RTC PA CATHETER
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Transcript RTC PA CATHETER
Swan Gantz Catherter
and the Meaning of its
Readings
Justin Chandler
Surgical Critical Care Fellow
The Pulmonary Artery Catheter and
Its History
The Pulmonary Artery Catheter and
Its History
Cardiac catheterization dates back to Claude
Bernard
used it on animal models
Clinical application begins with Werner
Forssmann in the 1930s
inserted a catheter into his own forearm, guided it
fluoroscopically into his right atrium, and took an Xray picture of it
The Pulmonary Artery Catheter and
Its History
The pulmonary artery catheter
introducted in 1972
Frequently referred to as a SwanGanz catheter, in honor of its
inventors Jeremy Swan and
William Ganz, from Cedars-Sinai
Medical Center
The “sail” or balloon tip was a
modification of the simple portex
tubing method developed by
Ronald Bradley
Ganz added the thermistor
Indications
Diagnostic indications:
Shock states
Differentiation of high vs low
pressure pulmonary edema
Primary pulmonary
hypertension
Valvular disease
Intracardiac shunts
Cardiac tamponade
Pulmonary embolus
Monitoring and management
of complicated acute
myocardial infarction
Assessing hemodynamic
response to therapies
Management of multiorgan
failure
Severe burns
Hemodynamic instability after
cardiac surgery
Assessment of response to
treatment in patients with
primary pulmonary
hypertension
Therapeutic indications:
Aspiration of air emboli
Placement
Place an introducer
Hand ports off to RN, inspect and have RN flush
catheter
R IJ > L SC > R SC > L IJ
Femoral is an option
if CCO, leave tip in the holder to calibrate
Place swandom on catheter
Insert about 15cm and the inflate balloon
Slowly and steadily advance catheter watching the
waveforms
NB When wedged, not the volume required
Placement
Typical Cather Insertion Landmarks
Anatomic Structure
Distance
Right atrium
20 to 25 cm
Right ventricle
30 to 35 cm
Pulmonary artery
40 to 45 cm
Pulmonary capillary wedge
45 to 55 cm
Conformation
Zones of West
Insertion tips
Turn CVP off!
Once in the RV advance to PA quickly to
avoid coiling, ventricular arrhythmia.
Difficulty getting into PA
Valsava
Calciun iv
HOB up
Basics to Remember
Hemodynamic variables should not be
interpreted in isolation
Integration of variables with the clinical situation
increases the accuracy of assessment
Trends are generally more useful than isolated
variables at a single point in time
What does a PAC tell us?
Direct measurements
CVP
PA (systolic and
diasotolic)
PAOP (wedge)
SvO2 (mixed)
Calculated data
Stroke volume (SV/SVI)
Cardiac output (CO/CI)
Vascular resistance (SVR,PVR)
Oxygen delivery
Extended calculations
CCO
Stroke work
End diastolic volume, EF
Variables of Hemodynamics
Variable
Assessment
Stroke volume/index
Pump performance
Cardiac output/index
Blood flow
CVP/RAP
R heart filling pressure
PAOP/Wedge
L heart filling pressure
SvO2
Tissue oxygenation
Normal Values
Variable
Value
Stroke volume (SVI)
50-100 mL/beat (25-45)
Cardiac output (CI)
4-8 L/min(2.5-4.0)
CVP/RAP
2-6 mmHg
PAOP/Wedge
8-12 mmHg
SvO2
0.60 – 0.75
Additional Values
Variable
Value
SVR (SVRI)
900-1300 (1900-2400)dynes
sec/cm5
PVR
40-150 dynes sec/cm5
MAP
70-110 mmHg
Equations to Remember
CO = SV x HR or SV = CO / HR
SV = EDV – ESV or EDV x EF
C = ΔV/ΔP
SVR = (MAP – CVP) x 80 / CO
