Functional Dysphonia/ Muscle Tension Dysphonia (MTD) SPPA 6400 Voice Disorders Tasko
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Transcript Functional Dysphonia/ Muscle Tension Dysphonia (MTD) SPPA 6400 Voice Disorders Tasko
Functional Dysphonia/
Muscle Tension Dysphonia (MTD)
SPPA 6400 Voice Disorders Tasko
Muscle Tension Dysphonia:
A Functional Voice Disorder
What is a functional voice disorder?
Voice disorder in the absence of
structural/neurological pathology
Pathology insufficient to explain the degree
of dysphonia
Voice disorder based on abuse/misuse
causally linked to anatomical abnormalities
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Functional dysphonia vs.
muscle tension dysphonia
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Muscle Tension Dysphonia (MTD)
Presumed Etiology
Excess or dysregulated activity of the intrinsic and extrinsic
laryngeal muscles
Possible Sources
Technical misuse of the vocal mechanism
Learned adaptations following upper respiratory infection
Compensation for underlying vocal fold pathology
↑ laryngeal tone 2° to laryngopharyngeal reflux
Psychological/personality factors
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Muscle Tension Dysphonia (MTD)
Key Features
Laryngeal/paralaryngeal hypertonicity
“stiff” larynx
Larynx in unnatural position high in neck
Laryngeal muscle “cramping”
No unique voice quality/glottic configuration
Pre-treatment MTD samples
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Muscle Tension Dysphonia (MTD)
Some Trends
Occurs predominantly in women (90 %)*
May account for > 10 % of cases referred to
multidisciplinary voice clinics
Often the most severely affected voices encountered
Commonly follows URI symptoms*
Past history of voice problems (80%)*
Varies in response to treatment
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*Roy et al. (1997)
Recognizing Muscle Tension Dysphonia
Patient history
Auditory-perceptual Features
Laryngoscopic Features
Direct clinical examination:
Manual assessment of laryngeal musculoskeletal
tension
Primary or Diagnostic therapy
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Patient History
Vocal symptoms…
Can have a sudden onset
May have had periods of resolution
May have developed along with a URI
Symptoms suggestive of excess musculoskeletal tension
Laryngeal tenderness, soreness, pain, tightness, “swellings” which
intensify with extended voice use
Pain radiates to one or both ears
Unilateral symptoms are more common
Vocal fatigue, increased effort
Restricted dynamic range
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Patient History
Voice Use History…
may not reveal patterns of excessive voice use
Psychosocial History…
may reveal elevated stress
Stress may be coincident with history of vocal
symptoms
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Auditory Perceptual Features
Generally…
Severity of voice quality disturbance typically
consistent across a range of speech tasks
Signs are usually continuous and rarely
intermittent (no islands of normal speech)
Typically shows no improvement with falsetto
or singing
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Auditory Perceptual Features
The most disordered voices produced with
normal larynges
Wide range & variety of voice qualities
Possible existence of 5 auditoryperceptual clusters
qualitatively distinct
within a cluster, voices vary from mild-severe
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Auditory-perceptual Clusters
Cluster 1
Cluster 2
Diplophonia, intermittent pitch & voice breaks
Cluster 4
Sustained harsh, strained (tension)
Cluster 3
Persistent glottal fry
Aphonia (continuous)
Cluster 5
Elevated pitch (falsetto) – with & without strain, aphonia
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THOUGHT QUESTION
Why don’t persons with MTD
develop laryngeal pathologies?
