SUBSTANCE ABUSE IN HEALTHCARE PROFESSIONALS
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Transcript SUBSTANCE ABUSE IN HEALTHCARE PROFESSIONALS
Alcohol-Related Problems in
Special Populations:
S. Pirzada Sattar, MD
Creighton School Of Medicine
Department of Psychiatry
Copyright ALCOHOL MEDICAL SCHOLARS
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Substance Use Disorders in
Special Populations
Geriatric population
HIV+ , & Gay and lesbian groups
African Americans & other minorities
Immigrants
Psychiatric patients
Children & Youth
Women
Physicians
Incarceration
Homeless
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Athletes
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DEFINITIONS
Substance Use Disorders(SUDs)
DEPENDENCE
Tolerance
Withdrawal
Inability to cut down/control use
Considerable time spent using/obtaining/recovering
Important activities given up/reduced
Use despite negative consequences
ABUSE (less severe)
Failure to fulfill role obligations
Use in hazardous situations
Recurrent, related legal problems
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SUBSTANCE USE DISORDERS IN
GERIATRIC PATIENTS
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Substance Use Disorders (SUDs) in Geriatric
Patients Are Often Overlooked
Substance users stereotyped as young
Physicians miss substance use
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Geriatric Patients with SUDs are Often
Evaluated by Physicians
Frequent evaluation an opportunity to screen
Higher rates of SUDs in medical facilities
Substance use complicates medical illnesses
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Increased Substance Use Effects in Geriatric
Patients
Increased BAC because:
•
Decreased lean body mass
•
Decreased total body water
•
Decreased gastric alcohol
dehydrogenase
Alcohol and drugs more intoxicating in
geriatric patients
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Description of Alcohol Use Disorders in
Geriatric Patients: Prevalence
16% Men > 2 drinks per day, 15% Women >
1 drink per day
Up to 31% men, 21% women > 3 drinks daily
in retirement communities
Up to 21% alcohol dependence in medical
patients
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Alcohol Use Disorders (AUDs):
Early Onset (< Age 60)
About 2/3 of geriatric AUDs
Greater financial, legal and social problems
than later onset
Heavier drinkers than later onset patients
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AUDs: Late Onset ( > Age 60)
About 1/3 of geriatric AUDs
Aging social drinkers more intoxicated with
same dose
Cognitive disorder in heavy drinkers
Social drinkers who increase drinking after
losses
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I. Medical Complications of Alcohol in
Geriatric Patients
Cirrhosis: 60% 1 year death rate > age 60
vs. 7% in younger patients
Heart Effects
• Women more susceptible
• Alcoholic women 4 X coronary artery disease vs. non-alcoholic
women
• Atrial fibrillation common, “holiday heart” increases risk
• Increased stroke risk
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II. Medical Complications
Increase in cancers of liver, esophagus,
nasopharnx and colon
Thrombocytopenia, macrocytosis
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III. Medical Complications
Neurologic
• Increased dementia, Wernicke’s
encephalopathy, Korsakoff’s psychosis
Psychiatric
• Alcohol-induced mood disorder
• Pseudodementia from mood disorder
• Suicide
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Other SUDs
Less data than AUDs
Low prevalence of illicit drug use
• Few IV drug users survive
• Reduced access to illicit substances
High prevalence of prescription drug use disorders
• 25% using psychotropic medications
• This includes benzodiazepines and opioids
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Importance of Physician Screening
Medical complications
Doctors in an important position to intervene
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DSM-IV Criteria for Substance Dependence
Maladaptive pattern and 3 or more of the following in a 12 month
period:
Tolerance (often reduced in geriatric patients).
Withdrawal (often delayed, with mental status changes in geriatric
patients).
Greater amount of use or longer duration than expected.
Unsuccessful efforts to reduce use.
Large amount of time obtaining, using and recovering from use.
Important activities reduced or given up.
Continued substance use despite its aggravation of physical or
psychological problem.
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DSM-IV Criteria for Substance Abuse
•
•
•
•
Maladaptive use and 1 of the following in 12
month period:
Failure to fulfill obligations at work school or
home.
Recurrent use when physically hazardous.
Recurrent related legal problems.
Continued use despite recurrent social or legal
problems.
