Neurological Assessment File
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Transcript Neurological Assessment File
Jane E. Binetti DNP MSN RN
Generally
part of any overall physical
Past Medical History
• If neuro problems are suspected:
Avoid suggesting what the problem is
History of the illness is important for the pt. to recall
May not be a reliable historian
Medications
Surgery
or treatments
• Cancer therapies
Developmental progression
Functional ability
• ADL’s
• Falls
Generally, six primary nerve functions
are assessed:
• Mental status, cranial nerves, motor, sensory,
cerebellar function, and reflexes
Mental
Status reflects functional ability
• General appearance
• Cognition
A and O
Language
Memory
• Mood and affect
• 12 Pair
• I Olfactory
• II Optic
• III Oculomotor
• IV Trochlear
• V Trigeminal
• VI Abducens
• VII Facial
• VIII Acoustic
(vestibulocochler)
• IX Glossopharangeal
• X Vagus
• XI Spinal accessory
• XII Hypoglossal
CN I = Olfactory
• Ability to smell
• Chronic infections and smoking decrease ability
• Aberrancy could be tumor, basilar fracture
CN II = Optic
• Tests visual fields
• One eye at a time
• Stare at nose, while identifying peripheral objects
• Snellen Eye Chart
• Aberrancy lesions on optic nerve or anywhere it
tracks through the brain
CN III = Oculomotor
• Constriction and accommodation
• PERRL or PERRLA
• Convergence
• Ptosis
CN IV = Trochlear
CN VI
= Abducens
All are tested together
• Follow a finger left/right and up/down
• Disconjugate gaze
• Nystagmus
Oculocephalic
reflex:
• With eyelids open, turn
the head
• Normal reflex = eyes move
in opposite direction
• Abnormal = eyes move
with head
Oculovestibular
reflex:
• Cold water test (caloric)
V = Trigeminal Nerve
• Tested with eyes closed
• Sensory with pin
• Motor with teeth clenching
• Corneal reflex is sensory with V
VII = Facial Nerve
• Corneal reflex is motor with VII
• Blink to threat
• Raise brows
• Shut eyes tight
• Purse lips, smile/frown
CN VIII
= Acoustic / Vestibulocochlear
• Eyes closed, MD uses ticking watch, or whispers a
directive
• More diagnostic are:
Weber: tuning fork for equilateral hearing
Rinne: tuning fork for bone vs air conduction
Audiometry (ABR)
Typically for auditory dysfunction like vertigo or tinnitus
CN
IX = Glossopharyngeal
• Responsible for tongue and swallowing
• Palate and uvula should rise
CN
X = Vagus
• Responsible for pharynx, larynx, gi, cardiac, lungs
Tested
together: both innervate the pharynx
• IX reflects sensory piece of Gag reflex
• X is motor piece causing retching
CN XI = Accessory Nerve
• Accessory muscles
• Shrug shoulders
• Turn your head against resistance
CN XII = Hypoglossal Nerve
• Stick your tongue out
• Midline
• Up and down
• Side to side against a
tongue blade
Information
on bulk power of muscle, and
coordination and balance
• Resistance, grasp
• Pronator drift
• Balance and coordination
• Finger to nose testing
• Patient should normally have symmetrical
strength and motion
Hypotonia
- flaccid
Hypertonia - spastic
Myoclonus – muscle spasms
Athetosis – slow writhing movements
Chorea – involuntary, purposeless
Dystonia – impaired tone
Patient
should be able to discriminate:
• Touch with eyes closed
• Sharp vs dull
• Temperature differences
• Vibration
• Positional sense
Finger placement
Romberg test
• Cortical sensorium
Graphesthesia, stereognosis
Tendons attached to skeletal muscles
have receptors that will react
• Tapped with reflex hammer
• Range is 0/5 – 5/5 with 2/5 as “normal”
• Arreflexia to hyperreflexia with clonus
• Generally Biceps or Patellar reflexes are tested
Triceps, Brachioradialis and Achilles may be used
Jane E. Binetti DNP RN
Common complaint
• Can be functional
• Often benign but can be key assessment
Causes can be intracranial and
extracranial
Not all cranial tissue is sensitive to pain
Based on diagnostic criteria:
• Primary headaches include
Tension
Migraine
Cluster headaches
• Secondary headaches are caused by:
Sinus infections
Neck injuries
Strokes
Medication overuse can also cause headaches
A patient can have more than one kind