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Laryngoscopic Features
Dysregulated muscle activity = myriad of
glottic/supraglottic contraction patterns
Rammage & Morrison (2001) suggest a
distinct set of laryngoscopic patterns
Controversial
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MTD Type 1: Laryngeal Isometric
(+/- benign mucosal disease)
Principle feature:
posterior glottic chink
Presumed due to ↑ PCA
activity
Suggested association
with benign mucosal
lesions
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MTD Type 2a – Supraglottic Lateral Compression
Lateral compression
principally at the glottis
May be some ventricular
compression
↑ closed phase
↓ vibratory amplitude
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MTD Type 2b – Supraglottic Lateral Compression
Ventricular folds are
approximated
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MTD Type 3: Anterior-posterior supraglottic
compression
↓ distance between
anterior and posterior
glottis
Arytenoids “pull” toward
epiglottis
Associated with “BogartBacall” syndrome
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MTD Type 4 – Non-adducted hyperfunction
(- supraglottic compression)
Incomplete glottal closure
with normal mobility
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MTD Type 4 – Non-adducted hyperfunction
(+ supraglottic compression)
Incomplete glottal closure
with normal mobility
Concomitant compression
of the ventricular folds
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MTD Type 5 – Bowed vocal folds
“Spindle”-shape glottis
Also associated with
aging (presbylaryngis or
presbyphonia)
Neurologic conditions
(Parkinson’s Disease)
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Note
Relation between auditory-perceptual
judgments and laryngoscopic findings are not
straightforward
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Direct Clinical Assessment
Focal palpation of circumlaryngeal area to
determine…
Presence of tenderness and/or pain
Laryngeal Stiffness
Presence of nodularity or taut bands
Reduced mobility of the larynx
Extent of laryngeal elevation
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Manual Assessment of Laryngeal
Musculoskeletal Tension
“All patients with voice disorders, regardless of
etiology should be tested for excess
musculoskeletal tension, either as a primary or
secondary cause of dysphonia” (Aronson, 1990)
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Manual Assessment of Laryngeal
Musculoskeletal Tension
Pressure is directed over the
Major horns of the hyoid bone
Superior border of the thyroid cartilage
Anterior border of sternocleidomastoid and into the
suprahyoid muscles
Determine size of the thyrohyoid space
Digital pressure should be just enough to
blanche (lighten in color) your nail bed
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From Aronson (1990)
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From Roy et al. (1996)
Treatment Options
Facilitating techniques designed to elicit easy,
relaxed phonation, phonation at optimal pitch, etc
General and focal relaxation
“Broad spectrum” treatments that focus on
increasing support and efficiency of phonatory
behavior
Manual Circumlaryngeal Techniques*
Pharmacologic Intervention (topical lidocaine)
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Manual circumlaryngeal techniques
Goals
Determine contribution of laryngeal/extralaryngeal
hypertonicity
Assure proper diagnosis and selection of appropriate
treatment
Avoid unnecessary medical or surgical management
Show Pre-Post Samples
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Manual circumlaryngeal techniques
A group of techniques
a “hands on” approach
Clinician manually repositions, repostures or
“massages” the laryngeal structure while
eliciting voice
Use voice task with a hierarchy of difficulty
Exploit facilitating techniques
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Manual circumlaryngeal techniques
May be used as
primary treatment technique for
musculoskeletal tension dysphonia (MTD)
diagnostic therapy to evaluate degree of
contribution of musculoskeletal tension to
voice disorder
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MCT: Reposturing techniques
1.
2.
3.
Compression in the A-P direction (push-back)
Impede laryngeal elevation (Pull down)
Medial compression and downward traction
(Reposturing)
Goal: Perturb the abnormal laryngeal posture and
evaluate change in voice quality
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Technique 1: Push Back Maneuver
Digital compression in the posterior direction within
the region of the larynx
Vary height and pressure
Suprahyoid
Hyoid
Infrahyoid
T-H space
Thyroid notch
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Technique 2: Pull Down Maneuver
Impede laryngeal elevation by applying downward
traction over the superior border of the thyroid
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Technique 3: Laryngeal Reposturing
Medial compression and downward traction
pressure directed over posterior aspect of thyroid
cartilage (and within T-H space)
Often helpful with non-adducted hyperfunction
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Circumlaryngeal massage
(manual laryngeal tension reduction)
What is it?
Circular motion over
Tips of major horns of the hyoid bone
Thyrohyoid space
Posterior border of the thyroid cartilage
Medial and lateral suprahyoid muscles
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Circumlaryngeal massage
(manual laryngeal tension reduction)
What is it?
Locate sites of focal tenderness, nodularity and tautness
Progress from superficial to deep pressure
Vary pressure according to patient tolerance
Patient must vocalize concurrently
Progressively increase complexity of voice stimuli
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Indications for improvement
(single session)
Improved voice quality
Pain reduction/relief
Normalized laryngeal height and mobility
Reduced muscle nodularity
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Factors affecting management of MTD using
MCT
Patient based factors
Motivation
Duration and severity of dysphonia
Persisting psychological issues
Primary and secondary gain, litigation etc…
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Clinician based factors
Technical skill
Clinician-patient dynamic
Communicate expectations
and confidence in procedure
Pt learns by doing (avoid
discussion)
Brisk therapeutic “pace”
Engage pt in process
Confront pt when effort ↓
Reinforce improvement
Expect successive
approximations to a normal
voice
Variety of facilitating
techniques
Know when to abandon a
technique or stick with it
Establish that patient is
responsible for change
May employ ‘negative’
practice
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Manual Circumlaryngeal Techniques
Evidence for clinical utility of MCT in
Functional dysphonia (muscle tension
dysphonia)
Roy et al. (1997) J Voice
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Short and long term effects of MCT
N=25
Some improvement following Tx (96%)
Normal or only mildly dysphonic following Tx
(64 %)
Deterioration of voice at follow up (25 %)
Improvement of voice at follow up (17 %)
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Short and long term effects of MCT
What about relapse?
