Newborn Assessment and Care (chapter twelve) power
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Transcript Newborn Assessment and Care (chapter twelve) power
The Normal Newborn
ASSESSMENT AND CARE
Three transition phases
Phase One: the first hour
Phase Two: from one to three hours
Phase Three: from two to 12 hours
Priorities in first hour
Cardiovascular assessment and support
Thermoregulation
Assessment and support of blood glucose
Identification
Observing urinary/meconium passage
Observing for major anomalies and for apparent
gestational age concerns
APGAR ASSESSMENT
One and five minutes
Meant to identify the need for neonatal resucitation
APGAR SCORE
APGAR SCORE
Criteria
0
1
2
Color
Blue or pale
Acro-cyanotic
CompletelyPink
Heart Rate
Absent
Slow
(< 100/min)
>100/min
Reflex irritability
No response
Grimace
Cough, sneeze,
cry
Muscle tone
Limp
Some flexion
Active motion
Respirations
Absent
Slow, irregular
Good, crying
Additional signs of respiratory distress
Persistant cyanosis
Grunting respirations
Flaring of the nostrils
Retractions
Respiratory rate >60
Heart rate >160 or <110
Maintaining thermoregulation
Referred to as maintaining a neutral thermal
environment
Heat loss is minimal
Oxygen consumption needs are at their lowest
Hypothermia can cause
Hypoglycemia
Increased oxygen needs
Four mechanisms of heat loss
and corresponding interventions
Evaporation
Dry infant immediately
Conduction
Place on mothers body skin to skin
Convection
Cover with a blanket, wear a cap
Radiation
Keep away from cold windows and cold objects
Mechanisms of heat loss
Vital Sign Normals
97.7-98.6 F (36.5-37 C)
110-160
A soundly sleeping baby can go to 80 bpm
A crying baby may be as high as 180
30-60
Voids and Stools
Document from the moment of birth
Urination sometimes missed in early minutes
Generally expect both within the first 24 hours
One really wet diaper per day of age until milk is
fully in.
Observation for Gestational Age
Thorough assessment with Ballard Scale done later
A quick assessment is done in the delivery room
This enables infants earlier admission to the nursery
and anticipatory intervention to the problems of pre
and post term infants
Quick Assessment of Gestational Age
Skin
Vernix
Hair
Ears
Breast tissue
Genitalia
Sole Creases
Resting Posture
Cracked Skin
Abundant Lanugo
Ear of a preterm infant
Areola and increased lanugo
Sole creases
Female genitalia, very preterm
Preterm and Term Genitalia
Male Genitalia
Comparison of resting posture
Preterm and Term Male Genitalia
Hypoglycemia
Criteria vary from source to source
LPN book says <40
RN book says <36 but a threapuetic objective of 45
mg/dl or greater
The brain is dependent on a steady supply of glucose
for its metabolism
Infants at Increased Risk for Hypoglycemia
Preterm/postterm
Infants of diabetic mothers
Large for gestational age
Small for gestational age
Infants with Intrauterine growth retardation
Asphyxiated infants
Infants who are cold stressed
Infants whose Moms took ritodrine or tgerbutaline
to stop preterm labor
Symptoms of Hypoglycemia
Jitteriness
Poor suck
Poor muscle tone
Feeding difficulties
Sweating
High pitched cry
Respiratory difficulty
Weak cry
Apnea
Lethargy
Low temperature
Seizures
Hypoglycemia protocol
Low risk infants have a serum glucose drawn only if
symptomatic
High risk infants will have one per a hospital
protocol
Protocol typically at birth and q 1 hour x 3
Routine Medications
Erythromycin Eye
Ointment
Aquamephyton
(vitamin K)
First Hepatitis B
vaccine
HBIG if Mother is
Hep B surface
antigen positive
Physical Characteristics
DURING PHASES TWO AND THREE
Nervous System: Reflexes
Head lag
Rooting reflex
Moro reflex
Suck
Rooting
Hand and foot grasp
Tonic Neck reflex
Babinski
Dancing reflex
Trunk incurvation
Magnet reflex
Observe for symmetry
Head Lag
Moro Reflex
Tonic Neck Reflex
Dancing Reflex
Suck Reflex
Hand Grasp
Foot Grasp
Head
Head circumference
Molding
Caput succedaneum
Cephalohematoma
Fontanelles
Anterior closes between 12-18 months
Posterior closes by the end of the 2nd month
Molding
Cehpalhematoma
Caput Succedaneum and Cephalhematoma
Eyes
Eye placement
Epicanthal folds
Blink reflex
Discharge
Pupil reaction
Follows to midline
Hearing
Check overall response to sudden sound
Moro reflex
Check for placement of ears
Low set ears may indicate a congenital anomaly
Most infants receive hearing screening within the
first week of life
Respiratory and Cardiovascular
Ongoing assessment of cardio respiratory status that
has occurred since birth
More thorough heart assessment
Murmur may be present until fetal openings have
completely closed however they must be carefully
verified by pediatrician
Femoral and brachial pulses
Abdominal breathing; nose breathers
Femoral Pulses
Brachial Pulses
Assessment of Respiratory Status
Musculoskeletal
Symmetry!!