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State Markers that Suggest Alcoholism
•
•
•
Gamma-glutamyl transferase (GGT): Sensitivity of 70% to
80% if 6-8 drinks per day consumed
Mean corpuscular volume (MCV) greater than 90 cubic
microns consistent with alcohol dependence
Carbohydrate deficient transferrin (CDT): Social over 14
units/liter and alcohol dependence over 20-30 units/liter
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Questionnaires that Raise Suspicion of
Alcohol Abuse or Dependence
• MAST-G is unique in that it is specific to
geriatric alcohol use disorders.
• AUDIT is comprehensive.
• CAGE and TWEAK are quick but have limited
sensitivity and specificity.
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Assessment of Drinking
2
2
1
1
TWEAK:
- T (tolerance) “How many drinks can you hold?”
- W (worried) “Have close friends or relatives worried
or complained about your drinking in the past year?”
- E (eye opener) “Do you sometimes take a drink in
the morning when you first get up?”
- A (amnesia) “Has a friend or family member ever
told you about things you said or did while drinking
that you could not remember?”
- K (cut town) “Do you sometimes feel the need to
cut down on your drinking?”
1
___
3 risky drinking
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Screening for SUDs other than AUDs
Methods less developed than for AUDs
Signs for concern (not specific) include:
• doctor shopping
• drug-seeking behavior
• decreased motivation
• trouble sleeping
• poor self care
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Treatment of SUDs
Identification
Intervention
Detoxification
Rehabilitation
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Identification
Doctor’s office, clinic and hospital
extremely important sites for identification
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Intervention in Geriatric patients
• Involve adult family members.
• Denial by family and peers.
• Reduced mobility.
• Losses and social isolation.
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Brief Intervention
• Two to three 10-15 minute counseling
sessions
• Identify problem, consequences and
formulate treatment plan.
• Non-confrontational and supportive.
• Tailored to individual needs and goals.
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I. Alcohol Detoxification Concerns in
Geriatric Patients
Confusion (rather than tremor) early withdrawal sign
Duration of withdrawal/hallucinosis increased
Rule out DTs in confused elderly
Replace electrolytes and nutrients
Short acting benzodiazepines (lorazepam)
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II. Alcohol Detoxification Concerns in
Geriatric Patients
Severe withdrawal or medical illness
managed inpatient
Otherwise outpatient with family support
Monitor symptomatology with Clinical Institute
Withdrawal Assessment for Alcohol (CIWAs)
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General Overview of Alcohol
Detoxification
Supportive treatment
Benzodiazepine taper
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Opioid Detox
Supportive Treatment
Medication
• Clonidine
• Methadone taper
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I. Rehabilitation Strategies for Geriatric
Patients
Psychotherapy
• Individual for substance use and social
needs from losses and isolation
• Group, family and network therapy for
damage to family and peer relationships
from substance use.
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II. Rehabilitation Strategies for Geriatric
Patients
Optimized by age-specific treatment
• Must fill the time formerly spent using
substances
• Senior centers often have alcoholics
anonymous (AA) groups and support
socialization
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Pharmacotherapy in Rehabilitation:
A Limited Role
Naltrexone reduces alcohol reinforcing effects but
does not clearly promote abstinence, monitor liver
transaminases
Disulfiram problematic with potential drug interactions
and co-morbid medical conditions
Acamprosate may modestly increase abstinence
rates but GI upset, FDA approval pending
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Alcohol and Drug Misuse in
HIV+ and
Gay and Lesbian Populations
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Prevalence
Approximately 1 million people have
contracted HIV since 2001
Half have died
>40% African American
>20% Hispanics
24% of HIV+ were IV drug users
10% reported same sex practices
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Prevention requires:
Greater tolerance, assessment,
testing
Community resources & outreach
Drug abuse treatment
Harm reduction, e.g. Needle
exchange programs, methadone,
buprenorphine etc.
