Transcript CHAPTER 8
CHAPTER 11
Parvovirus
Definitions of the virus:
Parvoviruses are the causative
agents of several important animal
diseases.
They require cells that are passing
through mitotic S phase to replicate
their DNA.
Parvovirus infections are most
severe in fetuses and neonates.
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Definitions of the virus:
This requirement for mitotic active
cells reflected in the tropism of
parvoviruses for rapidly dividing
hemopoietic precursors and
lymphocytes, and progenitor cells
of the intestinal mucosal lining.
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Definitions of the virus:
The family Parvoviridae comprises
two subfamilies: Paroviridae
(vertebrates).
The genus Parvovirus includes
feline panleukopenia virus and
closely related canine parvovirus,
porcine and chicken parvoviruses.
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Definitions of the virus:
The genus Erythrovirus includes
human parvovirus B19 and related
viruses of non-human primates,
bovine parvovirus type 3 and
chipmunk parvovirus.
The genus Dependovirus includes
adeno-associated viruses that are
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Definitions of the virus:
unable to replicate except in the
presence of a helper virus, an
adenovirus; goose and duck
parvoviruses; bovine parvovirus 2.
The genus Amadovirus includes
Aleutian mink disease virus; The
genus Bocavirus includes bovine
parvovirus and canine minute virus.
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INTRODUCTION
Parvovirus virions are nonenveloped, 25 nm in diameter, and
have icosahedral symmetry.
The capsid displays a hollow
cylinder at each fivefold axis of
symmetry that is surrounded by a
circular depression, prominent
protrusions around the threefold
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axis of symmetry and a depression
at each twofold axis of symmetry.
The capsid is composed of VP2 (90%)
and approximately 10% being the
overlapping but larger VP1 protein.
VP1 and VP2 are formed by
alternative splicing of the same
mRNA.
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The genome consists of a linear singlestranded DNA (4.5-5.5 kbp).
Individual virions of these viruses contain
single-stranded DNA of either polarity.
The genome contains two ORFs: an ORF in
the 3’ half of the genome that encodes the
non-structural proteins for DNA
transcription and replication; another ORF
towards 5’ half encodes the structural
proteins (CAP, VP, or S) of the capsid.
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The entire sequence of VP2 is
encoded within the VP1 gene.
A third structural protein, VP3, is
formed by cleavage of a peptide
from the amino terminus of VP2.
Parvoviruses are extremely stable to
environmental conditions such as
heat, pH, and disinfectant.
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The genome has terminal palindromic
sequences, enabling each end to form
hairpin or other complex base-paired
structures.
Transferrin receptor is the receptor for
canine parvovirus and feline panleukopenia
virus, and it directs the virus into the
clathrin-mediated uptake pathway.
Utilization of the transferrin receptor
facilitates replication of these viruses which
is upregulated on proliferating cells.
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Parvovirus replication occurs only in cells
that pass through mitotic S phase.
Parvovirus binds to sialic acid residues,
consistent with their ability to
hemagglutinate erythrocytes of other
species.
Virions traffic through the endosomal
pathways within the early and late
endosomes.
The VP1 protein contains a phospholipase
A2 enzymes in its N-terminal region.
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The non-structural protein (NS1) that is
encoded in the 3’ portion of the genome
serves (1) attached to the 5’ end of the viral
DNA during replication; (2) serves as a
helicase during replication and DNA
packaging; (3) serves as a site-specific
nickase; (4) mediates arrest of the cell in the
G1 phase.
A rolling-hairpin replication: the 3’-terminal
hairpin on the negative-sense DNA genome
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serves as a rolling-hairpin replication.
A rolling-hairpin replication: the 3’-terminal
hairpin on the negative-sense DNA genome
serves as a self-primer for the initiation of
synthesis of a double-stranded DNA
replicative intermediate.
The detection of a dimeric form of the
replicative intermediate, a head-to-head
concatemer of two covalently linked doublestranded forms- has led to a model in which
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the growing DNA strand replicates back on
itself to produce a tetrameric form.
The detection of a dimeric form of the
replicative intermediate, a head-to-head
concatemer of two covalently linked doublestranded forms- has led to a model in which
the growing DNA strand replicates back on
itself to produce a tetrameric form from
which two complete positive or negative
strands are generated.
