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Transcript midterm to final review
EMERGENCY
MEDICAL
TECHNICIAN
FINAL REVIEW
Barry Barkinsky
EMS-I, Paramedic
Medical Emergencies
Respiratory
Common Problems
Signs and Symptoms
Adequate / Inadequate
Treatment
Obstructive Lung Disease
Types
Emphysema
Chronic Bronchitis
Asthma
Causes
Genetic Disposition
Smoking & Other Risk Factors
Emphysema
Pathophysiology
Exposure to Noxious Substances
Exposure results in the destruction of the walls of the
alveoli.
Weakens the walls of the small bronchioles and results
in increase residual volume.
Increased Risk of Infection and Dysrhythmia
Emphysema
Assessment
History
Recent weight loss, dyspnea with exertion
Cigarette and tobacco usage
Lack of Cough
Emphysema
Assessment
Physical Exam
Barrel chest.
Prolonged
expiration and
rapid rest phase.
Thin.
Pink skin due to
extra red cell
production.
Hypertrophy of
accessory muscles.
“Pink Puffers.”
RESPIRATORY
Chronic Bronchitis
Pathophysiology
Results from an increase in mucus-secreting cells in
the respiratory tree.
Alveoli relatively unaffected.
Decreased alveolar ventilation.
Assessment
History
Frequent respiratory infections.
Productive cough.
Chronic Bronchitis
Physical Exam
Often
overweight.
Rhonchi present
on auscultation.
Jugular vein
distention.
Ankle edema.
Hepatic
congestion.
“Blue Bloater.”
RESPIRATORY
Bronchitis & Emphysema
Management
Maintain airway.
Support breathing.
Find position of comfort.
Monitor oxygen saturation.
Be prepared to ventilate.
Administer medications.
Bronchodilators.
Asthma
Pathophysiology
Chronic Inflammatory Disorder
Results in widespread but variable air flow
obstruction.
The airway becomes hyperresponsive.
Induced by a trigger, which can vary by individual.
Trigger causes release of histamine, causing
bronchoconstriction and bronchial edema.
Asthma
Assessment
Identify immediate threats.
Obtain history.
SAMPLE & OPQRST History
History of asthma-related hospitalization?
History of respiratory failure/ventilator use?
Asthma
Physical Exam
Presenting signs may include dyspnea, wheezing,
cough.
Wheezing is not present in all asthmatics.
Speech may be limited to 1–2 consecutive words.
Look for hyperinflation of the chest and
accessory muscle use.
Carefully auscultate breath sounds.
Asthma
Management
Treatment goals:
Correct hypoxia.
Reverse bronchospasm.
Reduce inflammation.
Maintain the airway.
Support breathing.
High-flow oxygen or assisted ventilations as indicated.
RESPIRATORY
(Trauma)
Medical Emergencies
Cardiac Compromise
Cardiac Emergencies
Signs and Symptoms
Treatment
Managing Specific Cardiovascular
Emergencies
Angina Pectoris
Myocardial Infarction
Heart Failure
Hypertensive Emergencies
Cardiogenic Shock
Cardiac Arrest
Peripheral Vascular and Other Cardiovascular
Emergencies
Angina Pectoris
Causes of Chest Pain
Cardiovascular, including acute coronary syndrome, or
thoracic dissection of the aorta
Respiratory, including pulmonary embolism,
pneumothorax or pneumonia.
Gastrointestinal, including pancreatitis, hiatal hernia,
esophageal disease, gastroesophageal reflux, peptic ulcer
disease.
Musculoskeletal, chest wall trauma.
Angina Pectoris
Field Assessment
Signs of Shock
Chest Discomfort
Typically sudden onset, which may radiate or be
localized to the chest.
Patient often denies chest pain.
Duration
Episodes last 3–5 minutes.
Pain relieved with rest and/or nitroglycerin.
Angina Pectoris
Breathing
History
Past episodes of angina:
Episodes of angina that are increasing in frequency,
duration, or severity are significant.