Most common headache reported
Gradual onset of band-like tightening
Not affected by activity
Episodic vs. Chronic – minutes to days
Cause
• Once thought to be head/neck muscle
contraction
• EBP suggests neurovascular
Abnormal neuron sensitivity
Symptoms
• No nausea, band like pressure
• Photophobia, phonophobia
• Palpable neck tension
Diagnostics
• Patient history
Treatment
• Typically non-opiod analgesics
• ASA or Tylenol
Usually unilateral, throbbing, recurring
headache
Typically related to trigger
• Foods
• Weather
• Stress
• Hormones
• Fatigue
Risk factors:
• Most common onset 20-30
• 17% females and 6% males in US
• Family history – most have first degree family
• Low socioeconomics
• Heavy workload
• Low education level
• Frequent tension headaches
IHS
Sub Classifications of Migraines
• Without Aura
Most common type
Formally called “common” migraine
• With Aura
Only about 10% of patients
Formally called “classic” migraine
Auras
are sensory signals
• Visual, auditory or motor symptoms
Sounds, smells, patterns of light, scotomas, weakness,
sensation of movement
Prodromes
are physical signals
• Photophobia
• Hyperactivity, irritability
• Food cravings
Migraine
can last hours to days
Patients withdraw to quiet, dark places
Throbbing pain matches the pulse
Some people are incapacitated by them,
but others work through them
Some feel they worsen over time
Done
by History
Patients may keep diaries to find
triggers
Neuro exam may be “normal”
• Diagnostic tests may not show anything
CT
and MRI are not routine
• Variation of usual symptoms may be cause for
secondary causes to be ruled out
Mild:
• Non opioids usually work well
ASA
Acetaminophen
Ibuprofen
Combination meds with caffeine
• Other behavioral therapies
Moderate
to severe:
• Triptans are first line therapy
• Reduce neurogenic inflammation of vessels
Cause vasoconstriction
• Best taken preemptively at onset
Sumatriptan (Imitrex)
Rizatriptan (Maxalt)
Zolmitriptan (Zomig)
Sumatriptan/Naproxen (Treximet) combination
• Watch your cardiac patients! - vasoconstriction
Topiramate (Topamax)
• Antiseizure drug taken daily as preventative
• Can cause seizures if stopped abruptly
• Push fluids to avoid calculi
• Can cause drowsiness
Beta adrenergic blockers:
• Propranolol (Inderal)
• Atenolol (Tenormin)
Alpha adrenergic blockers:
• Ergotamine (Ergomar)
• Dihydroergotamine (DHE)
Analgesic/Anti-inflammatory
• Cambia (Diclofenac)
Analgesic
combinations
• Fiorinal (Butalbital/Aspirin/Caffeine)
• Fioricet (Butalbital/Acetaminophen/Caffeine)
Corticosteroids
• Dexamethasone (Decadron)
Tricyclics
SSRIs
Alpha
and Beta adrenergic blockers
Anti-seizure meds
Ca channel blockers
Biofeedback
Relaxation
Cognitive behavioral therapy
Rare
headache ~ 0.1% of population
Exacerbate and remit
Cause:
• Not clearly known
Triggers: ETOH, weather, odors
• Trigeminal nerve
• Dysfunction of intracranial vessels
• Sympathetic nervous system
• ? hypothalamus
A
pt complaining of sharp, stabbing pain
Lasts minutes to hours
Many a day/many days
Cluster is weeks to months long
• Sharp head pain:
eye, forehead, cheek
• Tearing, swelling,
pallor, flushing even
ptosis
Primarily
by History
Keeping a diary is helpful
Other tests done to rule out a more
significant cause:
• CT, MRI, MRA, LP
• R/O tumor, aneurysm, infection, neuro disease
Clusters
often happen suddenly at night
and are brief, meds are little help
• Hi flow O2 is helpful
100% at 6-8L for 10 minutes
Can repeat after 5 minute breaks
Pt at home would need O2 supply
Triptans
can be useful
Prohylactic tx:
• Verapamil, lithium, ergotamine, NSAIDS
• Intranasal lidocaine spray
For
refractory Cluster Headaches:
• Nerve blocks
• Ablation
• Percutaneous radiofrequency
• Deep brain stimulation
• Overuse of
analgesics or any
meds used for tx can
cause rebound
• Avoidance of overuse
and ensuring slow
withdrawal is helpful
• Avoidance of triggers
•Hunger
•Fatigue
Help
pts:
• With life style changes, diet, smoking
• Relaxation/distraction/meditation/yoga
• Identify and avoid triggers
• Management
Early identification
Compliance with treatment
Stress management