Transcript PPT

Learning Disabilities
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A word on definitions…
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A “learning disability” (LD) is an administrative
term
Each school board has it’s own definition.
In Ontario, there are 14 definitions of LDs
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boards, provincial an federal government (Helps, 2000)
DSM-IV has 3 subtypes:
Reading Disorder
Mathematics Disorder
Disorder of Written Expression
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Prevalence
 Different definitions makes it is hard to determine
prevalence
 Estimates range from 2 to 20% of the population
(Smith, 1994).
 Literature defines Specific Language Impairments (SLI)
separately from dyslexia
 1/2 of children in special needs classes have a
diagnosed LD.
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Case Study
 When I’m in class, there’s not enough time to copy from
the board. I lose my place. I try hard but get lost. It takes
me a long time to check nearly every word to find my
place. It’s really easy to get frustrated.
 When my teacher checks my work I have lots of
mistakes. I don't like having to stay in to finish copying
from the board. I can't read the joined up writing, I don't
always know what letter to write. I never know what I
have written. I can't see the words on the whiteboard.
They can move around and sometimes I see two words
the same.
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Case Study
 I didn’t want to ask for help because I didn’t want people
to know I have a disability. I didn’t want people to think I
was stupid.
 It was also hard when I was able to get a notetaker for
my classes. There were a few people who didn’t believe
I had a problem and who said that I was lazy.
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DSM-IV Criteria
1. Reading Disorder
2. Mathematics Disorder
3. Disorder of Written Expression
A. Achievement in any of these areas, as measured
by individually administered standardized tests,
is substantially below that expected given the
person’s chronological age, measured
intelligence, and age-appropriate education.
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DSM-IV Criteria
B. The disturbance in Criterion A significantly
interferes with academic achievement or
activities of daily living that require the ability.
C. If a sensory deficit is present, the difficulties are
in excess of those usually associated with it.
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Pure Alexia
 Pure word blindness
• Affects vision, cannot read their own writing
 Able to write (no agraphia or other writing
disability)
 Able to recognize words that are spelled aloud to
them (auditory)
 No visual agnosia (ability to recognize objects)
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Alexia: Pathology
 Occipital lobe
lesions
 Preventing visual
information from
reaching (left)
extrastriate
cortex
 Lesions to
posterior corpus
callosum
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How do we read?
At
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least two different processes:
Phonetic (sound-based) reading
unfamiliar words
requires recognition of individual symbols
sounds associated with letters and numbers
Whole-word reading
familiar word, we recognize it by its shape
context: “reading is predicting”
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Model of the reading process
Visual
Perception
Cognition
Whole Word
Recognition
Phonetic
Word
Recognition
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Model of the reading process
Visual
Perception
Whole Word word
Recognition feature
extraction
Cognition
word
recognition,
phonemic
expression
Phonetic
Word
Recognition
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Model of the reading process
Visual
Perception
Cognition
Whole Word word
Recognition feature
extraction
word
recognition,
phonemic
expression
Phonetic letter
Word feature
Recognition extraction
phoneme
recognition,
expression
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Whole word reading
Evidence comes from the study of dyslexias:
 Acquired: brain damage in those who already
know how to read
• Result of some trauma (tumour, stroke)
 Developmental: difficulties in children learning
to read
• Congenital illness or early childhood trauma
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Dyslexias
1. Surface Dyslexia
 Deficit in whole word reading
 Errors related to visual appearance of words
 Semantic understanding is normal
 They rely predominantly on phonetic reading for
understanding
 Cannot read irregular words
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Dyslexias
2. Phonological Dyslexia
 Are able to read familiar words using whole word
method
 Cannot linking letters to phonemes
 Trouble reading or learning unfamiliar words or
nonwords
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Dyslexias
3. Word-form or spelling dyslexia
 Cannot recognize whole words or sound them out
phonetically
 Can recognize individual letters and can read
words if naming the letters one at a time
 Very slow readers
 Normal auditory recognition
• Words spelt aloud
• Word memory intact
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Dyslexias
4. Direct dyslexia
 Phonemic and whole word reading intact
• Able to read aloud
 Semantic deficit: Cannot link words to meaning
• Cannot understand words they’re saying
 Cannot match words with corresponding pictures
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Dyslexias
5. Comprehension without reading
 Unable to read phonetically
 Some comprehension of words they cannot read
 However, can match words they can’t read with
pictures
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Dysgraphia: Writing Disorders
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Two types:
Motor control
directing movements to form letters and words
Various cognitive-based disorders
writing numbers but not letters
writing uppercase but not lowercase letters
Writing consonants but not vowels
writing single letters but not words
They have normal motor control
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How do we write?