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Parvovirus infections of the fetus or
newborn at critical stages of organogenesis
when there is considerable cell divisions
may result in developmental defects.
Replication of parvovirus is restricted in
hemopoietic precursors, lymphocytes, and
progenitor cells of intestinal mucosa of
older animals.
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Parvovirus infections of the fetus or
newborn at critical stages of organogenesis
when there is considerable cell divisions
may result in developmental defects.
Replication of parvovirus is restricted in
hemopoietic precursors, lymphocytes, and
progenitor cells of intestinal mucosa of
older animals.
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Many parvoviruses cause acute infections
that last only a few days, others persist for
long periods in the faeces of apparently
robust host immune responses.
Disease develops in persistently infected
mink as a result of the high levels of
circulating antigen-antibody complexes that
deposit in tissues and initiate a type III
hypersensitivity reaction that results in
tissue injury and destruction.
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Circovirus
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DEFINITION
The family Circoviridae includes
viruses with circular single-stranded
DNA genomes, and which share
common physicochemical and
genomic properties.
The family contains two genera
(Circovirus and Gyrovirus).
Circovirus uses an ambisense genome
strategy, with viral genes in different
orientations.
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DEFINITION
The genus Circovirus includes beak
and feather disease virus, canary
circovirus, goose circovirus, pigeon
circovirus, and porcine circoviruses 1
and 2.
Chicken anemia virus is the type
member of the genus Gyrovirus, in
which the viral genes are all in the
same orientation.
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They are small (approximately 20-25
nm in diameter), non-enveloped,
spherical in outline, with T=1
icosahedral symmetry.
Virions are made up of 60 capsid
subunits that package the viral circular
single-stranded DNA.
Chicken anemia virus has 12 trumpetlike structures that are less obvious in
the other circoviruses.
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Mature virions appear in diagnostic
specimens and in linear “strings of
pearls” in cell-free diagnostic
specimens.
Beak and feather disease virus,
porcine circovirus 1 and 2, and the
other members of the genus Circovirus
utilize an ambisense transcription
strategy-some genes are encoded in
the viral sense DNA and others in the
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Mature virions appear in diagnostic
specimens and in linear “strings of
pearls” in cell-free diagnostic
specimens.
Beak and feather disease virus,
porcine circovirus 1 and 2, and the
other members of the genus Circovirus
utilize an ambisense transcription
strategy-some genes are encoded in
the viral sense DNA and others in the
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complementary strand.
Beak and feather disease virus has
three ORFs and porcine circovirus has
four; in each case there is one major
capsid protein.
The genes of chicken anemia virus are
all encoded in the complementary
positive-sense DNA strand that is
transcribed to give a single
polycistronic transcript.
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Chicken anemia virus has three ORFs;
one encodes the VP1 (present in
virions); another virus-encoded VP3,
apoptin, induces apoptosis of T
lymphocytes.
These viruses are stable ; not
inactivation by heating.
CAV hemagglutinate RBC and binds to
sialic acid on the cell surface.
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Viral DNA replication occurs in the
nucleus and requires cellular proteins
and other components produced
during the S phase.
Replication of the genome occurs via a
rolling circle that originates at a stemloop structure.
The viral replication is maximized in
actively dividing cells in young animals.
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Viral DNA replication occurs in the
nucleus and requires cellular proteins
and other components produced
during the S phase.
Replication of the genome occurs via a
rolling circle that originates at a stemloop structure.
The viral replication is maximized in
actively dividing cells in young animals.
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Replication of porcine circovirus is
enhanced during periods of immune
stimulation that result in proliferation
of lymphocytes in which virus can
replicate.
CAV replication in the oviduct may be
regulated by estrogen, particularly
during egg laying, to allow more
efficient vertical transmission.
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Replication of porcine circovirus is
enhanced during periods of immune
stimulation that result in proliferation
of lymphocytes in which virus can
replicate.
CAV replication in the oviduct may be
regulated by estrogen, particularly
during egg laying, to allow more
efficient vertical transmission.
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The apoptin causes destruction of
infected lymphocytes and promotes a
relative immune suppression that
favors virus persistence.
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