Dr. Nefcy*s Top 10 Rashes - Overseas School Health Nurses
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Transcript Dr. Nefcy*s Top 10 Rashes - Overseas School Health Nurses
Sandra Leipheimer MSN, APRN, BC-PNP
Heidelberg High School
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Continually
renewed
Stores fat and water
Provides protection
Gets rid of waste
Regulates
temperature
Largest organ of the
body
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Bed Bugs:
Cimex lectularius
(Cimidieae)
Harmful?
Do not transmit any
infectious agents
Only stay on skin to feed on a
few drops of blood
Treatment
Aimed at itch- AH and
corticosteroids
Treat secondary infections
from scratching
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Lice
Pediculosis (place)
Facts
Not “medically
necessary” to
remove NITS
Most are non-viable
(dead or hatched)
Personal decision by
parent
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Lice
feed on human
blood
Not a sign of poor
hygeine
Transmitted by direct
contact
Do not spread
disease
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What
to put on school fact sheet?
Not a major health issue
Nuisance factor
RX: Least intrusive to Most intrusive treatment
Mechanically remove lice (risk reduction)
OTC treatment ( oovacidal)
Rx
Examine all family members for live (crawling) lice
If infested –treat
If not – need not be treated
Myths and Treatment folklore
ARE
THEY ACTUALLY INFESTED?
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History
When started?
What else going on?
Other S & S?
Rx = better or worse?
Exposure?
Associated
Signs & Symptoms
Fever
URI
Previous illness or treatment
Exam
Skin + other symptoms
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Superficial
Infections
Bacterial
Fungal
Viral
Tattoos
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Caused
by S. aureus a
normal skin colonizer
in up to 50% of people
Yellow, oozing, crusty
sore, often starts in
nose Itch is common
Requires antibiotic
Excluded for 24 hrs
and keep covered at
school (contagious)
1-3 days incubation
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Bullous
Staph. aureus- fluid filled
blisters
Non-Bullous
S. aureus & streptococcus –
crusted
MRSA becoming more
common
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Skin
lesions usually caused by different strain
than those causing “strep throat”
Can cause glomerulonephritis if strain is GrA
B-hemolytic
Blood or brown sugar (maple sugar; coke) urine
May
culture lesions to be sure what is
infectious agent
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Caused
by
inflammation to the
hair follicles
shaving/friction from
tight clothes; ingrown
Typically
infected with
S. aureus
Avoid trigger
Antibiotics if infection
suspected
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Usually
caused by
S. aureus
Increased frequency
with MRSA and other
antibiotic resistant
organisms
Need oral antibiotics
and often need
drainage
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Methicillin
Resistant Staphylococcus Areas
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Exclusion
Policy for Schools?
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Spread
Skin to Skin
Touching contaminated objects (drainage)
Regular Cleaning and Disinfecting
No evidence that spraying or fogging rooms or surfaces
with disinfectant works better than focusing on
frequently touched surfaces
Wash hands: soap and water alcohol-based sanitizers
Take showers: immediately after exercise; don’t share
items
Use barriers: cover cuts; towels on items
Wash uniform: dry completely in dryer; wash after each
use
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Culture
suspicious lesions
Not return to play until:
Appropriate antibiotic taken for at least 72 hours
Drainage from wound has stopped
No new lesions in past 48 hours
CDC:
do not exclude unless MD directed…
Sport-specific guidance should be in place
Excluded if wounds cannot be properly covered
“properly covered” = securely attached
bandage/dressing containing all drainage and
remain intact during activity
No water; whirl pools; therapy pools
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Nontuberculous mycobacteria (NTM) “various
types”
M. chelonae exists in tap water
MRSA and “others”…
Persistent inflammatory reaction
Located within margins of recent intradermal
tattoo
Cause- ? using tap water or distilled water for ink
Many believe distilled or reverse-osmosis water
is sterile
Many other reasons…
Cartilage piercings >> delay healing
“Allergies” >>> contact dermatitis
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What
do you think??