LSW = (MAP – LVEDP) x SV x 0.0136
To convert to index: divide by BSA
BSA = [Ht + Wt-60]/100 (in cm & kg)
Cardiac Output
Major determinate of oxygenation delivery to
tissue
Abnormalities are viewed in the context of
SV/SI and SvO2
Remember: a normal CO/CI may be associated
with a low SV/SI in the presence of tachycardia
Factors Affecting CO
Physiologic
Dysrhythmias
Septal defects
Tricuspid regurg
Respirations
Technical
Bolusing technique
Themistor malfunction
Factors not affecting
CO:
Iced vs room temp
NSS vs D5
Pt elevation (<45o)
5 cc vs 10 cc
CO Measurement
Typically done with thermodilution method
A cold solution of fixed volume is injected and a
thermsitor measures the change in temperature
The area under the curve is integrated to calculate
the CO
The waveform should be examined to determine if
the technique was good
If the accuracy is in doubt, the Fick method may
be used
CO Waveforms
Fick Method
CO = VO2 / [CaO2 – CvO2] * 10
SaO2 and SvO2 often substituted
CO = VO2 / [SaO2 – SvO2] * Hgb * 1.34* 10
VO2 is not usually measured
Can use 3.5 mL/kg or 125 mL/m2
If metabolic rate is abnormal, the calculation may be
incorrect
Stroke volume
If low
Inadequate volume (hypovolemia)
Impaired ventricular contraction
(ischemia/infarction)
Increased SVR (drugs)
Valve dysfunction (MVR)
If high
Low vascular resistance (sepsis, drugs)
CVP
Reflects R heart diastolic function and volume
status
60-70% of blood volume is in venous system
Abnormalities are viewed in the context of
SV/SI
If high (>6) implies right ventricular dysfunction,
especially if SV is low
If low (< 2) implies hypovolemia especially if SV is
low
CVP
High
Hypervolemia
RV failure
Tricupid stenois/regurg
Cardiac tamponade
Cardiac pericarditis
Pulm HTN
Chronic LV failure
Low
Hypovolemia
Venodiliation
PAOP
Reflects left ventricular end
diastolic volume
Assumes a static column
of blood from ventricle to
catheter during diastole and
consistent compliance
Abnormalities are viewed in
the context of SV/SI
If high (>18) implies left
ventricular dysfunction,
especially if SV is low
If low (< 8) implies
hypovolemia especially if SV is
low
PAOP
High
Hypervolemia
LV failure
Cardiac tamponade
Cardiac pericarditis
Mitral stenosis/regurg
Atrial myxoma
Pulmonary diseases
Low
Hypovolemia
Aortic regurg
Elevated LVEDP
(>25mmHg) with
decreased compliance
PAOP
Conditions in Which PAD Does Not Equal
PAOP (1 – 4 mm Hg)
Increased PVR
Pulmonary hypertension
Cor pulmonale
Pulmonary embolus
Eisenmenger’s syndrome
Filling Pressures
If low, but other parameters are normal may
only require observation
If CO/CI are also low, treatment may be warranted
If SvO2 and/or SV/SI are also low treatment is
needed
Pulmonary congestion also warrants treatment
SvO2
Reflects the balance between oxygen delivery
and utilization
The larger the abnormality, the greater the risk
of hypoxemia
Remember: a normal or high SvO2 may
represent a threat to tissue oxygenation
SvO2
A low SvO2 usually warrants investigation
Evaluate:
SV/SI
May require treatment, even if CVP/PAOP are normal
Hb/Hct
SaO2 (>90%)
Reasons for oxygen consumption to be elevated
Abnormally high SvO2 may be indicative of a
septal defect
Continuous Cardiac Output
Newer generation catheter
Uses continuous cardiac output measurements
without need for bolusing
Allows for right heart “volumetric” data