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There are four possible ways:
Phonetic: Sound them out before writing
may do this with long, unfamiliar words
Imagery: Transcribing an image of what a word
looks like
visual image of word is kept in mind during
writing
Serial memory: Memorization of letter sequences
memory of sequences of letters in a word
Motor memory: often repeated written words
(signature)
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Dysgraphias
1. Phonetic (phonological dysgraphia)
 Unable to sound out words and write them
phonetically
 They cannot write unfamiliar words or
pronounceable nonwords
 Can visually image familiar words, then write
them (visual, not phonetic strategy)
 Caused by damage to the (left) superior temporal
lobe
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Dysgraphias
2. Image-Based (orthographic dyslexia)
 Disorder of visually based writing
 Can only sound out familiar words
 Can write pronounceable nonsense words
 Have difficulty spelling irregular words
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Dysgraphias
3. Serial Memory
 memorizing sequences of letters that spell
particular words (I.e. spelling bee)
 Cippolitti and Warrington (1996): patient with a
Left Hemisphere stroke
• Disrupted ability to spell words
• Impaired ability to recognize a word that examiners
would spell aloud
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Dysgraphias
4. Pure Motor Memory (direct dysgraphia)
 Can Write words that are dictated to them even
thought they cannot understand these words
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Neuropathology
1. Superior temporal lobe (Galaburda and
colleagues, 1985-1990)
 perisylvian cortex (surrounding syvian fissure)
 abnormalities in part of Wernicke’s area called
the planum temporale associated with
developmental dyslexia
 left planum temporale is usually larger in normal
controls
 in dyslexics they are both equal size
 not all people with dyslexia show this anomaly
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Neuropathology
2. Visual and auditory pathway abnormalities
 Magnocellular layer of LGN (thalamus) may be
damaged (Galaburda & Livingstone, 1993)
• Motion and spatial location
 MGN damage in thalamus can cause difficulties in
hearing
 Dyslexics often have difficulties with perception
of letters and of movements in space
• Particularly with developmental dyslexia
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What and Where Pathways
Albright & Stoner, 2002
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Neuropathology
More on visual pathway deficits…
 Transposition of letters is commom: moving
around, merging, and becoming blurry
 Maybe related to eye movements, since fixation
is often unsteady
 Motion sensitive areas of V5 in the extrastriate
cortex (posterior temporal lobe) are not activated
in persons with LDs to moving stimuli
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Etiology
1. Focal neuroanatomical abnormalities
 Cell migration in perisylvian structures
• Ectopias: abnormal positioning
• Focal microgyri: small lesions
 Cytoarchitecture in thalamic nuclei
• Disorganized, smaller cell bodies in magnocellular LGN
and MGN neurons
 Genetic correlate of these abnormalities found in
mice
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Etiology
2. Primary phonological deficits cause secondary
sensory and motor deficits
 Perisylvian anomalies are primary
 Cortical lesions produce secondary thalamic
abnormalities
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Affecting relay neurons to vision, audition, motor
centres
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Other Information
• LD is a lifelong disorder.
• Many people who have LDs may either
compensate for their difficulties, or avoid
situations where their weaknesses are likely to
show.
• More and more universities have special services
for those with LDs.
• Not so in the workplace – problems may go
unnoticed.
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