Chapter 2 - Biological Basis of Behavior
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Transcript Chapter 2 - Biological Basis of Behavior
chemicals released by
vesicles in sending
neuron
travel across the
synapse and bind to
receptor sites on
receiving neuron
2 TYPES =
EXCITATORY = stimulate the brain, increase the
likelihood that the neuron will fire an action potential
INHIBITORY = calm the brain, balance mood & are
depleted when excitatory are overactive
Neurotransmitters bind to the receptors of the
receiving neuron in a “key-lock mechanism”.
Serotonin
various functions = body
temp., sleep, mood,
appetite, and pain
Low levels =implicated
in depression & probs
with immune syst.
High = Serotonin
Syndrome
stimulant medications or
caffeine in your daily
regimen can cause a
depletion of serotonin
Norepinephrine (AKA
Noradreneline)
Prepares you for action
important for attentiveness,
emotions, sleeping, dreaming,
and learning
causes blood vessels to
contract & heart rate to
increase
GABA
Gamma-Amino Butyric Acid
An inhibitory
neurotransmitter
“nature’s VALIUM-like
substance”
Related probs = anxiety,
seizures, Huntington’s disease
Valium and similar
antianxiety drugs work at
GABA synapses
Dopamine
main focus neurotransmitter
Affects neurons associated with
voluntary movement
role in learning, memory, and emotions
Loss of dopamine-producing cells =
Parkinson’s Disease
Excess = focusing issues, less motivation,
schizophrenia
Stimulants (ex: cocaine, meds for
ADD/ADHD, caffeine) cause dopamine
to be pushed into the synapse so that
focus is improved
BUT cause a depletion over time
Acetylcholine
triggers muscle contraction
important role in arousal and attention
Loss = linked to Alzheimer’s Disease
Endorphins
linked to pain control and
to pleasure
Reduce pain by inhibiting
or “turning down” neurons
that transmit pain
information
natural, opiate-like
neurotransmitters
“morphine within”
Dopamine pathways
are involved with
diseases such as
Parkinson’s disease
caused by a
deterioration of brain
neuron’s that produce
dopamine (it is still
unknown why this
occurs)
Although not the sole cause of
schizophrenia, dopamine
unbalance is consistently
seen found in patients with
schizophrenia
Drugs that prevent dopamine
from binding to receptors
reduce the symptoms of
schizophrenia
Agonist
Increases production
Activates the neuron
receptor that it
attaches to
Antagonist
Decreases production
Deactivates the
neuron receptor that
it attaches to