RVEDV, RVEF, and RVSV
RVSW and RVSWI
Also provides continuous SvO2 measurements
Additional Reference Numbers
(R)EDV (SV/EF)
100-160 ml
(R)EDVI
60-100 ml/m2
ESV (EDV-SV)
50-100 ml
ESVI
30-60 ml/m2 (*)
LVSWI
45-75 gm-m/m2/beat
RVSWI
5-10 gm-m/m2/beat
Waveform Analysis
Changes in pressure waveforms are due to:
Blood entering or leaving a chamber
Changes in wall tension (contraction/relaxation)
Are always preceded by electrical stimulation
Waveforms are also affected by changes in
intrathoracic pressure (present as rhythmic
changes)
The Waves
The Waves - CVP/RA
The a wave occurs with atrial contraction
The c wave occurs with closure of the tricuspid valve
It occurs at the end of the QRS (RST junction)
The v wave occurs with filling of the atria with the tricupid valve closed
It occurs after the P wave in the PR-interval
Occurs after the T wave
The mean of the a wave is the CVP
The Waves - RV
Has a sharp, rapid upstroke and a rapid down stroke
Falls to near zero
The Waves - PA
Characteristics
Rapid up stroke and
down stroke
Dicrotic notch (closure of
pulmonic valve)
Smooth runoff
End systolic wave occurs
after the T wave
End diastolic occurs
after the QRS
The Waves - PAOP
Characteristics
May contain 3 waves
a atrial contraction
c closure of mitral valve
(often absent)
v filling of atria with mitral
valve closed
Found after the QRS
Found well after the T
Mean PAOP
Average the a wave
a Wave Differential
Large
Tricuspid or mitral regurg
Decreased ventricular
compliance
Loss of A-V synchrony
Junctional rhythms
Tachycardia (>130)
Absent
A-fib
Junctional rhythms
Paced rhythms
Ventricular rhythms
v Wave Differential
Large
Tricuspid or mitral regurg
Noncompliant atrium
Ventricular
ischemia/failure
Absent
V-fib
Asystole
PEA
Diagnosis by Waveform
Mitral insuffiency
Prominent v wave
Proximity of v and a
waves
Returns to a more normal
configuration after
afterload reduction
Diagnosis by Waveform
VSD
Presents with increased
SvO2
Note the delay in the v
wave
May respond to afterload
reducers
Diagnosis by Waveform
Cardiac Tamponade
As with constrictive
pericarditis, there is
equalization of diastolic
pressures
Note the loss of the y
descent in cardiac
tamponade
Diagnosis by Waveform
Constrictive pericarditis
Note the equalization of
the diastolic pressures
Unlike tamponade, there
is an exaggeration of the y
descent due to a more
rigid pericardium
Points to remember
Intrathoracic pressure during inhalation and
exhalation cause pressures in the heart to vary
Therefore all pressures should be measured at endexpiration when intrathoracic pressure is closest to
zero
Points to Remember
Limitations in hemodynamic monitoring
Ventricular filling pressures do not always accurately reflect ventricular
filling volume
The PAOP is normally slightly (1-5 mm Hg) less than the PAD pressure
The pressure-volume relationship depends upon ventricular compliance
If compliance changes, the pressure-volume relationship changes
This relationship stills exists with pulm hypertension due to LV failure
However, with an ↑ PVR or tachycardia (>125 bpm) this relationship may
breakdown and the PAD becomes significantly higher than the PAOP
The PAOP may not equal LVEDP when
there is high alveolar pressures
when the catheter tip is above the left atrium
severe hypovolemia
tachycardia (130 bpm)
in mitral stenosis.
