Burst Fractures
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Transcript Burst Fractures
A Class for Foreign MD Students
Spine Trauma
Dr. Yue Wang
王
跃
MD,
PhD
Department of Orthopedic Surgery
The First Affiliated Hospital, college of Medicine, ZheJiang University
浙江大学医学院附属第一医院骨科
Anatomy of the Spine
Cervical spine, C,
Thoracic Spine, T, 12
Lumbar Spine, L, 5
Sacrum, S, 5, fused
Coccyx, 3-4, fused
AP and Lateral View
Osteology: The Vertebra
The shape of the
vertebrae changes!
C
T
L
The Cervical vertebrae
atlas, C1
C3-7
axis, C2
The lumbar spine
The thoracic and lumbar vertebrae
Articular processes
Pars
Superior
Articular
Process
Pars
interarticularis
Zygapophyseal
Joint
(Facet Joint)
Inferior
Articular
Process
Arthrology
Intervertebral Discs
Fibrocartilaginous joint
Increase in size from C to L
Ratio remains the same
Make up 20-30% of length of column
The intervertebral disc
Two components: nucleus and anulus fibrosus
Major Ligaments of the Spine
Anterior Longitudinal Ligament - ALL
Dense band along anterior and lateral surface of the
vertebral bodies from C2 to sacrum
Posterior Longitudinal Ligament – PLL
Runs along posterior surface of vertebral bodies (anterior to
spinal canal)
Ligamentum flavum
Supraspinous lig. and interspinous lig.
Ligaments
Ligaments of the spine
Spinal Cord
Part of the CNS along with brain
Contained within vertebral canal
Extends from cranium to 1st-2nd lumbar
vertebrae
Lumbar roots & sacral nerves for a
“horse-like tail” called cauda equina
2 plexuses
Brachial, lumbosacral
Nerves
Each vertebrae has a nerve that exits
either below or above it
31 pairs of spinal nerves
8 cervical nerves
12 thoracic nerves
5 lumbar
5 sacral
1 coccygeal
The thoracic spine
Spinal Injuries
Suspected spinal injuries
High
speed crash: MVA
Unconscious
Multiple
injuries
Neurologic
Spinal
deficit
pain/tenderness
Spinal injury
• 5% worsen neurologically at hospital
• Protection is a priority
• Detection is a secondary priority
• Spinal evaluation complicated by multiple
traumae;
Mechanism of Injury
Axial compression:
Compress/crush the C spine; tends to fracture the
vertebra into pieces
Distraction
Pull apart the spinal elements
Usually associated with flexion or extension forces
Mechanism of Injury
Mechanism of Injury
Flexion:
Severe forward bending
In cervical spine, often leads to neurological
damage
Extension:
Severe backward bending
Not always associated with neurological trauma
Mechanism of Injury
Mechanism of
Injury
Shear:
Mechanism of
Injury
Rotational
Rescue
Save
the life: ABC;
Evaluate/identify
a patient with spinal
injury;
Using
a spinal board;
Determine
appropriate disposition;
Classification of Neurologic
Injury
Frankel Score
A: Complete loss of motor and sensory function
B: Only sensory function remains
C: Motor function is present but of no practical use
(i.e., can move legs but not walk)
D: Motor function impaired (i.e.can walk but not
with normal gait)
E: No neuro impairment noted
Diagnosis
Spinal injury
• Physical examinations;
• X-ray;
• CT;
• MRI;
• Electromyogram (EMG);
C Spine Injury
The C spine is involved in more than half of
all the patients with traumatic spine injuries
Stability is a crucial key point to determine the
choice of management, and it is determined by
the integrity of the anatomical structures
constituting the cervical spine
Lesions with spinal cord involvement are unstable
One of the most common pitfalls in
patients with cervical spine injury is
missed or delayed diagnosis!
Reasons:
1) inadequate radiographs (44%) ;
2) misinterpretation of adequate
radiographs (47%)
Goals of Management
Decompression
Alignment
Stabilization
Reconstruction, if necessary
Adequate management
starts from
A proper classification of the injury type
Assessment of stability of the affected segment
Instability : “the loss of the ability of the spine
under physiological loads to maintain
relationships between vertebrae in such a way that
the spinal cord or nerve roots are not damaged or
irritated and deformity or pain does not develop.