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“THINK
BEFORE YOU INK”
www.fda.gov Tattoos & Permanent Makeup
NOV 00 JUN 08 FEB 10 AUG 12 FDA Notices
related to unregulated materials and health
risks
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Tinea
– sounds like a bug, but really a fungus
(trichophyton, microsporum)
Name of group of diseases caused by fungus
Named for location of infection
Acquired by touching infected person, damp
surfaces, pets
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Generally
a circular
lesion (hence the
name “ring worm”
Raised (can be
blistery)
Itchy
Red scaly ring with
central clearing
Treat topically with
anti-fungal ointment
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Tinea
in the scalp
Patchy areas of hair
loss or breakage and
scale
Must be treated with
oral medications
Can extend to a
kerion (hypersensitivity reaction)
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Non scarring
Noninflammatory
alopecia
Twist or pull hair
Deny behavior
Done in private
7 X > kids than adults
2 X Girls > Boys
Scalp most common
Eyelashes and eyebrows
Psychosocial
issues
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Moist
area between toes
Itchy, red, blisters, cracking, peeling
Nails can also become infected= tinea
unguium
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Itching
in groin, thigh folds, anus
Red, raised, scaly patches that blister and
ooze
Patches have sharply-defined edges
Redder around outside with normal skin tone in
center
Usually
starts in creases of upper thigh and
does not involve scrotum/penis but may
spread to anus causing itching and discomfort
Usually less severe than other tinea
infections but lasts longer
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Also
known as eczema: “the itch that rashes”
Hypersensitivity reaction similar to allergy
Long-term swelling and redness
(inflammation of skin)
May lack certain proteins in the skin which
leads to greater sensitivity
Often accompanies asthma
Eczema = compromised skin barrier @ critical
point in development
Strong link with food allergies
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Eczema + Food Allergies + Asthma
ANAPHYLAXIS
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Very, very itchy
Red/salmon colored
patches
Can look like pustules
Likes the antecubetal
and popliteal fossa
Actually allergy
mediated
Treated with
emollients/steroids
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Subgroup
of disorders results from
hypersensitivity to physical or mechanical
factors
Cold urticaria
Pressure -induced urticaria and angioedema
Aquagenic urticaria
Solar urticaria
Exercised- induced urticaria
History and distribution
Confirmed by challenge
Cold Challenge ( immediate … 4 hours later)
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Trauma
–induced pressure
urticaria
Initial white line
= reflexive vasoconstriction followed by
pruritic, erythematous swelling
Wheal
or Flare
Reaction
Chronic ? Cause
Interferes with skin
testing = false +
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Eye
“rashes” or Conjunctivitis
Bacterial
Viral
Allergic
Vernal
Chemical
Irritant
Drugs
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Bacterial
etiology
Very contagious
Red conjunctiva,
itchy not painful,
purulent drainage
Should not be
associated with fever
Treat with topicals
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Chlamydia
Gonoccocal
Herpes
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Typically
associated
with an upper
respiratory infection
Watery, red, irritated
Doesn’t usually have
any discharge
Refer anyone who
wears contacts and
has a red eye to a
doctor
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Typically
occurs in
someone with
seasonal allergies
(hay fever)
Itchy/bumpy/puffy
and red
Improves with topical
drops and oral antihistamines
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Symptoms
URI & Cough
Red, Watery Eyes
Tired
Koplik’s
spots (2-3 days after above)
Blotchy Rash (3-5 days after symptoms)
Starts on face at hairline
Spreads downward to neck, trunk, arms, legs,
feet
Fever spike with rash (~ 104 F 40 C)
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Caused
by infection of human papilloma virus
Common – fingers and toenails
Subungual (under) periungual ( around) nails
Plantar-
soles of feet (painful)
Genital – STI (condyloma)
Flat – appear wear shaving ( most common in
children however)
Trends:
Children- warts tend to go away
Adults- tend to stay
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Viral
etiology
Difficult to treat
Can occur anywhere
Most therapies aimed
at triggering the
immune system
Treat with topical
irritant/duct
tape/cryo/laser
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Also viral (poxvirus)
Difficult to treat
Contagious
Center has viral load
Most advocate leaving
them alone, though can
currette or treat with
topical irritant
If many lesions- may be
immune system concern
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Non-specific
reactions
Irritants
Infections
Immune System Disease
Allergies
Cold & Heat
Chemicals
Wind
Sun
Evil Eye
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Typically associated with
an allergic reaction
Can be seen as a
response to viral
infection, foods,
medications or ?? =
idiopathic
Refer if S&S of other
system: cough/wheeze
or swelling of
lips/tongue; N & V
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Acute hypersensitivity
reaction
Can be seen in response
to drugs, illness (viruses,
bacteria) foods and
immunizations
May look similar to
hives, but typically not
pruritic, look like
targets, can be painful,
and unlike hives, persist
(are fixed)
No treatment
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Drugs
Barbiturates
PCNs
Phenytoin
Sulfonamides
EM Minor – better in 2-6 weeks; can recur
Herpes simplex
Mycoplasma
EM Major = SJS
Reaction to medication
Infections; radiation Rx; UV light
Epidermal necrolysis – morality risk high