Five finger and five toes!!!
Clavicles
Movement of arms
Hips for developmental hip dysplasia
Lower legs/feet for “club foot”
Back: curvatures, cysts or dimples
Hip Check
Hip Check Skin Folds
GenitoUrinary
Male or female
Male
Testes descended
Proper placement of meatus
Female
Teach parents about pseudomenstruation
Always watch for and record voids!!!
Gastrointestinal
Passage of meconium
Placement and patency of anus
Abdomen should be soft and non tender
Round but not distended
Bowel sounds are present after first hour of birth
Umbilical cord inspection
Skin, many normal findings
Acrocyanosis
Desquamation
Epstein’s Pearls
Erythema toxicum
Harlequin Color
Milia
Mongolian Spots
Port Wine Stains *
The Normal Newborn
CARE MEASURES FOR THE
NORMAL NEWBORN PLUS A
LITTLE MORE.
Jaundice
Yellow coloring of an infants skin
Common and is caused by the natural breakdown of
RBCs in the infant after birth
Is never considered normal in the first 24 hours.
Physiologic Jaundice
Most jaundice in newborns is physiologic
It peaks between 48-72 hours
Usually disappears within a week
Usually benign
Can become elevated to a point of concern for the
baby
Significance of Jaundice
Bilirubin is toxic to the brain.
Bilirubin is prevented from entering the brain by
blood brain barrier under normal circumstances.
However the blood brain barrier isn’t well developed
in the newborn. Unconjugated bilirubin (lipid
soluble) could cross to the newborn and would cause
encephalopathy. (Kernicterus)
Physiologic Jaundice
Infants have extra RBCs due to fetal life
They need to be broken down by the body
Bilirubin is a component of the degradation of the
RBCs.
The liver is immature and does not conjugate and get
rid of the bilirubin fast enough.
More data on Physiologic Jaundice
RBC/Hgb level is higher than required
Neonatal RBC: 4.8-7.1 Infant: 4.2-5.2
Neonatal Hbg 14-24 Infant 11-17
Cells containing fetal hemoglobin have a shorter life
span
Bilirubin Nomogram
Phototherapy Nomogram
Other factors that will exacerbate physiologic
jaundice
Drugs
Hypoglycemia
Bruises
Hypothermia
Caput
Poor feeding
Cephalohematoma
Delayed passage
Fetal hypoxia
meconium
Trisomy 21
Polycythemia
Care to prevent hyperbilirubinemia
Early feeding
Frequent feeding
Neutral thermal environment
Prevention of hypoglycemia
Prevention of hypoxia
Causes of Pathologic Jaundice
Excessive hemolysis
Rh incompatibility
ABO incompatibility
G6PD defficiency
Infection
Metabolic/endocrine abnormalities
Delayed defecation/intestinal obstruction
Liver/biliary disease
Spleen pathology
Polycythemia
PHOTOTHERAPY
Care of Infant on Phototherapy
Risk of injury to eyes
Risk of injury to gonads
Risk of impaired skin integrity
Risk for fluid volume deficiency
Risk for hyperthermai or hypothermia
Risk of neurological injury
Imbalance nutrition
Parental anxiety
Exchange Transfusion
Isn’t he lovely?
Other Newborn Care issues
Bulb suctioning: RN 731 LPN 286
Umb cord care: RN 733 LPN 219
Heel Sticks: RN 741-43 LPN219
Circumcision: RN 755 See patient teaching page 757
LPN 290