Points to remember
Calculated variables (e.g. SVR, PVR & SV/SI)
are limited in value due to assumptions made in
their calculations
Complications
Air embolism
Arrhythmias
S&S: hypoxemia, cyanosis, hypotension/syncope, “machinery
murmur”, elevated CVP, arrest
Tx: place in left lateral trendelenburg, FiO2 of 100%, attempt
aspiration of air, CPR
Prevention: keep balloon inflated, minimize insertion time
Tx: removal of catheter, ACLS
Heart blocks
Typically RBBB occurs, so avoid PACs in LBBB
Tx: transvenous/transcutaneous pacers, PACs with pacer
Complications
Knotting
Prevention: minimize insertion time, avoid pushing agaist
resistance, verify RA to RV transition
Tx: check CXR, attempt to unknot
Pulmonary artery rupture
S&S: hypoxemia, hemoptysis, circ collapse
Prevention: withdraw PAC if spontaneously wedges or
wedges with < 1.25 cc of air
Tx: stop anti-coagulation, affected side down, selective
bronchial intubation, PEEP, surgical repair (CPB or ECMO)
Complications
Pulmonary infarction
Prevention
Avoid distal positioning of catheter
Check CXR
Monitor PA EDP instead of PAOP
Pull back if spontaneous wedge occurs
Limit air in cuff (pull back if < 1.25 cc)
Tx
CXR
Check cath position, deflate and withdraw
Observe
Complications
Infection
Prevention!
Aseptic technique
Dead-end caps
Sterile sleeve (swandom)
Minimize entry into system
Avoid glucose containing fluid
Avoid over changing of tubing, etc (72-96 hr)
Remove catheter ASAP
Thrombus
Prevention – continuous flush +/- heparin
Tx – lytic agent ; remove catheter
Emerging Technology
Devices exist that use arterial pressure waveform to
continuously measure cardiac output
Variations of the arterial pressure are proportional to stroke
volume
Several studies demonstrate that SVV has a high sensitivity
and specificity in determining if a patient will respond
(increasing SV) when given volume (“preload
responsiveness”)
Limitations
Only used in mechanically ventilated pts
Wildly inaccurate when arrhythmias are present
Emerging Technology
Impedance Cardiography (ICG)
Converts changes in thoracic
impedance to changes in volume
over time
ICG offers noninvasive, continuous,
beat-by-beat measurements of:
Stroke Volume/Index (SV/SVI)
Cardiac Output/Index (CO/CI)
Systemic Vascular Resistance/Index
(SVR/SVRI)
Velocity Index (VI)
Thoracic Fluid Content (TFC)
Systolic Time Ratio (STR)
Left Ventricular Ejection Time (LVET)
Pre-Ejection Period (PEP)
Left Cardiac Work/Index
(LCW/LCWI)
Heart Rate
In a Nutshell
Right heart failure
Hypotension
Left heart failure
Low CI, high PVR
High PAOP, low CI, high
SVR
High PAOP, low CI,
CVP ≈ POAP
Low CVP, PAOP, CI
High SVR
Cardiogenic
Tamponade
Hypovolemia
High CVP,PAOP, SVR
Low CI
Sepsis
Low CVP, PAOP, SVR
High CI
References
Pulmonary Artery Catheter Education Project
http://www.pacep.org
Chatterjee, The Swan-Ganz Catheters: Past, Present, and Future: A Viewpoint.
Circulation 2009;119;147-152
Edwards Scientific
http://ht.edwards.com/presentationvideos/powerpoint/strokevolumevariation/s
trokevolumevariation.pdf
Question #1
Which one of the following statements is
most correct?
A) A CVP <2 mmHg usually reflects
hypovolemia if the SVI is>45 mL/beat/M2
B) A CVP >6 mmHg usually reflects RV failure
if the SVI is <25 mL/beat/M2
C) A PAOP >18 mmHg usually reflects LV
failure if the SVI is >45 mL/beat/M2
D) A PAOP <8 mmHg usually reflects
hypovolemia if the SVI is >25 mL/beat/M2
Answer #1
Which one of the following statements is
most correct?