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Drug
Atypical antipsychotics
Antibiotics
Sulpha
Penicillins
Other as listed
Skin
and mucous membranes reaction
Widespread pain
Facial swelling
Tongue swelling
Top
reaction- Medical Emergency
layer of skin>> necrosis & sloughing
Blistering & Erosion
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Starts
with a herald
patch (~ 1-3 weeks)
Confused as tinea
Fine
scaling oval
macules/papules
Pinkish brown
Christmas
tree
pattern
Lasts 6-12 weeks, no
treatment
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Very
itchy in 50 %
Can be concentrated in
groin, forearms, shin
Some report feeling mildly ill 1- 2 week before
herald patch
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Herpes
Simplex
Oral = “cold sores” Type 1
Genital = Type 2
But can occur in either area and either type
Herpes
Zoster
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Most
infections with
Type I are
asymptomatic
Most commonly
presents as
gingivostomatitis
Characterized by
fever, and painful
vesicles on oral
mucosa/gingiva
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Digital
Herpes
Painful
Contagious
Virus enters break in skin (torn cuticle) – from
own skin or others
60 % HSV-1 40 % HSV-2
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Chickenpox
– many
are vaccinated
Can be fatal for the
neonate
Fever, painful or
puritic versicles,
typically start on the
trunk and spread
Shingles = Zoster
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Reactivation
of
varicella
Very painful
Occurs in
dermatomal
distribution
Can be treated if
recognized early
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Zoster infects nerve on head
Facial nerve near inner ear
Painful rash on TM, canal,
earlobe, tongue, roof of
mouth, on same side as
weakness or face
Eye closing; motor movements -
Hearing loss on side
Vertigo
Urgent referral
Prompt RX
Steroids
Antivirals
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Streptococcal
infections (Group A strep or
strep pyogenes)
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Fever,
sore throat,
malaise, stomach s/s
Contagious
Treat with 10 days of
penicillin
Can go back to school
after 24 hrs on
antibiotics
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Strept Throat
with rash
S/T; Fever; H/A
Abd pain; N/V
+ lymph nodes
Rash appears 1-2
days: red and
sandpaper texture
After 7-14 days skin
rubs off / peels
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Can also cause glomerulonephritis (coke colored urine),
rheumatic fever, impetigo
Also associated with severe invasive infections – pneumonia,
arthritis, sepsis, toxic shock syndrome, etc
Rheumatic Fever
Appears 2-4 or 1-5 weeks following Strep infection
Inflammation is the cause of symptoms:
Inflammation of the heart - chest pain, fatigue, SOB
Inflammation of the joints - arthritis symptoms
Inflammation of the skin - skin rashes and nodules
Inflammation of the CNS (central nervous system) - chorea
(jerking), personality changes
Increased risk of fulminant bacteremia from strep pneumo in kids
with asplenia
Vaccine in US
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Mumps
and Measles
Must always keep in mind based upon local
immunization policies
World travel – one small planet
Immunization: concern with waning immunity
over time
WHO Travel Advice
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Viral
infection that
causes systemic
disease and swelling
of the salivary glands
More severe disease
the older you get
Not vaccinated
against in some
countries (Japan)
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Characterized
by
fever, cough, rash,
conjunctivitis
Encephalitis with
permanent brain
damage 1:1000
Death 1-3:1000
Worse if young, sick
and/or malnourished
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Petechiae,
Purpura and Vasculitis
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Red
blood cells
outside the vessel
walls – don’t blanch
Seen with low
platelets
Can also been seen
with trauma, cough,
increased pressure
(pertussis, asthma)
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Never
forget about
abuse – bruising or
petechiae, or other
signs of trauma – in
multiple stages of
healing, unusual
places, in strange
shapes
Obligated to report
!!!!!!!!!!!!!!!!!!!!!!!
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Large
areas of
cutaneous hemorrhage
Refer to doctor
Usually bad, may be
life threatening
Meningococcemia,
something wrong with
bone marrow
HSP(Henoch-Schönlein Purpura
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Tetrad:
Usually Self-Limiting
Usually children
Small minority of cases can
cause severe kidney and
bowel disease
Follows URI ~ 10 days
following
Mean age 5.9 years
Purpura is due to vasculitis
not low platelets (IgA in walls
of blood vessels)
Steroids ease symptoms and
may disrupt abnormal immune
response
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Purpura
Arthritis & Arthralgia
Swelling around joints
Kidney inflammation
Abdominal pain
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Erythema
Induration
Temperature
Lesion
Papule
Pustule
Macule
Vesicles
Hyper
/ Hypo pigmentation
Linear Oval Circular Target Concentric
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Size
of each or all lesions
Color
Description of shape/distribution of lesion(s)
Location
What areas are spared?
Trend
or Changes over time
Mark areas
Aggravating
or Alleviating Factors
Associated Signs or Symptoms
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1+1+1=3
History
2 Associated
S&S
Exam
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Potential
Assessment &
Recommendations
79
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https://identitfy.us.com
SchoolNurse.com CD Head Louse Infestations:
Evidence-Based Strategies & Best Practices for
Tackling Head Lic
NASN S.C.R.A.T.C.H.
http://www.cdc.gov/mrsa/groups/
http://www2.aap.org/new/idphotos.htm
MedlinePlus
www.cdc.gov
www.fda.gov
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