Medical and psychiatric treatment
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Alcoholics Anonymous
Most cities have gay/lesbian AA
meetings
Clients may need to be reminded that
AA is not specifically focused on religion
Extending the powerlessness over
alcohol concept to being powerless over
being gay may be helpful
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Recommendations
Screen IV drug users & gay and lesbian
clients for alcohol abuse and
dependence
HIV + may be referred to specific
treatment centers
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Substance Use Disorders
in
African Americans
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Goal of this section
Rare data
Policy/planning
Minorities and immigrants are different:
-traditional use of drugs in place
or culture of origin
-traumas suffered by Blacks and
other minorities
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Minorities discussed:
African American
Native American
Hispanics
Asian Americans
Pacific Islanders
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Long history
Marijuana and south Sahara
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Exposure in the New World
Patent medicine
Syringe development
Yet, use less common
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1940’s:
Heroin
Cough syrups
Prescription drugs
1960’s:
Marijuana
1990’s
Cocaine
2000’s
Methamphetamine
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Major proportion of soldiers are from
minorities
Easy access to alcohol, ? Drugs
Bring experiences home, e.g. PTSD
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Racial integration (hallucinogens)
Widespread cocaine use
Gangs/crack
-interdiction
-combinations with alcohol and heroin
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Alcohol
White
Black
Hispanic
Any type of drug
White
Black
Hispanic
20.4
14.7
19.9
9.2
8.6
9.2
35+
65.6
49.6
49.8
66.0
55.7
50.6
54.3
42.0
44.0
17.0
15.7
10.9
8.8
Type of drug
Age group
26-34
12-17 18-25
2.9
3.8
1.5
10.6
5.7
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Age group
Type of drug 12-17 18-25
26-34
35+
Marijuana
White
Black
Hispanic
7.3
7.3
6.9
14.4
13.9
8.3
6.6
9.2
3.6
1.9
3.3
1.0
Cocaine
White
Black
Hispanic
0.5
1.1
1.1
2.3
3.1
2.1
1.3
0.6
1.4
0.4
0.6
0.6
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Education:
Family:
-Schooling was denied to Blacks
-Current education
-?cohesiveness
-Home supervision
-Minority youth/single household
Religion:
Role of Religion for minorities (then & now)
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Better screening and diagnosis
Coordination schools/local
organizations
Culturally sensitive programs
Involvement of religion and clergy
education
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Summary
Discussion of historical and recent
patterns.
Areas of probable vulnerabilities, e.g.
“acculturation stress”.
Treatment options
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Substance Abuse
in Psychiatric
Patients
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Substance Abuse in
Psychiatric Patients
Prevalence
Commonly abused substances
Increases morbidity and mortality
Treatment is available and effective
Long term approach required
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Alcohol-Related Problems in
Special Populations:
YOUTH and WOMEN
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Drinking Rates in Youth
12th Graders
100%
75%
50%
25%
0%
Ever
Drank
Been
Drunk
>5
Drinks
Daily
Drinking
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Features of Drinking in Youth
Prognosis
Drink < age 14: risk of alcohol dependence
Typical experimentation sequence:
Cigarettes
or
Alcohol
Marijuana
Other drugs
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Assessing Drinking in Youth
Red flags:
Change in peers
Family problems
Grades drop
Mood swings
Common consequences:
Accidents
“3-Ds”: driving, dating, & drug related problems
Uncommon problems:
Severe withdrawal symptoms
Liver problems
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Assessing Drinking in Youth
2
2
1
1
TWEAK:
- T (tolerance) “How many drinks can you hold?”
- W (worried) “Have close friends or relatives worried
or complained about your drinking in the past year?”
- E (eye opener) “Do you sometimes take a drink in
the morning when you first get up?”
- A (amnesia) “Has a friend or family member ever
told you about things you said or did while drinking
that you could not remember?”
- K (cut town) “Do you sometimes feel the need to
cut down on your drinking?”
1
___
3 risky drinking
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Youth: Treatment
Formats
Multi-systemic treatments
Minnesota model
Therapeutic communities
Cognitive-behavioral
Groups
Brief intervention
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Youth: Treatment
Pathways to success
1.
Traditional routes
2.
Early individuation
3.
Family involvement
Outcome
Treatment helps
Inpatient = Outpatient
50-80% relapse in 1st year after treatment
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Women: Epidemiology
Women
Any alcohol in past year
62%
Men
83%
Has met Alcohol Abuse criteria
6% 13%
Has been Alcohol Dependent
8% 20%
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Women & Heavy Drinking
Risk of health problems:
Alcohol-related liver disease,
hepatitis, infections, anemia
STDs, UTIs, reproductive organ
problems, breast cancer
Violence victimization risk
Neurocognitive effects
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Alcohol & Pregnancy
Prevalence
16-35% of pregnant women drink
4% drink frequently or heavily
Fetal alcohol syndrome (FAS)
1.
Facial dysmorphia
2.
Growth deficiency
3.
CNS deficits
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Women: Detection
Wide variability
Social risk factors:
Low vocational skills and education
Substance use disordered partner
Social isolation
Screening tools:
TWEAK
AUDIT-C
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Women: Treatment Issues
Barriers
External
Internal
Optimal treatment for women
Reduce barriers
Combine with obstetrics or other healthcare
Women-only v. mixed setting
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SUBSTANCE USE
DISORDERS IN
PHYSICIANS
S. Pirzada Sattar, MD
School of Medicine
“WHY SHOULD I
STAY AWAKE?”
