Transcript document
PowerPoint® Lecture Slides
prepared by Vince Austin,
Bluegrass Technical
and Community College
CHAPTER
Elaine N. Marieb
Katja Hoehn
William A. Simmons
Human
Anatomy
& Physiology
SEVENTH EDITION
23
PART B
The Digestive
System:
Mechanism
Pharynx - know
From the mouth, the oro- and laryngopharynx
allow passage of:
Food and fluids to the esophagus
Air to the trachea
Lined with stratified squamous epithelium and
mucus glands
Has two skeletal muscle layers
Inner longitudinal
Outer pharyngeal constrictors
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Esophagus - know
Muscular tube going from the laryngopharynx to
the stomach
Travels through the mediastinum and pierces the
diaphragm
Joins the stomach at the cardiac orifice
http://youtube.com/watch?v=1Om_qQl_SsQ
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Esophagus - understand
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Figure 23.12
Esophageal Characteristics - understand
Esophageal mucosa – nonkeratinized stratified
squamous epithelium
The empty esophagus is folded longitudinally and
flattens when food is present
Glands secrete mucus as a bolus moves through
the esophagus
Muscularis changes from skeletal (superiorly) to
smooth muscle (inferiorly)
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Deglutition (Swallowing) - know
Coordinated activity of the tongue, soft palate,
pharynx, esophagus, and 22 separate muscle
groups
Buccal phase – bolus is forced into the oropharynx
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Deglutition (Swallowing) - know
Pharyngeal-esophageal phase – controlled by the
medulla and lower pons
All routes except into the digestive tract are sealed
off
Peristalsis moves food through the pharynx to the
esophagus
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Deglutition (Swallowing) - understand
Bolus of food
Tongue
Pharynx
Epiglottis
Glottis
Trachea
(a) Upper esophageal
sphincter contracted
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Deglutition (Swallowing) - understand
Uvula
Bolus
Epiglottis
Esophagus
(b) Upper esophageal
sphincter relaxed
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Deglutition (Swallowing) - understand
Bolus
(c) Upper esophageal
sphincter contracted
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Deglutition (Swallowing) - understand
Relaxed
muscles
Bolus of
food
Longitudinal
muscles
contract,
shortening
passageway
ahead of bolus
Gastroesophageal
sphincter closed
(d)
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Circular muscles
contract,
constricting
passageway
and pushing
bolus down
Stomach
Deglutition (Swallowing) - understand
Relaxed
muscles
Gastroesophageal
sphincter open
(e)
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Bolus of food
Tongue
Uvula
Pharynx
Epiglottis
Bolus
Epiglottis
Glottis
Esophagus
Trachea
(a) Upper esophageal
sphincter contracted
summary
Bolus
(b) Upper esophageal
sphincter relaxed
Relaxed
muscles
Bolus of
food
Longitudinal
muscles
contract,
shortening
passageway
ahead of bolus
Gastroesophageal
sphincter closed
(d)
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(c) Upper esophageal
sphincter contracted
Circular muscles
contract,
constricting
passageway
and pushing
bolus down
Stomach
(e)
Relaxed
muscles
Gastroesophageal
sphincter open
Stomach - know
Chemical breakdown of proteins begins and food
is converted to chyme
Cardiac region – surrounds the cardiac orifice
Fundus – dome-shaped region beneath the
diaphragm
Body – midportion of the stomach
Pyloric region – made up of the antrum and canal
which terminates at the pylorus
The pylorus is continuous with the duodenum
through the pyloric sphincter
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Stomach - know
Greater curvature – entire extent of the convex
lateral surface
Lesser curvature – concave medial surface
Lesser omentum – runs from the liver to the lesser
curvature
Greater omentum – drapes inferiorly from the
greater curvature to the small intestine
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Stomach - know
Nerve supply – sympathetic and parasympathetic
fibers of the autonomic nervous system
Blood supply – celiac trunk, and corresponding
veins (part of the hepatic portal system)
Normal stomach:
http://youtube.com/watch?v=Ln09qihUi3g&feature=related
Stomach ulcer:
http://youtube.com/watch?v=kRwMAMe1IWY&feature=re
lated
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know
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Figure 23.14a
Microscopic Anatomy of the Stomach - understand
Muscularis – has an additional oblique layer that:
Allows the stomach to churn, mix, and pummel
food physically
Breaks down food into smaller fragments
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Microscopic Anatomy of the Stomach - know
Epithelial lining is composed of:
Goblet cells that produce a coat of alkaline mucus
The mucous surface layer traps a bicarbonaterich fluid beneath it
Gastric pits contain gastric glands that secrete
gastric juice, mucus, and gastrin
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Microscopic Anatomy of the Stomach - understand
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Microscopic Anatomy of the Stomach - understand
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Microscopic Anatomy of the Stomach - understand