68 % report some evidence of recurrence of some
dysphonic symptoms
Recurrence is partial rather than complete
Occurs within 3 mos. following initial treatment
Less than 4 days in duration, self limiting (i.e.
resolves spontaneously)
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Concomitant MTD & Organic/Neurogenic
Dysphonia
Elevated laryngeal musculoskeletal tension
may co-occur in patients with documented
laryngeal pathology
Why? Cause, Effect, Complication
MCT have diagnostic & treatment utility with
these populations
SPPA 6400 Voice Disorders Tasko
Manual Assessment of Laryngeal
Musculoskeletal Tension
“All patients with voice disorders, regardless of
etiology should be tested for excess
musculoskeletal tension, either as a primary or
secondary cause of dysphonia” (Aronson, 1990)
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Examples
Pre-MCT
Polyp
CVA
Reinke’s edema
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Post-MCT
MCT with BMD Patients
N=18
Gender
Age
83% female
17% male
Mean: 44.1 years
SD: 13 years
Dysphonia Duration
Mean: 2.27 years
SD: 3.64 years
4/18 bilateral nodules
2/18 unilateral nodule
2/18 unilateral polyp
2/18 Reinke’s edema
5/18 TVF
irregularities/edema/erythe
ma
1/18 ventricular cyst
1/18 interarytenoid lesion
1/18 post-intubation
granuloma
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(Tasko, et al. 1994)
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Topical Lidocaine (J Voice (2000))
Use of topical lidocaine in the treatment of muscle tension dysphonia.
Dworkin JP, Meleca RJ, Simpson ML, Garfield I.
Department of Otolaryngology, Head and Neck Surgery, Wayne State University
School of Medicine, Detroit, Michigan 48201, USA. [email protected]
This investigation explored the potential usefulness of topical lidocaine in the
treatment of muscle tension dysphonia. Three patients with this disorder, who
were previously unresponsive to standard voice therapy, were treated with
lidocaine. In each case, the outcome was prompt, clinically significant, and
sustained. Persistently high-pitched and shrill vocal quality was converted to near
normal voice patterns within 15 minutes after transcricothyroid membrane
lidocaine injection. We suggest that this temporary and simple laryngeal and
tracheal anesthetic technique may have helped to break the perverse cycle of
hyperactive glottal and supraglottal muscle contractions evident in each of these
patients during phonation efforts. We discuss the possible sensorimotor
mechanism of action of this therapeutic technique.
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Atypical Presentation (video)
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Psychological/Personality factors & MTD
Issues
State vs. Trait Factors
Psychological Factors in Disease:
Cause, Effect or Catalyst
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Psychological/Personality factors & MTD
Empirical findings in a group with MTD
Neuroticism - ↓ emotional stability and ↑ reactivity
Extraversion-sociability, dominance, energy and
enthusiasm
↑ scores
↓ scores
Psychoticism – impulsivity/aggressiveness (high) vs.
agreeable/conscientiousness (low)
Similar to normal controls
Roy et al. (2000)
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Psychological/Personality factors associated with
MTD
Additionally,
Anxious
Somatization – physical complaints
Stress reactive
Alienated
Unhappy
Roy et al. (1997)
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MTD compared to Vocal Nodule Patients
Empirical findings in a group with vocal nodules
Neuroticism - ↓ emotional stability and ↑ reactivity
Extraversion-sociability, dominance, energy and
enthusiasm
Mildly ↑ scores
↑ scores
Psychoticism – impulsivity/aggressiveness (high) vs.
agreeable/conscientiousness (low)
↑ scores
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Roy et al. (2000)
Functional Dysphonia
When no structural abnormality exists, descriptive
terms are often used which imply a psychological
etiology. For example,
Psychogenic dysphonia
Conversion dysphonia
Hysterical dysphonia
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What is “hysterical conversion”?
“conversion” of psychological stress into physical
complaints
Psychiatric literature suggests it arises from
Bland emotional unconcern/effect
Numerous physical complaints
Denial of anxieties and fears
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Is MTD a “hysterical conversion”?
This triad of features was not observed with
patients with broad diagnosis of FD (MTD)
Patients are typically quite anxious and
concerned about problem
Short answer: No
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