoxia and maintain
hydration (1% require assisted ventilation)
Whooping cough
Bordetella pertussis (highly contagious)
Spread by droplet infection
Characteristic inspiratory whoop (may be absent in infants)
During paroxysms of coughing the child may go blue and vomit
May persist for 3 months
Culture organism from nasal swab
Erythromycin reduces infectivity but does not shorten duration of
symptoms
Asthma
Most common chronic respiratory disorder of childhood- 10%
Clinical diagnosis usually based on history and examination
If >5 able to demonstrate diurnal variation in PEFR
Wheeze= whistling noise made by the chest
History:
Examination
Nocturnal cough
Usually normal between attacks
Intermittent symptoms
In chronic severe asthma:
Triggers
-hyperexpansion
Exercise tolerance
Atopy
FH of asthma
Parental smoking
-pectus carinatum
-Harrison sulcus
Asthma Treatment
Aim- asymptomatic with no exacerbations
Educate child and parents on avoidance of triggers, importance of
regular therapy and correct inhaler technique
Stepwise approach: British Guidelines for Asthma Management:
Step 1: Inhaled SABA
Step 2: Low dose inhaled steroids (if requiring 3xday SABA)
Step 3: Add LABA or leukotriene antagonist
Step 4: Increase inhaled steroid. Oral Theophylline. Leukotriene
antagonist.
Step 5: Alternate day oral steroids.
Metered dose inhaler plus spacer:
useful in all children, highly effective as
do not rely on technique.
Metered dose inhaler: competent older children
Dry powder inhaler: 5 years +
Cystic Fibrosis
Autosomal recessive disease affecting 1 in 2500
Mutation in CFTR protein resulting in defective chloride channel,
increased viscosity of secretions in respiratory tract and pancreas
Consider in any child with recurrent chest infection or failure to thrive
Repeated infections lead to bronchial wall damage and abscesses
Deficiency of pancreatic enzymes results in malabsorption and
steatorrhoea
Diagnosis: screening performed as part of Guthrie test.
Gold standard diagnostic test is the sweat test. Genetic testing also
useful to confirm diagnosis.
CF Examination
General: small for age, Creon supplements or insulin around bed
Peripheral: finger clubbing
Airway: nasal polyps, hyperinflated chest, crackles
GI: scar from meconium ileus operation
CF Management
MDT approach
Respiratory: physio, aggressive treatment of lung infections
Nutritional: high calorie diet, vitamin ADEK, Creon (pancreatic
enzyme supplements)
Severe lung disease may be considered for lung
transplantation
Thank you