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Glands of the Stomach Fundus and Body understand
Gastric glands of the fundus and body have a
variety of secretory cells
Mucous neck cells – secrete acid mucus
Parietal cells – secrete HCL and intrinsic factor
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Glands of the Stomach Fundus and Body - know
Chief cells – produce pepsinogen
Pepsinogen is activated to pepsin by:
HCL in the stomach
Pepsin itself via a positive feedback
mechanism
Enteroendocrine cells – secrete gastrin, histamine,
endorphins, serotonin, cholecystokinin (CCK), and
somatostatin into the lamina propria
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Stomach Lining - know
The stomach is exposed to the harshest conditions
in the digestive tract
To keep from digesting itself, the stomach has a
mucosal barrier with:
A thick coat of bicarbonate-rich mucus on the
stomach wall
Epithelial cells that are joined by tight junctions
Gastric glands that have cells impermeable to HCL
Damaged epithelial cells are quickly replaced
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Digestion in the Stomach - know
The stomach:
Holds ingested food
Degrades this food both physically and chemically
Delivers chyme to the small intestine
Enzymatically digests proteins with pepsin
Secretes intrinsic factor required for absorption of
vitamin B12
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Regulation of Gastric Secretion - know
Neural and hormonal mechanisms regulate the
release of gastric juice
Stimulatory and inhibitory events occur in three
phases
Cephalic (reflex) phase: prior to food entry
Gastric phase: once food enters the stomach
Intestinal phase: as partially digested food enters
the duodenum
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Cephalic Phase - know
Excitatory events include:
Sight or thought of food
Stimulation of taste or smell receptors
Inhibitory events include:
Loss of appetite or depression
Decrease in stimulation of the parasympathetic
division
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Gastric Phase - know
Excitatory events include:
Stomach distension
Activation of stretch receptors (neural activation)
Activation of chemoreceptors by peptides, caffeine,
and rising pH
Release of gastrin to the blood
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Gastric Phase - know
Inhibitory events include:
A pH lower than 2
Emotional upset that overrides the parasympathetic
division
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Intestinal Phase - know
Excitatory phase – low pH; partially digested food
enters the duodenum and encourages gastric gland
activity
Inhibitory phase – distension of duodenum,
presence of fatty, acidic, or hypertonic chyme,
and/or irritants in the duodenum
Initiates inhibition of local reflexes and vagal
nuclei
Closes the pyloric sphincter
Releases enterogastrones that inhibit gastric
secretion
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Release of Gastric Juice: Stimulatory Events summary
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Release of Gastric Juice: Inhibitory Events summary
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Regulation and Mechanism of HCL Secretion - know
HCL secretion is stimulated by ACh, histamine,
and gastrin through second-messenger systems
Release of hydrochloric acid:
Is low if only one ligand binds to parietal cells
Is high if all three ligands bind to parietal cells
Antihistamines block H2 receptors and decrease
HCl release
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Regulation and Mechanism of HCl Secretion - detail
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Response of the Stomach to Filling - understand
Stomach pressure remains constant until about 1L
of food is ingested
Relative unchanging pressure results from reflexmediated relaxation and plasticity
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Response of the Stomach to Filling - know
Reflex-mediated events include:
Receptive relaxation – as food travels in the
esophagus, stomach muscles relax
Adaptive relaxation – the stomach dilates in
response to gastric filling
Plasticity – intrinsic ability of smooth muscle to
exhibit the stress-relaxation response
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Gastric Contractile Activity - understand
Peristaltic waves move toward the pylorus at the
rate of 3 per minute
This basic electrical rhythm (BER) is initiated by
pacemaker cells (cells of Cajal)
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Gastric Contractile Activity - know
Most vigorous peristalsis and mixing occurs near
the pylorus
Chyme is either:
Delivered in small amounts to the duodenum or
Forced backward into the stomach for further
mixing
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Gastric Contractile Activity - summary
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Regulation of Gastric Emptying - understand
Gastric emptying is regulated by:
The neural enterogastric reflex
Hormonal (enterogastrone) mechanisms
These mechanisms inhibit gastric secretion and
duodenal filling
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Regulation of Gastric Emptying - know
Carbohydrate-rich chyme quickly moves through
