Care of the Pediatric Patient with Cognition or Perception Problems
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Transcript Care of the Pediatric Patient with Cognition or Perception Problems
CARE OF THE PEDIATRIC
PATIENT WITH COGNITION OR
PERCEPTION PROBLEMS
Elizabeth Allen RN, MSN
LEARNING OBJECTIVES
Describe the neurologic anatomic and physiologic
differences between infants and children and
adults
Identify aspects of the pediatric neurologic
assessment including Glasgow Coma Scale and
signs of meningitis
Identify and create a treatment plan for pediatric
neurologic infectious diseases
Compare pediatric traumatic brain injuries for
different age groups
Describe pediatric pain management
ANATOMIC DIFFERENCES
Brain & spinal cord
Cranial Nerves
Head to toe
Cranial bones
Present, must mature
Myelination
Intact from birth
Reflexes
Large head
Brain growth
Not complete
Head vs. chest size
Blood brain barrier
NEUROLOGICAL DEVELOPMENT
Neurological system of the infant is incompletely
developed.
First year the neurons become myelinated.
Myelinization occurs in a cephalocaudal and
proximodistal manner.
NEUROLOGIC ASSESSMENT
Cerebellar
Noting
Functioning
milestones
Fine and gross motor skill
Behavioral
assessment
Waking activity
Responses to environment
Social interaction
Cranial
nerves
Motor system
Head control
Reflexes
Primitive reflexes
Stepping
Tonic-Neck
Moro/Startle
Sucking
NEUROLOGIC ASSESSMENT
Brudzinski’s sign
Kernig’s sign
Nuchal rigidity - neck
Photophobia
ICP
LOC
Glasgow Coma Scale
Glasgow Coma
Scale
Including Pediatric
Assessment
INCREASED INTRACRANIAL PRESSURE
Increased ICP creates
decreased CPP
(Cerebral Perfusion
Pressure)
Causes
Head injury
Bleeding
Space occupying lesion
Infection
Hydrocephalus
Infant Signs & Symptoms
Fontanels (bulging)
Sutures (widened)
Scalp veins (dilated)
Sunset eyes
Older child Signs &
Symptoms
Normal ICP < 20 mmHg
Headache
Nausea, vomiting
Decreased LOC, irritability
Pupil changes
Sunset eyes
Posturing
Cushing’s Triad (Late sign)
AMBLYOPIA
Amblyopia: Reduced
vision in 1 or both
eyes
From visual
deprivation of eye
Most often from
strabismus
Visual assessment
and correction by age
6 years
Strabismus
NEUROLOGIC INFECTIOUS DISEASES
Bacterial Meningitis
Infant Signs &
Symptoms
Poor
feeding/suck
Vomiting
High-pitched cry
Bulging fontanel
Fever or hypothermia
Poor muscle tone
Bacterial Meningitis
Child & Adolescent Signs
& Symptoms
Abrupt onset
Fever & chills
HA
Nuchal rigidity
Vomiting
Irritability
Seizures
Photophobia/alterations
in sensorium
BACTERIAL MENINGITIS
Nursing
Interventions
Droplet Isolation!
Maintain patent airway, ventilation support prn
Post-LP care
Assessment
Increased ICP
Nutrition
Pain control
Monitor IV antibiotic therapy
Monitor for complications- seizures, immobility
VIRAL MENINGITIS (ASEPTIC MENINGITIS)
Infant and Toddler
Signs & Symptoms
Irritability
Lethargy
Vomiting
Change in appetite
Children & Adolescent
Signs & Symptoms
Preceded by nonspecific
febrile illness
HA
Malaise
Muscle aches
N&V
Photophobia
Nuchal/spinal rigidity
VIRAL MENINGITIS
Treated
symptomatically
Hospitalized?
Decease stimulation
Hydration
Comfort measures
NEUROLOGIC INFECTIOUS DISEASES
Reye
Syndrome
Usually develops after mild viral illness
Strongly associated with Aspirin (salicylates) use
in viral infections
Acute encephalopathy and fatty infiltration of the
liver and other organs
Poor liver function
Hypoglycemia
Prolonged Prothrombin time
Developmental and neurologic deficits may occur
REYE SYNDROME
Stages
Progress with decreasing LOC
1.
Vomiting and drowsiness, listlessness
2.
Personality changes: irritable, aggressive
3.
Disorientation: confusion, irrational, combative
4.
