Transcript 58. When your muscles hypertrophy, these are the fibers that get

1. – 65.
1. Name this
protein.
actin
2. Name this specific band.
H Band
3. Name
this unit.
Fascicle (fasciculus)
4.
Name
this
unit.
Myofiber (muscle cell)
5. Name this
protein.
myosin
6. Name
bluish CT
perimysium
7. Name this dark line.
M line
8. Name these specific dark
lines.
Z line
9. Name
these purple
structures.
mitochondria
10. Name
this entire
structure.
myofiber
11. Name
these blue
structures.
Sarcoplasmic reticulum
12. Name
these yellow
structures.
Transverse tubules
13. Name
this unit.
triad
14. Name
this entire
unit; it is the
smallest unit
of a muscle
contraction
(red
bracket).
sarcomere
15. Name
the
cytoplasm
inside
sarcoplasm
16. Name this
covering.
muscle
epimysium
2
3
17. Which is
the
crossbridge?
1
4
4
1
2
18. Which is in the contacted state?
2
19. What is the name of this
myofilament?
actin
20. What is the name of this molecule?
ATP
21. What is the SPECIFIC role of this
molecule in muscle contraction? Allows
myosin to perform a(n)__________ so
actin is pulled inward.
power stroke
22. What is the name of the 3 purple
protrin complex?
troponin
23. What happens to the width of the A
band during contraction?
Stays the same
What condition are these muscles
in?
hypertrophy
25. What is
represented
by the green
circles in this
diagram?
calcium
26. Where specifically is calcium stored
in the muscle?
Sarcoplasmic
reticulum
27. What is the name of the gray
molecule?
tropomyosin
28. What is the other source for
recharging the ATP battery in the
muscles? (this compound is unique to
muscles)
Creatine phosphate
29.What is the specific function of
calcium in a muscle contraction?
Calcium ions bind to_________
which causes a change in the
conformation of the tropomyosin
complex that exposes the myosin
binding sites on the actin filament.
troponin.
30. What is this known as?
Sliding filament theory
31. What is this set-up called?
Motor unit
32. The products of aerobic
respiration are water, ATP, and _____.
Carbon dioxide
33. Name this
red area shown
by red arrow.
Motor end plate
34. Name this
yellow structure
of which you see
the end of.
Motor neuron
35. Name these
blue
“containers”.
Synaptic vescicles
36. What is the
general name for
the compound
that is in these
blue
“containers”.
neurotransmitter
37. Name these
structures shown
by red arrows.
Transverse tubules
38. Name this
entire area
shown by
bracket.
Neuromuscular
junction
39. During the contraction of a
sarcomere, calcium ions bind with
the protein _____.
troponin
40. This is a graph of
a muscle contraction.
What is it called?
myogram
41. Name this
blue part of a
muscle twitch.
Latent period
42. Name this
red part of a
muscle twitch.
Contraction
Contraction period
period
43. What is the bracketed part of
this graph representing dealing
with a muscle contraction?
Treppe or
summation
Tetanus (tetanic
contraction)
44. What is the bracketed part of
this graph representing dealing
with a muscle contraction?
45. Choose the type of muscular
contraction shown below.
isotonic
46. The reddish brown color of
muscle is due to the presence of
_____ molecules
myoglobin
47. Which molecule is produced
during exercise, resulting in the
oxygen debt?
Lactic acid
48. Would
there be
mostly slow
twitch or fast
twitch fibers in
this part of
your
Thanksgiving
Turkey?
Fast twitch
49. The minimum stimulus needed to
cause a contraction is called the _____.
Threshold stimulus
50. What type of muscle
is found in these
structures?
Smooth muscle
51. A large broad sheet of
connective tissue, such as on the
abdomen or head, that muscles
can attach to is called what?
aponeurosis
52. – 57. NAME THE
MUSCULAR
DISORDER/DISEASE
.
52.
state of muscular rigidity that begins 34 hours after death and lasts about 24
hours
•After death, Ca+2 ions leak out of the
SR and allow myosin heads to bind to
actin
•Since ATP synthesis has ceased,
crossbridges cannot detach from actin
until proteolytic enzymes begin to
digest the decomposing cells.
Rigor mortis
53.
an involuntary and forcibly
contracted muscle that does not
relax
cramp
54. a group of muscle-destroying
deseases inherited as a sex-linked
recessive trait; the muscle shrinks and
no longer contracts; it is due to the
absence of a protein that “props” up the
muscle cell.
Muscular dystrophy
55. This disorder causes extreme
muscle fatigue
It is an autoimmune disease - one's
own body makes antibodies against
one’s own ACh receptors
Myasthenia gravis
56. A bacteria from undercooked
foods produces a toxin which
prevents ACh release at the N-M
Junction
•The result is no
muscle stimulation and no
muscle movement; results in
paralysis and death
botulism
57. This bacterial disease
heightens the chemical signal from
the nerve to the muscle which
causes the muscles to
continuously tighten up in a huge
continuous contraction or spasm.
tetanus
58. – 65. WRITE
WHETHER EACH
STATEMENT
DESCRIBES SLOW
TWITCH OR FAST
TWITCH MUSCLES.
58. When your muscles
hypertrophy, these are the fibers
that get larger.
fast
59. These are red.
slow
60. These are easily fatigued.
fast
61. These are best in long slow
sustained contractions.
slow
62. These have more
mitochondria.
slow
63. These would be more
prominent in marathon runners
than sprinters.
slow
64. These do not have the
specialized myoglobin.
fast
65. Would be more numerous in
leg muscles that pectoral
muscles.
slow
THE END