Angina Pectoris
Management
Relieve anxiety:
Place the patient in a position of physical and emotional comfort.
Administer oxygen.
Consider medication administration:
Nitroglycerin tablets or spray
Angina Pectoris
Special Considerations
Patients with new-onset often require hospitalization.
Symptoms not relieved by rest, nitroglycerin, and
oxygen may indicate an overall worsening of the
disease or the early stages of a myocardial infarction.
Patients may refuse transport after pain is relieved,
even though the underlying problem is not addressed.
Myocardial Infarction
Pathophysiology
Death and necrosis of
heart muscle due to
inadequate oxygen supply.
Causes may include
occlusion, spasm, acute
volume overload,
hypotension, acute
respiratory failure, and
trauma.
Location and size
dependent on the vessel
involved.
Myocardial Infarction
Effects of a Myocardial Infarction
Dysrhythmias
Heart Failure
Goals of Treatment
Pain Relief
Reperfusion
Myocardial Infarction
Field Assessment
Breathing
Signs of Shock
Chief Complaint
Typically related to chest pain.
Evaluate using OPQRST:
Discomfort > 30 minutes.
Radiation to arms, neck, back, or epigastric region.
Patients may minimize symptoms.
Feelings of “impending doom.”
Myocardial Infarction
Other Symptoms
Nausea and vomiting
Diaphoresis
Myocardial Infarctions & the ECG
Dysrhythmias:
VF, VT, Asystole, PEA.
Dysrhythmias are the leading cause of death in MI.
Myocardial Infarction
Management
Transport
Rapid transport indicated when acute MI suspected
Prehospital
Administer oxygen.
Consider medication administration:
Aspirin
Nitroglycerin
Nitroglycerine
Indications
Contraindications
Side effects
Dosage
Heart Failure
Left Ventricular
Failure
Pathophysiology
Results in
increased back
pressure into
the pulmonary
circulation.
Heart Failure
Right Ventricular
Failure
Pathophysiology
Results in
increased back
pressure into the
systemic venous
circulation.
Pulmonary
Embolism
Heart Failure
Congestive Heart Failure
Pathophysiology
Reduction in the heart’s stroke volume causes fluid
overload throughout the body’s other tissues.
Heart Failure
Field Assessment
Pulmonary Edema:
Cough with copious amounts of clear or pink-tinged sputum.
Labored breathing, especially with exertion.
Abnormal breath sounds, including rales, rhonchi, and wheezes.
Paroxysmal Nocturnal Dyspnea (PND)
Medications:
Diuretics.
Medications to increase cardiac contractile force.
Home oxygen.
Heart Failure
Mental Status
Breathing
Mental status changes indicate impending respiratory failure.
Signs of labored breathing.
Tripod positioning.
“Number of pillows.”
Skin
Color changes.
Peripheral and/or sacral edema.
Heart Failure
Management
General management:
Avoid supine positioning.
Avoid exertion such as standing or walking.
Maintain the airway.
Administer oxygen.
Avoid patient refusals if at all possible.
Hypertensive Emergencies
Hypertensive Emergency
Causes
Typically occurs only in patients with a history of
HTN.
Primary cause is noncompliance with prescribed
antihypertensive medications.
Also occurs with toxemia of pregnancy.
Risk Factors
Age-related factors
Race-related factors
Hypertensive Emergencies
Field Assessment
Initial Assessment
Alterations in mental state
Signs & Symptoms
Headache accompanied by nausea and/or vomiting
Blurred vision
Shortness of breath
Epistaxis
Vertigo
Hypertensive Emergencies
History
Known history of hypertension
Compliance with medications
Exam
BP > 160/90
Signs of left ventricular failure
Strong, bounding pulse
Abnormal skin color, temperature, and condition
Presence of edema
Hypertensive Emergencies
Management
Maintain airway.
Administer oxygen.