Delirium, seizures, coma, loss of deep tendon
reflexes, respiratory arrest
Symptoms of Reye Syndrome in infants do not
follow a typical pattern
Symptoms appear most commonly after a viral
illness
(National Reye Syndrome Foundation, 2013)
REYE SYNDROME
Signs
& Symptoms
Abrupt change in LOC
Vomiting
Liver enzymes, ammonia levels elevated
Blood sugars low
PT prolonged
Bilirubin remains normal
Liver biopsy shows small fat deposits
REYE SYNDROME
Nursing
Interventions
Monitor
ICP
Fluid restrictions
Neurological
assessments
Monitor lab values
Ammonia
Glucose
Blood gases
Provide
emotional
support
Medications
Vitamin
K
Phenytoin
Corticosteroids
Family
education
ACCIDENTS & HEAD INJURIES
Shaken baby syndrome
Head trauma
Falls
Child abuse
MVA
Near-Drowning
SHAKEN BABY SYNDROME
Pathophysiology – central nervous system injury
from repeated coup and contra-coup injury
Symptoms
blindness or eye damage
delay in normal development
seizures
damage to the spinal cord
Retinal hemorrhage
paralysis
brain damage
death
SHAKEN BABY SYNDROME
Maintain cardiopulmonary function
Prevent complications- Secondary Brain Injury
Promote recovery
Maximize developmental function
Provide emotional support
Role of Social Work
Discharge planning and home care teaching
Resources
HEAD INJURY
Mild to Severe
Highest rate of death in adolescents 15-19 years,
then <5 years
Side effects of trauma = cerebral edema &
increased ICP
Major cause falls
Child abuse, shaken baby syndrome <1yr.
Other causes
MVA
Bicycle, skateboard, snowboard, skiing
Alcohol or drug-related MVAs, sports injuries in
teens
HEAD INJURY
Primary Brain Injury
Develop
at time
of trauma
Direct blow
Inc. ICP
Apnea
Loss of
consciousness
Secondary Brain Injury
Results
as
response to injury
Few hours to
weeks post injury
Inc. ICP can
result in
irreversible brain
damage by
decreased CPP
(cerebral perfusion
pressure)
HEAD INJURY
Nursing
Interventions
Assessment
Seizure precautions
Decrease environment stimulus
Coordinate rehabilitation
Coordinate resource services
Administer meds as ordered
Corticosteroids
Seizure medications
Fosphenytoin
Phenobarbital
Child and Family Education
OTHER HEAD INJURIES
Skull fractures
Contusion
Concussion
http://www.cdc.gov/headsup/basics/index.html
Subdural hematoma
Epidural hematoma
SEIZURE DISORDER
Most are idiopathic
Genetic factors- associated with syndromes
Head injury
Stroke/cerebrovascular disorders
Metabolic disturbances
Electrolyte
Hypoglycemia
Renal failure
Hepatic failure
Hypoxia
SEIZURE DISORDER
Chronic disorder characterized by recurrent
seizures, result of underlying brain abnormality
Intractable seizures
Can suffer from poor self-esteem, academic
failure, poor social relationships
Anti-epileptic Drugs
For some surgery may be an option to control
seizures
Prolonged, uncontrolled seizures often result in
developmental delays, neurological damage
SEIZURE DISORDER
Priority Nursing
Diagnoses
Risk for aspiration
Risk for injury r/t type
of seizure and possible
loss of consciousness
Altered family
processes r/t having
child with chronic
illness
Nursing Interventions
Keep calm
A = Airway
Safety
Seizure Medications
Benzodiazepine (lorazepam,
intranasal midazolam)
Phenobarbital
Tegretol
Assessment
Provide emotional support
for patient/family
Resource information and
F/U
CONGENITAL NEUROLOGIC PROBLEMS
Neural tube defects
Spina bifida occulta
Meningocele
Myelomeningocele
Hydrocephalus
Cerebral Palsy
SPINA BIFIDA
Congenital neural tube defect affects head and
spinal column
Approximately 1500 births in US per year
Higher the defect the greater the neurologic
dysfunction
Cause unknown (possibly chemicals, medications,
maternal low folic acid levels, genetic)
SPINA BIFIDA
Pre Op Nursing Care
Sac care
Position
Assess motor function
Assess bowel and
bladder function
Nutrition/Hydration
Post Op Nursing Care
Monitor VS
Assess
Infection
CSF Leak
ICP
Positions
Pain management
Education
SPINA BIFIDA
Associated
Musculoskeletal
Talipes (clubfoot), dislocated hip, scoliosis, kyphosis
neurogenic bladder, hydronephrosis, renal damage, UTI,
incontinence
Genitourinary
Gastrointestinal
Hydrocephalus
cognitive deficit, visual perceptual problems, sensory