It might be my colleague
It might be my patient
It might be me
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KEY POINTS
SUDs similar to the general population
Benzodiazepines and opioids higher
Identification is often difficult and delayed
Treatment outcomes are often better
Impaired Physician Programs are helpful
67
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EPIDEMIOLOGY
VERY VARIABLE!!!!
Population studied
Methods used
Terminology
Diagnostic criteria
Changes over time?
Concern about anonymity
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EPIDEMIOLOGY
General
Similar rates of SUDs to general population
8-14%
Less SUDs compared to other occupations
Roofers, painters
Increased rates of use & SUDs with:
Benzodiazepines
Prescription opioids
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EPIDEMIOLOGY
Medical Students
Use begins prior to medical school
Types of drugs same as general pop.
Alcohol use & dependence variable
Drug use and dependence less
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EPIDEMIOLOGY
Residents
Rates of dependence:10-14%
Alcohol & illicit drug use begins prior
Benzo & opioid use begins during
Self-treatment
Self-prescribed
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EPIDEMIOLOGY
Practicing Physicians
Prevalence of dependence: 8-14%
Still means 60-75,000 affected M.D.s in U.S.!!!!
Use & misuse of prescription opioids
& benzodiazepines up to 5Xs higher
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EPIDEMIOLOGY
By Specialty
HIGHEST
Emergency Medicine
Psychiatry
LOWEST
OB-GYN
Pathology
Radiology
Pediatrics
Anesthesiology
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REASONS FOR USE
Recreational
Seen more in medical students
Performance Enhancement
Seen more in Emergency Medicine
Self-medication (pain, anxiety, “stress”)
Seen more in residents & attendings
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PROGRESSION
Family
Community
Finances
Spiritual/emotional
Physical health
Job performance
Often one of the last things affected
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CONTRIBUTING
FACTORS
Family History
Personality characteristics
Health/lifestyle
Stress???
Availability???
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IDENTIFICATION
Urine drug screening
Employment/school application
Physician screening
Impaired Physicians Programs
Reporting
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“WARNING SIGNS”
Isolation
Friction with colleagues
Disorganization
Inaccessibility
Frequent absences
Rounding on patients at odd hours
Inappropriate or forgotten orders
Slurred speech during off-hours calls
Prescriptions for family members
OD or suicide attempt
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WHY THE DELAY
IN DETECTION?
Independence
“Malignant denial”
“I can take care of myself”
“Knowledge is protective”
Fear of consequences
“Conspiracy of silence”
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“CONSPIRACY OF
SILENCE”
Reputation
Financial
Fear & intimidation
Professional pride
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REPORTING
Ethical obligation
Disabled Doctors Act
Federal law
Requirements vary by state
Protection from law suit varies
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TREATMENT
THE GOOD NEWS!!!
Variable data
Most show better outcomes
70-90% “success rate”
• little correlation with substance
• little correlation with specialty
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TREATMENT
Goals
Abstinence
Acceptance of chronic disease concept
Identification of triggers
Development of non-chemical coping
skills
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TREATMENT
Key Factors For Success
Duration of aftercare
Physician’s Health Program involvement
Family involvement
12-Step involvement
Witnessed urinalysis
Contingency contract
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TREATMENT
Stumbling Blocks
Uniqueness
Role-reversal
Over-identification w/ performance
Identification (by treatment provider)
Medical knowledge
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TREATMENT
Physician-specific
In-Patient
Talbott, Farley
12-Step
“Caduceus meetings”
Pros & Cons
Combined approaches
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“RE-ENTRY”
Most return to practicing medicine
Change to a less high-risk specialty
Imposed prescribing restrictions
Altered work schedule
Specialization in addictions
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“PREVENTION”
Medical school policies
Medical school education
State Impaired Physicians Programs
• Protect the public
• Provide “rehabilitation” (vs punishment)
JCAHO-mandated hospital programs
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KEY POINTS
A Review
SUDs similar to the general population
Benzodiazepines and opioids higher
Identification is often difficult and delayed
Treatment outcomes are often better
Physician Rehab Programs are our friends
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WHERE TO GET HELP
State Agency
#
School Resources
#
Your email address
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NURSES
Rates similar to general population
Higher use of benzodiazepines & opioids
more parenteral use
Higher in emergency room & critical care
Especially difficult to monitor
Watch for diversion
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DENTISTS
Less good data
More use of inhaled anesthetics
Possibly higher opioid use and SUDs
Related to higher suicide rate?