Cardiogenic Shock
Pathophysiology
General
Inability of the heart to meet the body’s metabolic needs.
Often remains after correction of other problems.
Severe form of pump failure.
High mortality rate.
Causes
Tension pneumothorax and cardiac tamponade.
Impaired ventricular emptying.
Impaired myocardial contractility.
Trauma.
Cardiogenic Shock
Field Assessment
Initial Assessment
Chief Complaint
Chief complaint is typically chest pain, shortness of
breath, unconsciousness, or altered mental state.
Onset may be acute or progressive.
History
History of recent MI or chest pain episode.
Presence of shock in the absence of trauma.
Cardiogenic Shock
Mental Status
Airway and Breathing
Restlessness progressing to confusion
Dyspnea, labored breathing, and cough
PND, tripod position, accessory muscle retraction, and
adventitious lung sounds
Circulation
Hypotension
Cool, clammy skin
Cardiogenic Shock
Management
Maintain airway.
Administer oxygen
Identify and treat underlying problem.
Cardiac Arrest
Sudden Death
Causes
Electrolyte or acid–base imbalances
Electrocution
Drug intoxication
Hypoxia
Hypothermia
Pulmonary embolism
Stroke
Drowning
Trauma
Cardiac Arrest
Field Assessment
Initial Assessment
ECG
Unresponsive, apneic, pulseless patient
Dysrhythmias
History
Prearrest events
Bystander CPR
“Down time”
Cardiac Arrest
Management
Resuscitation
Return of Spontaneous Circulation
Role of Basic Life Support
General Guidelines
Manage specific Dysrhythmias.
AED
CPR.
AED
(Automatic External
Defibrillator)
AED
(Automatic External
Defibrillator)
Indications
AED
(Automatic External
Defibrillator)
Contraindications
AED
(Automatic External
Defibrillator)
Joules
AED
(Automatic External
Defibrillator)
# of Shocks
AED
(Automatic External
Defibrillator)
If NO SHOCK
Advised
Peripheral Vascular and Other
Cardiovascular Emergencies
Aneurysm
Pathophysiology
Ballooning of an arterial wall, usually the aorta, that results from
a weakness or defect in the wall
Types
Atherosclerotic
Dissecting
Traumatic
Peripheral Vascular and Other
Cardiovascular Emergencies
Abdominal Aortic
Aneurysm
Often the result of
atherosclerosis
Signs and symptoms
Abdominal pain
Back/flank pain
Hypotension
Urge to defecate
Peripheral Vascular and Other
Cardiovascular Emergencies
Dissecting Aortic Aneurysm
Caused by degenerative changes in the smooth
muscle and elastic tissue.
Blood gets between and separates the wall of the
aorta.
Can extend throughout the aorta and into
associated vessels.
Peripheral Vascular and Other
Cardiovascular Emergencies
Acute Pulmonary Embolism
Pathophysiology
Blockage of a pulmonary artery by a blood clot or
other particle.
The area served by the pulmonary artery fails.
Signs and Symptoms
Dependent upon size and location of the blockage.
Onset of severe, unexplained dyspnea.
History of recent lengthy immobilization.
Medical Emergencies
Altered Mental Status (AMS)
Causes
Treatment
Medical Emergencies
Diabetes
Most common cause
Signs and Symptoms
Treatment
Medical Emergencies
Seizures
Seizures
Generalized Seizures
Tonic-Clonic
Aura
Loss of Consciousness
Tonic Phase
Clonic Phase
Postseizure
Postictal
Seizures
Partial Seizures
Simple Partial Seizures
Involve one body area.
Can progress to generalized seizure.
Complex Partial Seizures
Characterized by auras.
Typically 1–2 minutes in length.
Loss of contact with surroundings.
Seizures
Assessment
Differentiating Between Syncope & Seizure
Bystanders frequently confuse syncope and seizure.