A) A CVP <2 mmHg usually reflects
hypovolemia if the SVI is>45 mL/beat/M2
B) A CVP >6 mmHg usually reflects RV failure
if the SVI is <25 mL/beat/M2
C) A PAOP >18 mmHg usually reflects LV
failure if the SVI is >45 mL/beat/M2
D) A PAOP <8 mmHg usually reflects
hypovolemia if the SVI is >25 mL/beat/M2
Question #2
Identify the condition most consistent with
the following hemodynamic profile:
SvO2 ... 0.50 ... PAOP ... 21 mmHg
CI ... 2.2 L/min/M2 ...CVP/RA ... 4 mmHg
SVI ... 23 ml/beat M2 ... HR ... 98
A) Hypovolemia
B) Hypervolemia
C) LV dysfunction/failure
D) Bilateral ventricular failure
Answer #2
Identify the condition most consistent with
the following hemodynamic profile:
SvO2 ... 0.50 ... PAOP ... 21 mmHg
CI ... 2.2 L/min/M2 ...CVP/RA ... 4 mmHg
SVI ... 23 ml/beat M2 ... HR ... 98
A) Hypovolemia
B) Hypervolemia
C) LV dysfunction/failure
D) Bilateral ventricular failure
Question #3
Identify the condition most consistent with
the following hemodynamic profile: SvO2 ...
0.47 ... PAOP ... 4 mm Hg
CI ... 2.0 L/min/M2 ... CVP/RA ... 2 mm
Hg
SVI ... 19 ml/beat/M2 ... HR ... 111
A) Hypovolemia
B) Hypervolemia
C) LV dysfunction/failure
D) Bilateral ventricular failure
Answer #3
Identify the condition most consistent with
the following hemodynamic profile: SvO2 ...
0.47 ... PAOP ... 4 mm Hg
CI ... 2.0 L/min/M2 ... CVP/RA ... 2 mm
Hg
SVI ... 19 ml/beat/M2 ... HR ... 111
A) Hypovolemia
B) Hypervolemia
C) LV dysfunction/failure
D) Bilateral ventricular failure
Question #4
Which of the combined set of hemodynamic values is of
greatest concern?
A) CO = 6.9 L/min; CI = 3.8 L/min/M2 SV = 63 mL/beat;
SVI = 34 mL/beat/M2 BP = 102/52 mm Hg SvO2 = 0.83
B) CO = 4.3 L/min; CI = 2.5 L/min/M2 SV = 43 mL/beat;
SVI = 25 mL/beat/M2 BP = 94/62 mm Hg SvO2 = 0.64
C) CO = 6.3 L/min; CI = 3.7 L/min/M2 SV = 64 mL/beat;
SVI = 37 mL/beat/M2 BP = 90/56 mm Hg SvO2 = 0.75
D) CO = 3.8 L/min; CI =2.3 L/min/M2 SV = 73 mL/beat; SVI
= 43 mL/beat/M2 BP = 100/58 mm Hg SvO2 = 0.72
Answer #4
Which of the combined set of hemodynamic values is of
greatest concern?
A) CO = 6.9 L/min; CI = 3.8 L/min/M2 SV = 63 mL/beat;
SVI = 34 mL/beat/M2 BP = 102/52 mm Hg SvO2 = 0.83
B) CO = 4.3 L/min; CI = 2.5 L/min/M2 SV = 43 mL/beat;
SVI = 25 mL/beat/M2 BP = 94/62 mm Hg SvO2 = 0.64
C) CO = 6.3 L/min; CI = 3.7 L/min/M2 SV = 64 mL/beat;
SVI = 37 mL/beat/M2 BP = 90/56 mm Hg SvO2 = 0.75
D) CO = 3.8 L/min; CI =2.3 L/min/M2 SV = 73 mL/beat; SVI
= 43 mL/beat/M2 BP = 100/58 mm Hg SvO2 = 0.72
Question #5
Immediate treatment of pulmonary artery
rupture may include all of the following
except:
A) Discontinuation of anticoagulation
B) Placing patient in lateral position with
unaffected side down.
C) Selective bronchial intubation
D) PEEP
Answer #5
Immediate treatment of pulmonary artery
rupture may include all of the following
except:
A) Discontinuation of anticoagulation
B) Placing patient in lateral position with
unaffected side down.
C) Selective bronchial intubation
D) PEEP
E) Hire a lawyer