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PHARMACISTS
Estimates of dependence: 10-18%
Less parenteral use
@ 50% have used CS w/o script
@ 20% on regular basis
primarily self-medication
@ 60% of students have used CS w/o script
@ 40% on regular basis
primarily recreational
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VETERINARIANS
Little good data
More Ketamine use
Other higher-potency opioids
Inhaled anesthetics
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Substance Use In Athletes
S. Pirzada Sattar, MD
Developed for the Alcohol Medical Scholars Program
Copyright ALCOHOL MEDICAL SCHOLARS
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INTRODUCTION
Major problems facing sport today
Growing attention
Deaths of elite athletes
Increasing attention of media
Contrary to the ethical principles of
athletic competition
Wide spread among athletes
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DRUGS MISUSED BY ATHLETES
• Therapeutic drugs
OTCs, diuretics, opioids, beta-blockers,
etc.
Performance enhancing drugs
Amphetamines, ephedrine, caffeine,
anabolic steroids, growth hormone, etc.
Drugs typically misused
Alcohol, nicotine, marihuana, cocaine, etc.
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GOALS
Historical perspective
Factors influencing athletes to use
drugs
Types of drugs athletes useconsequences and myths
Preventing and treating drug use in
athletes
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Historical perspective
Ancient civilizations
Mushrooms, herbs, liquor
19th Century
Alcohol, caffeine, nitroglycerine, opium,
strychnine, trimethyl
World War II
Amphetamines, testosterone
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Historical perspective
Post war era
Amphetamines continue
Anabolic steroids
Newer agents
Blood doping
Erythropoietin
Growth hormone
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Currently prohibited by IOC
Drugs
Stimulants, opioids, anabolic agents, diuretics,
peptide hormones
Methods
Blood doping, artificial oxygen administration,
plasma expanders, pharmacological, chemical
and physical manipulation
In certain circumstances
Alcohol, cannabinoids, local anesthetics,
blockers
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What factors influences athletes?
Belief that competitors take drugs
Determination to do anything to win
Pressures from coaches, parents, peers
Community attitudes and expectations
Financial rewards
Media influence
Belief of enhanced performance
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THERAPEUTIC DRUGS
OTCs
NSAIDs, laxatives, ephedrine, analgesics,
weight loss meds, corticosteroids, local
anesthetics
Low potential for misuse
Increased risk of further injury, GI bleed,
anemia, eating disorders
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THERAPEUTIC DRUGS
Diuretics
Rapid weight loss
Boxing, wrestling, judo
Excretion or dilution of illegal substances
Overall negative impact on performance
Dehydration, hypotension, muscle cramps,
electrolyte imbalance
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THERAPEUTIC DRUGS
Opioids
Prescription pain killers most common
Allow performance while injured
75% used after injury only
Increased risk of further injury,
dependence, drowsiness, mental clouding;
in high doses: respiratory depression,
hypotension
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THERAPEUTIC DRUGS
Beta-Blockers
Anti-tremor, anxiolytic effect
Shooters, ski jumpers, archery
Negative effect on endurance
Depression, bronchospasm, fatigue
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PERFORMANCE ENHANCING DRUGS
CNS Stimulants
Amphetamines
• Delay fatigue, increase alertness,
enhance speed, power, endurance,
concentration
Hypertension, angina, vomiting, abdominal
pain, cerebral hemorrhage, dependence,
death
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PERFORMANCE ENHANCING DRUGS
CNS Stimulants
Caffeine
• Shortened reaction time, improved
concentration, diuresis
• Glycogen sparing leading to delayed fatigue
• > 12 ug/mL is a positive urine per IOC
Dyspepsia, cardiac damage, combination with
other stimulants (e.g. ephedrine) may be fatal
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PERFORMANCE ENHANCING DRUGS
Systemic stimulants
Adrenalin
• In local anesthetics
Ephedrine and pseudoephedrine
• Cold and allergy remedies
Phenylpropanolamine
• Diet pills
Similar effects to the amphetamines in high doses
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PERFORMANCE ENHANCING DRUGS
Anabolic androgenic steroids
Derivatives of testosterone
First use generally later than other drugs
Drug and method sought for maximum anabolic
and minimum androgenic properties
Sprinting, weight lifting, body building
Acne, abnormal LFTs, feminization, virilization,
premature closure of the epiphysial plates,
behavioral changes “roid rage”, CVAs,