Seizures
Patient History
History of Seizures
History of Head Trauma
Any Alcohol or Drug Abuse
Recent History of Fever, Headache, or Stiff Neck
History of Heart Disease, Diabetes, or Stroke
Current Medications
Phenytoin (Dilantin), phenobarbitol, valproic acid (Depakote), or
carbamazepine (Tegretol)
Physical Exam
Signs of head trauma or injury to tongue, alcohol or drug abuse
Seizures
Management
Scene safety & BSI.
Maintain the airway.
Administer high-flow
oxygen.
Treat hypoglycemia if
present.
Do not restrain the
patient.
Protect the patient
from the environment.
Maintain body
temperature.
Seizures
Management
Position the patient.
Suction if required.
Provide a quiet
atmosphere.
Transport.
Seizures
Status Epilepticus
Two or More Generalized Seizures
Seizures occur without a return of consciousness.
Management
Management of airway and breathing is critical.
Monitor the airway closely.
Medical Emergencies
Stroke (CVA)
Stroke & Intracranial Hemorrhage
Occlusive Strokes
Embolic & Thrombotic Strokes
Hemorrhagic Strokes
Stroke & Intracranial Hemorrhage
Signs
Facial Drooping
Headache
Aphasia/Dysphasia
Hemiparesis
Paresthesia
Gait Disturbances
Incontinence
Symptoms
Confusion
Agitation
Dizziness
Vision Problems
Stroke & Intracranial Hemorrhage
Transient Ischemic Attacks
Indicative of carotid artery disease.
Symptoms of neurological deficit:
Symptoms resolve in less than 24 hours.
No long-term effects.
Evaluate through history taking:
History of HTN, prior stroke, or TIA.
Symptoms and their progression.
Stroke & Intracranial Hemorrhage
Management
Scene safety & BSI
Maintain the airway.
Support breathing.
Obtain a detailed history.
Position the patient.
Protect paralyzed extremities.
Medical Emergencies
Allergic Reaction
(Anaphylaxis)
Allergies and Anaphylaxis
Allergic Reaction
An exaggerated response by the immune system
to a foreign substance
Anaphylaxis
An unusual or exaggerated allergic reaction
A life-threatening emergency
Anaphylaxis
Causes
Assessment Findings
in Anaphylaxis
Focused History & Physical Exam
Focused History
SAMPLE & OPQRST History
Rapid onset, usually 30–60 seconds following exposure.
Speed of reaction is indicative of severity.
Previous allergies and reactions.
Physical Exam
Presence of severe respiratory difficulty is key to
differentiating anaphylaxis from allergic reaction.
Assessment
Findings in
Anaphylaxis
Physical Exam
Facial or laryngeal edema
Abnormal breath sounds
Hives and urticaria
Hyperactive bowel sounds
Vital sign deterioration as
the reaction progresses
Management of Allergic Reactions
Scene safety
Protect the airway.
Support breathing.
Establish IV access.
Administer
medications:
Epinephrine
Epi-Auto Injector
Indications
Epi-Auto Injector
Contraindications
Epi-Auto Injector
Dosage
Epi-Auto Injector
Actions
Epi-Auto Injector
Side Effects
Epi-Auto Injector
Administration
Medical Emergencies
Poisons and Overdose
How they enter the body
Treatment
Environmental
Heat
Cold
Water Emergencies
Trauma Emergencies
Bleeding
Bleeding
External Types,
Treatment ( In order)
Hemorrhage Classification
Capillary
Slow, even flow
Venous
Steady, slow flow
Dark red
Arterial
Spurting blood
Pulsating flow
Bright red color
Bleeding
Internal, S/S,
Treatment
Hemorrhage Control
Internal Hemorrhage
Hematoma
Pocket of blood between muscle and fascia
Humerus or Tibia/Fibula fracture: 500-750mL
Femur fracture: 1,500mL
UNEXPLAINED SHOCK is BEST attributed
to abdominal trauma
General Management
Immobilization, Stabilization, Elevation
Hemorrhage Control
Internal Hemorrhage
Epistaxis: Nose Bleed
Causes: Trauma, Hypertension
Treatment: Lean forward, pinch nostrils
Hemoptysis
Esophageal Varices
Chronic Hemorrhage
Anemia
Trauma Emergencies
Shock
In a Nutshell…..