dysfunction, paralysis, muscle weakness, feeding
difficulties, swallowing problems, sleep apnea
Integumentary
constipation, impaction, incontinence
Neurologic
Problems
Skin breakdown related to immobility, incontinence
Psychosocial
Latex sensitivity/ allergy
SPINA BIFIDA
Nursing
Interventions
Assistance with bowel and bladder function
Promote Mobility
Maintain skin integrity
Positioning & turning
Provide emotional support
Resources
Child and Family Education
HYDROCEPHALUS
Imbalance between production and absorption of
CSF
Most common congenital defect (50%)
Etiology
Congenital
Acquired from meningitis, trauma, hemorrhage in
premature infant
Idiopathic (50%)
If untreated can cause permanent brain damage
related to increased ICP
HYDROCEPHALUS
Ventriculoperitoneal
Shunt
HYDROCEPHALUS
Signs & Symptoms
All Children
Vomiting
Lethargy
Cheyne-Stokes
respiratory pattern
Infant
Inc. head circumference
Split cranial sutures
High-pitched cry
Bulging fontanels
Irritability when awake
Seizures
Signs & Symptoms
Toddlers & Older
Children
Setting-sun eyes
Seizures
Irritability
Papilledema
Decreased LOC
Inc. B/P
HA
Difficulty with balance
& coordination
HYDROCEPHALUS
Priority Nursing
Diagnoses
Altered tissue
perfusion (cerebral)
High risk for infection
Risk for impaired skin
integrity r/t large size
of head and inability
to move
Nursing Care
Ventriculoperitoneal
Shunt
Provide post-op care
Assess
Infection
ICP
HOB?
Antibiotic therapy
Provide emotional
support
Child and Family
Education
CEREBRAL PALSY
Non-progressive motor and posture dysfunction
secondary to anoxic damage to motor centers in
fetal or infant brain( up to 2 yrs.)
70% prenatal (fetal), 20% perinatal, 10% <2 years
2- 2.5:1000
Abnormal muscle tone and lack of coordination
4 types: spastic, dyskinetic, ataxic & mixed
Symptoms depend on area of brain involved
CEREBRAL PALSY
All infants that show developmental delays,
feeding difficulties, abnormal muscle tone should
be evaluated
Turn infant’s head to one side, persistent asymmetric
tonic neck reflex beyond 6 mo. Indicates pathologic
condition, if any primitive reflexes persist should
suspect CP
Other complications:
Intellectual disabilities, vision impairment, hearing
loss, delays in speech and language, seizures
CEREBRAL PALSY
Priority Nursing
Diagnoses
Impaired physical
mobility
Self-care deficit
Impaired verbal
communication
Altered nutrition: less
than body requirements
High risk for injury r/t
neuromuscular,
perceptual, or cognitive
impairments
Fatigue
Nursing Interventions
Provide adequate
nutrition
Maintain skin integrity
Promote physical
mobility
Developmental
progression
Safety
Emotional support
Team effort
Child and Family
Education
DEVELOPMENTAL DEFICITS
Pervasive
Developmental Disorders
Autistic spectrum disorders
Asperger’s syndrome, Rett’s disorder, childhood
disintegrative disorder
Approximately 1% of children, 1 in 100-150
Neurodevelopmental Disorder
Screening at well checks
Early intervention
PERVASIVE DEVELOPMENTAL DISORDERS
Priority
Nursing Diagnoses
Communication, verbal, impaired
Social interaction, impaired
Injury, risk for
Caregiver role strain, risk for
Coping, family, compromised
COGNITIVE DISORDERS
Learning
Disabilities
Alteration in information reception and processing
Not low IQ
Mental
Retardation
Intellectual functioning and adaptive behavior
Congenital developmental disability
Low IQ
Trisomy 21 (Down Syndrome)
COGNITIVE DEFICITS
Expected Outcomes:
Learning Disabilities
Will compensate through new strategies
Mental Retardation
Reach highest level of independence
Encourage optimal family use of resources
COGNITIVE DEFICITS
Nursing Care:
Language, Motor Delays = Risk for Learning
Disabilities
Refer for testing
Early Developmental Testing
Identify cognitive delays
Support Group Referral
Help Families
Set goals: life and learning skills
Promote self-esteem
Partner with Families
Plan health interventions together
PEDIATRIC PAIN MANAGEMENT
Non-pharmacological
Distraction
Play
Position
Heat/cold
Guided Imagery
Sucrose
Pharmacological
Non-narcotic
Acetaminophen
Ibuprofen (>6 months)
NO aspirin
Narcotic
Morphine
Fentanyl
Acetaminophen with
codeine