cardiomyopathy
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PERFORMANCE ENHANCING DRUGS
Beta 2 agonists
Isoproterenol, epinephrine, norepinephrine
Sympathomimetic amines, anabolic properties
Cardiac arrhythmias in overdose, headaches
Peptide hormones: HCG
Increases testosterone
Maintains testicular volume with anabolic steroid
use
Ovarian cysts
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PERFORMANCE ENHANCING DRUGS
Pituitary and synthetic gonadotropins
Increases testosterone, anti- estrogenic
Ovarian cysts
Corticotropins
Increase testosterone
Rare and related to excess corticosteroidspituitary suppression, ¯ immunity,
osteoporosis, hyperglycemia
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PERFORMANCE ENHANCING DRUGS
Growth hormone
Increase muscle mass & decrease fat
mass
Gigantism, acromegaly, hypothyroidism,
cardiac disease, myopathies, arthritis,
diabetes mellitus, impotence, osteoporosis
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PERFORMANCE ENHANCING DRUGS
Erythropoietin (EPO)
Stimulates RBC production
Increases oxygen carrying capacity
CVAs
Blood doping
RBC transfusion, artificial oxygen carriers
Increases oxygen carrying capacity
Allergic reactions, sludging of blood
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FOOD SUPPLEMENTS
Viewed as legal means of gaining edge
76-100% of athletes use vs. 50%
general population
May or may not contribute to enhanced
performance
Creatine, colostrum, antioxidants, sodium
bicarbonate, vitamins, proteins, amino
acids
Adverse effects not investigated
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TYPICAL DRUGS OF MISUSE
Most common: marijuana, cocaine, alcohol
Generally have negative effect on performance
Substance misuse same in college athletes vs. nonathletes
Decrease in use of marijuana, amphetamines and
cocaine, but increase in smokeless tobacco use,
1985-1996
Most drugs first used in junior or senior high school
(for recreation not performance)
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TYPICAL DRUGS OF MISUSE
Alcohol
Most frequently used
Negative impact on reaction time, handeye coordination, balance, strength
Excessive heat production and dehydration
Cardiovascular and GI complications,
nutritional deficiencies, dependence
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TYPICAL DRUGS OF MISUSE
Cocaine
Minimal performance enhancing effect
Heightened arousal and increased
alertness with low doses
Over confidence leading to increased
risk of injury
MI, CVA, seizures, arrhythmias,
dependence
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TYPICAL DRUGS OF MISUSE
Cannabinoids
Most frequent illegal drug used in the US
Male athletes have higher incidence than nonathletic peers (opposite for females)
Initial use in high school
Psychomotor impairment, distorted perception,
amotivational syndrome; decreased
testosterone with long-term use
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TYPICAL DRUGS OF MISUSE
Nicotine
Majority use in form of smokeless tobacco
Males >> females
52% of baseball players, 26% of varsity football
players used smokeless tobacco (early 1990s California
college survey)
Highest risk for baseball players
Cardiovascular and pulmonary disease, oral cancers,
dependence
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PREVENTION AND TREATMENT
Drug testing
Commonplace in amateur and professional
sports
65% of college athletes agree with testing
37% agreed that positive should result in
disqualification
67% of college athletes believe that drug
testing deters drug use
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DRUG PROGRAMS
Administered by leagues and
associations (NCAA, NFL, NBA)
Responsible for relevant events, fairness,
quality of competition, safety, image of their
athletes and events
Deter use by testing and discipline
Some include evaluation and treatment
Coaches can discourage use
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DRUG PROGRAMS
Identify individuals with drug problem to
facilitate treatment
Keys to successful drug program:
Inclusion of all involved parties
Reliable and sensitive testing program
Consistent discipline
Evaluation of effectiveness
Confidentiality
Early prevention
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CHALLENGES
Most drugs not prescribed
Viewed as essential for success
Easy access to drugs
Physician dilemma/role
Monitoring side effects
Why?, discuss pro/cons, appraisal, explore
options
Need for collaboration
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SUMMARY
Substance use in athletes dates to ancient
times
Multiple factors why athletes use drugs
Types of drugs used range from therapeutic
and performance enhancing to typical drugs
of misuse
Programs are in place to address drug use in
athletes
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