SHOCK is…
INADEQUATE
TISSUE
PERFUSION
Stages of Shock
Compensated Shock
Decompensated Shock
Minimal Change
System beginning to fail
Irreversible Shock
Ischemia and death imminent
Etiology of Shock
Hypovolemic Shock
Loss of blood volume
Distributive Shock
Prevent appropriate
distribution of nutrients and
removal of wastes
Anaphylactic
Septic
Hypoglycemia
Obstructive Shock
Interference with the blood
flowing through the
cardiovascular system
Tension Pneumothorax
Cardiac Tamponade
Pulmonary Emboli
Cardiogenic Shock
Pump failure
Respiratory Shock
Respiratory system not able to
bring oxygen into the alveoli
Airway obstruction
Pneumothorax
Neurogenic Shock
Loss of nervous control from
CNS to peripheral vasculature
Trauma Emergencies
Soft Tissue
Introduction to
Soft-Tissue Injury
Skin is the largest, most important organ
16% of total body weight
Function
Protection
Sensation
Temperature Regulation
AKA: Integumentary System
Introduction to
Soft-Tissue Injury
Epidemiology
Open Wounds
Over 10 million wounds present to ED
Most require simple care and some suturing
Up to 6.5% may become infected
Closed Wounds
More Common
Contusions, Sprains, Strains
A&P of Soft Tissue Injuries
Skin Layers
Epidermis
Outermost layer
Helps prevent infection
Dermis
Upper Layer (Papillary Layer)
Lower Layer (Reticular Layer)
Integrates dermis with SQ layer
Blood vessels, nerve endings, glands
Loose connective tissue, capillaries and nerves
Sebaceous & Sudoriferous Glands
Subcutaneous
Adipose tissue
Heat retention
Pathophysiology of
Soft-Tissue Injury
Closed Wounds
Contusions
Ecchymosis
Hematomas
Crush Injuries
Open Wounds
Abrasions
Lacerations
Incisions
Punctures
Impaled Objects
Avulsions
Amputations
Trauma Emergencies
Penetrating Injuries
Trauma Emergencies
Evisceration
Trauma Emergencies
Impaled Object
Trauma Emergencies
Amputation
Management of
Soft-Tissue Injury
Objectives of Wound Dressing & Bandaging
Hemorrhage Control
Direct Pressure
Elevation
Pressure Points
Consider
Ice
Constricting Band
Tourniquet
USE ALL COMPONENTS TOGETHER
Management of
Soft-Tissue Injury
Objectives of Wound Dressing & Bandaging
Sterility
Keep the wound as clean as possible
If wound is grossly contaminated consider cleansing
Immobilization
Prevents movement and aggravation of wound
Do not use an elastic bandage: TQ effect
Monitor distal pulse, motor, and sensation
(continued)
Management of
Soft-Tissue Injury
Pain & Edema Control
Cold packs
Moderate pressure over wound
Dressing & Bandage Materials
Sterile & Non-sterile Dressings
Occlusive/Non-occlusive Dressings
Adherent/Non-adherent Dressings
Adherent: stick to blood or fluid
Absorbent/Non-absorbent
Sterile: Direct wound contact
Non-sterile: Bulk dressing above sterile
Absorbent: soak up blood or fluids
Wet/Dry Dressings
Wet: Burns, postoperative wounds (Sterile NS)
Dry: Most common
Trauma Emergencies
Burns
Classification
Superficial
Partial-Thickness
Full-Thickness
Severity
Depth
Body Surface Area (BSA)
Burn Depth
Superficial Burn:
1st Degree Burn
Signs & Symptoms
Reddened skin
Pain at burn site
Involves only
epidermis
Burn Depth
Partial-Thickness
Burn: 2nd Degree
Burn
Signs & Symptoms
Intense pain
White to red skin
Blisters
Involves epidermis &
dermis
Burn Depth
Full-Thickness Burn:
3rd Degree Burn
Signs & Symptoms
Dry, leathery skin
(white, dark brown, or
charred)
Loss of sensation (little
pain)
All dermal layers/tissue
may be involved
Trauma Emergencies (Burns)
Rule of Nines (Adult)
Head and Neck:
9%
Each Upper Ext:
Anterior Trunk:
Posterior Trunk:
Each Lower Ext:
9%
18 %
18 %
18 %
Genitalia:
1%
Trauma Emergencies (Burns)
Rule of Nines
(Child)
Head and Neck:
18 %
Each Upper Ext:
Anterior Trunk:
Posterior Trunk:
Each Lower Ext:
Genitalia:
9%
18 %
18 %
14 %
1%
Trauma Emergencies
Burns
Rule of Palm
Location
Preexisting Medical Problems
Age
5 – 55
Source
Treatment
Rule of Palms
A burn equivalent to the size of the patient’s
hand is equal to 1% body surface area (BSA)
Pathophysiology of Burns
Types of Burns
Thermal
Electrical
Chemical
Radiation
Thermal Burns
Heat changes the molecular structure of
tissue
Denaturing (of proteins)
Extent of burn damage depends on
Temperature of agent
Concentration of heat
Duration of contact
Systemic Complications
Hypothermia
Hypovolemia
Disruption of skin and its ability to thermoregulate
Shift in proteins, fluids, and electrolytes to the burned
tissue
General electrolyte imbalance
Eschar
Hard, leathery product of a deep full thickness burn
Dead and denatured skin
Systemic Complications
Infection
Organ Failure
Special Factors
Greatest risk of burn is infection
Age & Health
Physical Abuse
Elderly, Infirm or Young
Assessment of Thermal Burns
General Signs & Symptoms
Pain
Changes in skin condition at
affected site
Adventitious sounds
Blisters
Sloughing of skin
Hoarseness
Burnt hair
Edema
Paresthesia
Hemorrhage
Other soft tissue injury
Musculoskeletal injury
Dyspnea
Chest pain
Assessment of Thermal Burns
Burn Severity
Minor
Superficial
Partial Thickness
Full Thickness
<50% BSA
<15% BSA
<2% BSA
Moderate
Superficial
Partial Thickness
Full Thickness
>50% BSA
>15% BSA
>2% BSA
Critical
Partial Thickness
>30% BSA
Full Thickness
Inhalation Injury
>10% BSA
Any partial or full thickness burn involving hands, feet, joints,
face, or genitalia
Management of
Thermal Burns
Local & Minor Burns
Local cooling
Partial thickness: <15% of BSA
Full thickness: <2% BSA
Remove clothing
Cool or Cold water immersion
Management of
Thermal Burns
Moderate to Severe Burns
Dry sterile dressings
Partial thickness: >15% BSA
Full thickness: >5% BSA
Maintain warmth
Prevent hypothermia
Consider aggressive fluid therapy
Moderate to severe burns
Management of
Thermal Burns
Moderate to Severe Burns
Caution for fluid overload
Frequent auscultation of breath sounds
Prevent infection
Management of
Thermal Burns
Inhalation Injury
Provide high-flow O2 by NRB
Consider intubation if swelling
Consider hyperbaric oxygen therapy
Assessment & Management of Electrical,
Chemical & Radiation Burns
Electrical Injuries
Safety
Turn off power
Energized lines act as whips
Establish a safety zone
Lightning Strikes
High voltage, high current, high energy
Lasts fraction of a second
No danger of electrical shock to EMS
Assessment & Management of Electrical,
Chemical & Radiation Burns
Chemical Burns
Scene size-up
Hazardous materials team
Establish hot, warm and cold zones
Prevent personnel exposure from chemical
Specific Chemicals
Phenol
Dry Lime
Sodium
Riot Control Agents
Assessment & Management of Electrical,
Chemical & Radiation Burns
Specific Chemicals
Phenol
Industrial cleaner
Alcohol dissolves Phenol
Irrigate with copious amounts of water
Dry Lime
Strong corrosive that reacts with water
Brush off dry substance
Irrigate with copious amounts of cool water
Prevents reaction with patient tissues
Assessment & Management of Electrical,
Chemical & Radiation Burns
Riot Control Agents
Agents
Irritation of the eyes, mucous membranes, and respiratory
tract.
