Care of the Pediatric Patient with Gastrointestinal Problems
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Transcript Care of the Pediatric Patient with Gastrointestinal Problems
Care of the Pediatric
Patient with
Gastrointestinal Problems
Elizabeth Allen RN, MSN
Learning Objectives
1. Review the normal structure and function
of the GI system, including the pediatric
anatomical differences.
2. Review the pharmacological and surgical
measures utilized for the various GI
conditions
3. Recognize dehydration with appropriate
interventions.
4. Select appropriate diet for Celiac disease.
Anatomic and Physiologic
Differences
Newborn swallowing reflex until 6 weeks
Newborn stomach small with increased peristalsis
4-6 months- Insufficient enzymes to aid digestion
First 2 years- kidneys less efficient regulation of acidbase balance, less able to concentrate urine
Infants & Children- greater body surface area
Urine output
Infants 2 cc/kg/hr.
Children 1 cc/kg/hr.
Dehydration
Infants and young
children more
susceptible
Physiologic differences
Phototherapy or
overhead warmer
Insensible losses:
Increased respiratory
rate, fever
Vomiting and diarrhea
Drainage or blood loss
Body Weight Composed
of Water (Especially
extracellular fluid)
Newborn- 75% total
body water
Infant- 65% total body
water
Child/Adolescent- 50%
total body water
Dehydration Assessment
Level of Consciousness
Thirst
Urine output- weigh diapers, Foley
Urine Specific Gravity (>2 years)
Skin Turgor
Fontanelle
HR, BP
Capillary Refill
Skin color, temperature
Degrees of Dehydration
Mild
% of Body
Weight Lost
<5%
Moderate
Severe
6-9%
10% or
more
Urine Output
Usual
<1 ml/kg/hr.
Very
decreased or
absent
Fluid
Replacement
Oral
Oral/IV
IV
IV Fluid bolus with isotonic crystalloid only!!
Pediatric Fluid resuscitation with 20 ml/kg
Degrees of Dehydration
Mild
Moderate
Severe
Level of
Consciousness
Alert
Lethargic, Sleepy, Irritable
(Infants)
Alert, restless (older children)
Lethargy, unresponsiveness (Infant)
Conscious, anxious (older children)
Mucus
Membranes
Moist
dry
Parched, non elastic skin turgor
Normal
Dark and diminished
Markedly decreased or absent
No change
Increased HR BP normal or
slightly decreased
Increased HR, Decreased BP
Warm, Cap
Refill <2 sec
Capillary refill >2 sec
Cool, Discolored
Cap Refill >3-4 sec
No thirst
Thirsty
Greatly Increased unless lethargic
Normal
Slightly sunken, decreased
tears
Sunken, decreased or absent tears
Flat
Sunken
Sunken
Urine
LOC
Extremities
Thirst
Eyes
Fontanelle
(London et al., 2014)
Dehydration
What type of acid-base disturbance
would you expect from prolonged
vomiting and diarrhea?
Respiratory Acidosis
Respiratory Alkalosis
Metabolic Acidosis
Metabolic Alkalosis
Abnormal Motility
Constipation
Functional Constipation
Normal G&D, Increase fluids, fiber, May
resolve as solid food is added to diet
May treat pharmacologically with
osmotic agent (polyethylene glycol)
Abnormal Motility
Hirshprung Disease
Congenital Aganglionic Megacolon
Lack of peristalsis
Rectum and colon
More often in males
Abnormal Motility
Hirshprung Disease
Symptoms
Abdominal Distention
Failure to pass meconium in first 48
hours
Treatment
Chronic progressive constipation
Bloody diarrhea
Enterocolitis as complication- life
threatening emergency
Abnormal Motility
Hirshprung Disease
Treatment
Colectomy of aganglionic section
Primary Repair
Placement of temporary colostomy
Assess for Enterocolitis!
