Transcript Hemodynamic disorders p.1 - Patho

Infectious and parasitic diseases
SYLLABUS:
RBP(Robbins Basic Pathology) Chapter:
General Pathology of Infectious Diseases
PBD (Pathologic Basis of Disease)
Viral Infections and Bacterial Infections
(from the chapter Infectious Diseases)
Infectious and parasitic diseases
50
Follicular hyperplasia of the lymph node
119
Candidiasis (esophagus)
241
Enterobiasis (appendix)
123
Trichinosis (muscle)
122
Cysticercosis (CNS)
Follicular hyperplasia of the lymph node
(Reactive lymphoid hyperplasia, follicular pattern)
-increase in number and size of germinal centers,
due to expansion of follicular center, occasionally of
mantle as well
- follicular polarization retained
Follicular hyperplasia of the lymph node
Follicular hyperplasia of the lymph node
Candidiasis (esophagus)
Organisms:
a mixture of budding yeast forms (blastoconidia)
and pseudohyphae, tubular structures separated by
narrowed areas
Tissue reaction:
usually purulent but may be minimal or
granulomatous
Candidiasis (esophagus)
Candidiasis (esophagus)
Enterobiasis (appendix)
Tissue:
- normal mucosa
- or eosinophilic reaction
- or acute appendicitis
Worms:
- distinctive lateral alae („thorns” in cross section)
- sections may show eggs within worms (eggs may
also be seen alone)
Enterobiasis (appendix)
Enterobiasis (appendix)
Trichinosis (muscle)
Worms:
- encysted coiled roundworms
Tissue:
-muscle cells with inflammation including eosinophils and
macrophages
- fiber necrosis
- larval death triggers eosinophilic granulomatous
inflammation leading to calcification
or
- a chronic, inactive stage where encysted organisms are
found in the absence of any cellular response.
Trichinosis (muscle)
Trichinosis (muscle)
Cysticercosis (CNS)
Worm:
- fluid-filled cyst with eosinophilic laminated wall
lined by monolayer of rare nuclei
- older lesions fibrotic and calcified
Tissue:
- little inflammation unless ruptured
Cysticercosis (CNS)
Cysticercosis (CNS)