the duodenum
Fat-laden chyme is digested more slowly causing
food to remain in the stomach longer
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Regulation of Gastric Emptying - summary
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Small Intestine: Gross Anatomy - know
Runs from pyloric sphincter to the ileocecal valve
Has three subdivisions: duodenum, jejunum, and
ileum
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Small Intestine: Gross Anatomy - know
The bile duct and main pancreatic duct:
Join the duodenum at the hepatopancreatic ampulla
Are controlled by the sphincter of Oddi
The jejunum extends from the duodenum to the
ileum
The ileum joins the large intestine at the ileocecal
valve
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Small Intestine: Microscopic Anatomy - know
Structural modifications of the small intestine wall
increase surface area
Plicae circulares: deep circular folds of the mucosa
and submucosa
Villi – fingerlike extensions of the mucosa
Microvilli – tiny projections of absorptive mucosal
cells’ plasma membranes
http://youtube.com/watch?v=xu5jDCX2cHM
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Duodenum and Related Organs – be able to label
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Small Intestine: Microscopic Anatomy – be able to
label
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Small Intestine: Histology of the Wall - understand
The epithelium of the mucosa is made up of:
Absorptive cells and goblet cells
Enteroendocrine cells
Interspersed T cells called intraepithelial
lymphocytes (IELs)
IELs immediately release cytokines upon
encountering Ag
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Small Intestine: Histology of the Wall - know
Cells of intestinal crypts secrete intestinal juice
Peyer’s patches are found in the submucosa
Brunner’s glands in the duodenum secrete alkaline
mucus
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Intestinal Juice - know
Secreted by intestinal glands in response to
distension or irritation of the mucosa
Slightly alkaline and isotonic with blood plasma
Largely water, enzyme-poor, but contains mucus
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Liver - know
The largest gland inside the body
Superficially has four lobes – right, left, caudate,
and quadrate
The falciform ligament:
Separates the right and left lobes anteriorly
Suspends the liver from the diaphragm and anterior
abdominal wall
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Liver - understand
The ligamentum teres:
Is a remnant of the fetal umbilical vein
Runs along the free edge of the falciform ligament
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Liver: Associated Structures - know
The lesser omentum anchors the liver to the
stomach
The hepatic blood vessels enter the liver at the
porta hepatis
The gallbladder rests in a recess on the inferior
surface of the right lobe
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Liver: Associated Structures - know
Bile leaves the liver via:
Bile ducts, which fuse into the common hepatic
duct
The common hepatic duct, which fuses with the
cystic duct
These two ducts form the bile duct
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Gallbladder and Associated Ducts – be able to label
http://youtube.com/watch?v=bNMsNHqxszc
&feature=related
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Liver: Microscopic Anatomy - understand
Hexagonal-shaped liver lobules are the structural
and functional units of the liver
Composed of hepatocyte (liver cell) plates
radiating outward from a central vein
Portal triads are found at each of the six corners of
each liver lobule
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Liver: Microscopic Anatomy - understand
Portal triads consist of a bile duct and
Hepatic artery – supplies oxygen-rich blood to the
liver
Hepatic portal vein – carries venous blood with
nutrients from digestive viscera
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Liver: Microscopic Anatomy - know
Liver sinusoids – enlarged, leaky capillaries
located between hepatic plates
Kupffer cells – hepatic macrophages found in liver
sinusoids
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Liver: Microscopic Anatomy - know
Hepatocytes’ functions include:
Production of bile
Processing bloodborne nutrients
Storage of fat-soluble vitamins
Detoxification
Secreted bile flows between hepatocytes toward
the bile ducts in the portal triads
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Composition of Bile - know
A yellow-green, alkaline solution containing bile
salts, bile pigments, cholesterol, neutral fats,
phospholipids, and electrolytes
Bile salts are cholesterol derivatives that:
Emulsify fat
Facilitate fat and cholesterol absorption
Help solubilize cholesterol
Enterohepatic circulation recycles bile salts
The chief bile pigment is bilirubin, a waste product
of heme
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The Gallbladder - know
Thin-walled, green muscular sac on the ventral
surface of the liver
Stores and concentrates bile by absorbing its water
and ions
Releases bile via the cystic duct, which flows into
the bile duct
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Regulation of Bile Release - know
Acidic, fatty chyme causes the duodenum to