Stable or unstable
Lesions with spinal cord involvement are severe and unstable
and require surgical management.
On the other hand, lesions without spinal cord involvement
can evolve toward a stable or unstable lesion
Lesions that mainly involve bone structures may determine
temporary instability.
Successful fracture healing (with bony callus formation) and
subsequent stabilization of these lesions may be obtained
with reduction and immobilization.
On the other hand, ligamentous or osteo-ligamentous lesions
may cause irreversible instability with secondary dislocation.
Imaging Evaluation
Alignment/Bones /Soft Tissue
Look for
Normal atlanto-occipital alignment
Predental space 3 mm or less
Prevertebral soft tissue space less than 5 mm
anterior to C3
Spinal canal plain film AP diameter 13 mm or
greater
Any horizontal translation of one vertebra on
the next
Fanning of the space between spinous
processes
Fracture of any bone
Upper C spine trauma
Jefferson’s fracture
Burst fracture of rings of C1
Sometimes without neurological
deficit
Hangman’s fracture
Involves C2
Sudden hyperextension of head and
neck forces vertebrae against spinal
cord
Complete neurological loss
Radiograph
Odontoid fractures
Type 1 fractures at tip
Type 2 fractures at waist
Type 3 fractures at base
MRI
Lower C Spine Fractures
n
Facet joint dislocation
May occur without fracture
Possible spinal canal compromise
Thoraco-lumbar fractures
axial
may result in anterior discoligamentous disruption and
posterior compression fractures of facets, laminae, or spinous
processes
rotation
the posterior ligamentous and osseous elements fail in tension
hyperextension
Axial load may result in a burst fracture
flexion/distraction
compression
Combine compressive forces and flexion/distraction
mechanisms and are highly unstable
shear
Shear forces produce severe ligamentous disruption and are
often associated with spinal cord injury
Classifications
White
and Panjabi defined clinical instability of
the spine :
Loss of the ability of the spine under
physiologic loads to maintain relationships
between vertebrae in such a way that there is
neither damage nor subsequent irritation to
the spinal cord or nerve root and, in addition,
there is no development of incapacitating
deformity or pain from structural changes.
Denis’ 3 columns theory &
Classifications
minor and major injuries:
minor injuries
included fractures of the
articular, transverse, and
spinous processes as
well as the pars
interarticularis.
Major injuries
were divided into
compression fractures,
burst fractures, flexiondistraction (seat-belt)
injuries, and fracture
dislocations
Wedge/compression fractures
These injuries may involve both endplates(A),
the superior endplate only(B),
the inferior endplate only (C), or
a buckling of the anterior cortex with both endplates
intact (D)
Burst fractures
Five subgroups:
type A fracture is characterized
by #’s of both the superior and
inferior end plates, usually as a
result of an axial load.
The types B and C burst
fractures involve the superior
and inferior end plates,
respectively. result from an axial
load coupled with a flexion load.
An axial and a rotational load
give rise to a type D burst
fracture.
The type E fracture is
characterized by widened
pedicles with a burst and lateral
flexion injury.
Flexion-Distraction fractures
(Seat-belt/Chance Fractures)
Fracture dislocations
General guidance
Treatment based on Denis
classification
Wedge
most
Fractures
treated with external orthosis
surgical
indications:
failure
of brace progressive deformity
>50%
height loss
>30°
kyphosis
Burst
canal
Fractures
compromise & neuro deficit should be treated
with surgical decompression
Consider non-operative if:
neuro intact
kyphosis <20-30°
residual spinal canal > 50%
anterior body height > 50% of posterior height
posterior column intact
purely boney injury
show stability in supine/upright x-rays in brace
Seat-belt/Chance Fractures
usually unstable
if purely osseous injury consider trial of bracing and close
serial xray f/u
if ligamentous injury healing less predictable, probably best to
do posterior fusion
Fracture-Dislocation
generally all unstable and require surgery
associated with >75% neuro injury (1/2 of which are complete),
dural tears, exposed/avulsed roots
usually posterior fusion (early anterior fusion dangerous since
associated visceral injury is common)
staged anterior decompression if necessary
Case 1
Post-operative surgery
Case 2
Post-operative
Thanks!
The Rock Mountain, 2012