Colostomy
Teach proper care to parents
Monitor for fluid loss
Post op fecal incontinence or constipation may
persist
Abnormal Motility
GER/GERD
GER affects approximately 50%
infants 0-3 months
More common in
Asthma
Neurological Disorders
CF
Abnormal Motility
GERD Symptoms
Weight loss or no gain
Irritable
Vomiting
GERD Treatment
H2 blocker
PPI
Pro-kinetic agents
Frequent URI’s
Upright after feeds- 30
minutes
Positive pH probe study
Small frequent feeds
Thickened formula
Gastostomy tube
placement
Fundoplication
Fundoplication
Obstruction
Esophageal Atresia
Defect occurs in 4th-5th
week gestation
90% with Esophageal
Atresia also have TEF
Esophagus ends as dry
pouch or connects to
trachea with fistula
May have associated
defects
Tracheoesophageal
Fistula (TEF)
Defect occurs in 4th-5th
week gestation
Allows oral secretions,
liquids entry to lungs
Obstruction
Treatment:
Pre- op
NPO, position
Esophageal
Fistula and TEF
3 Classic signs in Infant:
Cyanosis
Choking
Coughing
Excessive Drooling,
aspiration, abdominal
distention (air)
Suction
NGT insertion, IV
hydration and antibiotics
Surgery ASAP
Ligate fistula,
insert
gastrostomy tube
Anastomosis of
esophagus
May be a 2 step
process
Obstruction
Pyloric Stenosis
Hypertrophy of pyloris muscle causes stenosis
Difficult passage from stomach to duodenum
More common in males (4:1)
Unknown cause
Symptoms begin 2-8 weeks after birth
Projectile vomiting immediately after feeds
Hungry, irritable, unable to gain weight
May present with dehydration, metabolic alkalosis
Olive sized mass R upper quadrant
Obstruction
Pyloric Stenosis
Treatment
Fluid replacement
Correct abnormal
fluid balance,
electrolytes
Surgical repairLaparoscopic
Pyloromyotomy
Obstruction
Intussusception
Telescoping of
intestine into itself
65% in infants <1
year
More common in
males
Obstruction
Intussusception
Symptoms
Intussusception
Treatment
Abrupt onset
Air enema- works 90%
of cases
Acute abdominal pain
Intermittent pain
Vomiting
Currant jelly stools- mix
of blood and mucus
May have palpable
abdominal mass
Then Barium enema
NGT
Surgical repair, removal
of necrotic tissue
Obstruction
Multidisciplinary
approach
Cleft Lip and Palate
Lip repair to
improve feeding
Can occur together or
separately
Defects occurs at 5-12
weeks gestation
Often associated with
other defects or
syndromes
Prone to otitis media
Speech problems
Obstruction
Cleft Lip and
Palate
Important to assess
palate in newborn
Cleft lip repair in first
weeks to 6 months of
life
Cleft palate repair at
approximately 18
months
Obstruction
Cleft Lip and Palate
Pre Op Nursing
Care
Feedings
Hydration
Prevent
aspiration
Post Op Repair
No sucking for 2
weeks
Nutrition
Elbow restraints
Pain control
Abnormal Absorption/Metabolism
Celiac Disease
Intolerance to gluten
Accumulate amino acid glutamine toxic to mucosal
cells and villi
First affects fat absorption
Steatorrhea- Large amounts of foul smelling, greasy stools
Symptoms arise 6 months- 2 years with solid food
introduction- may not be diagnosed until adulthood
Diagnosis
Fecal fat content
Duodenal or jejeunal biopsy
Gluten free diet- Diagnostic & Treatment
Abnormal Absorption/Metabolism
Pin Worms
Intestinal parasitic disorder- most common worm
infestation in US
Untreated water
Hand to mouth
Symptoms
Itching
Restless
Clinical Therapy
Mebendazole, pyrantel pamoate, albendazole
Household containment
Abnormal Absorption/Metabolism
Failure to Thrive (FTT)
Organic versus Non-organic
Symptoms
Persistent failure to eat
No weight gain/or weight loss
Small size
Irritable
Possible developmental delays
Management
Assessment
Growth
Home
Referrals