release:
Cholecystokinin (CCK) and secretin into the
bloodstream
Bile salts and secretin transported in blood
stimulate the liver to produce bile
Vagal stimulation causes weak contractions of the
gallbladder
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Regulation of Bile Release - know
Cholecystokinin causes:
The gallbladder to contract
The hepatopancreatic sphincter to relax
As a result, bile enters the duodenum
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Regulation of Bile Release - understand
4 Vagal stimulation causes
weak contractions of
gallbladder
3 Bile salts
and secretin
transported via
bloodstream
stimulate liver
to produce bile
more rapidly
5 Cholecystokinin
(via bloodstream)
causes gallbladder
to contract and
hepatopancreatic
sphincter to relax;
bile enters
duodenum
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
2 Cholecystokinin
and secretin enter the
bloodstream
6 Bile salts reabsorbed into blood
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Regulation of Bile Release - understand
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
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Regulation of Bile Release - understand
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
2 Cholecystokinin
and secretin enter the
bloodstream
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Regulation of Bile Release - understand
3 Bile salts
and secretin
transported via
bloodstream
stimulate liver
to produce bile
more rapidly
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
2 Cholecystokinin
and secretin enter the
bloodstream
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Regulation of Bile Release - understand
4 Vagal stimulation causes
weak contractions of
gallbladder
3 Bile salts
and secretin
transported via
bloodstream
stimulate liver
to produce bile
more rapidly
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
2 Cholecystokinin
and secretin enter the
bloodstream
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Regulation of Bile Release - understand
4 Vagal stimulation causes
weak contractions of
gallbladder
5 Cholecystokinin
(via bloodstream)
causes gallbladder
to contract and
hepatopancreatic
sphincter to relax;
bile enters
duodenum
1 Acidic, fatty chyme
entering duodenum causes
release of cholecystokinin
and secretin from
duodenal wall
enteroendocrine cells
2 Cholecystokinin
and secretin enter the
bloodstream
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3 Bile salts
and secretin
transported via
bloodstream
stimulate liver
to produce bile
more rapidly
Review
http://youtube.com/watch?v=1nFwO9iU5Y&feature=related
http://youtube.com/watch?v=LpaEWpYUdDQ
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Functions of the large intestine
Reabsorb water and compact material into feces
Absorb vitamins produced by bacteria
Store fecal matter prior to defecation
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The Large Intestine
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Figure 24.23a
Figure 24.23 The Large Intestine
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Figure 24.23b, c
The rectum
Last portion of the digestive tract
Terminates at the anal canal
Internal and external anal sphincters
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Histology of the large intestine
Absence of villi
Presence of goblet cells
Deep intestinal glands
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Physiology of the large intestine
Reabsorption in the large intestine includes:
Water
Vitamins – K, biotin, and B5
Organic wastes – urobilinogens and
sterobilinogens
Bile salts
Toxins
Mass movements of material through colon and
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The
Defecation
Reflex
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Figure 24.25
Processing and absorption of nutrients
Disassembles organic food into smaller fragments
Hydrolyzes carbohydrates, proteins, lipids and
nucleic acids for absorption
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Carbohydrate digestion and absorption
Begins in the mouth
Salivary and pancreatic enzymes
Brush border enzymes
Disaccharides and trisaccharides
Monosaccharides
Absorption of monosaccharides occurs across the
intestinal epithelia
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Lipid digestion and absorption
Lipid digestion utilizes lingual and pancreatic
lipases
Bile salts improve chemical digestion by
emulsifying lipid drops
Lipid-bile salt complexes called micelles are
formed
Micelles diffuse into intestinal epithelia which
release lipids into the blood as chylomicrons
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Protein digestion and absorption
Low pH destroys tertiary and quaternary structure
Enzymes used include pepsin, trypsin,
chymotrypsin, and elastase
Liberated amino acids are absorbed
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Absorption
Water
Ions
Nearly all that is ingested is reabsorbed via
osmosis
Absorbed via diffusion, cotransport, and active
transport
Vitamins
Water soluble vitamins are absorbed by diffusion
Fat soluble vitamins are absorbed as part of
micelles
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Digestive Secretion and Absorption of Water
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