No permanent damage
General Signs & Symptoms
CS, CN (Mace), Oleoresin, Capsicum (OC, pepper spray)
Coughing, gagging, and vomiting
Eye pain, tearing, temporary blindness
Management
Irrigate eyes with normal saline
Assessment & Management of Electrical,
Chemical & Radiation Burns
Radiation Burns
Notify Hazardous Materials Team
Establish Safety Zones
Hot, Warm, & Cold
Personnel positioned Upwind and Uphill
Decontaminate ALL rescuers, equipment and
patients
Musculoskeleta
l System
Pathophysiology of the
Musculoskeletal System
Joint Injury
Sprain
Subluxation
Dislocation
Bone Injury
Open Fracture
Closed Fracture
Hairline Fracture
Impacted Fracture
Musculoskeletal
Ligament
Musculoskeletal
Tendon
Pathophysiology of the
Musculoskeletal System
Pediatric Considerations
Geriatric Considerations
Flexible nature
Osteoporosis
Pathological Fractures
Pathological diseases
Pathophysiology of the
Musculoskeletal System
General Considerations with musculoskeletal
injuries
Neurological compromise
Decreased stability
Muscle spasm
Bone Repair Cycle
Osteocytes produce osteoblasts
Deposition of salts
Increasing strength of matrix
Musculoskeletal Injury
Management
General Principles
Protecting Open Wounds
Positioning the limb
Immobilizing the injury
Checking Neurovascular Function
Trauma Emergencies
Injuries
Painful, swollen, deformed extremities
Assessment
Signs and Symptoms
Splinting
Upper Extremities
Lower Extremities
Hip / Pelvis
Musculoskeletal Injury
Management
Splinting Devices
Rigid splints
Formable Splints
Soft Splints
Traction Splints
Other Splinting Aids
Vacuum Splints
Air Sprints
Cravats or Velcro Splints
Fracture Care
Joint Care
Muscular & Connective Tissue Care
Trauma Emergencies
Injuries to Head
Nervous System
Brain Injuries
Direct
Indirect
Patient Assessment
Signs and Symptoms
Neurological Assessment
Trauma Emergencies
Injuries to Spine
MOI
Assessment
Signs and Symptoms
Treatment
Immobilization
Helmets
Collars
LSB
Seated Patient
Musculoskeletal Injury
Management
Care for Specific Joint Injuries
Hip
Knee
Ankle
Foot
Shoulder
Elbow
Wrist/Hand
Finger
Joint Injuries
Alert for
neurological
Compromise
Triage
Command at
Mass-Casualty
Incidents
Incident Commander (IC)
Coordinates all scene activities
Also called Incident Manager (IM) or
Officer in Charge (OIC)
The first on-scene unit must assume command
and direct all rescue efforts at a mass-casualty
incident (MCI)
Singular vs. Unified Command
Singular command
One person coordinates the incident.
Most useful in smaller, single-jurisdictional incidents.
Unified command
Managers from different jurisdictions share
command.
Fire, EMS, law enforcement
Establishing Command
First arriving unit establishes command.
Assign command early in an incident.
Establish a command post.
EMS Branch Functions
Triage
Treatment
Transport
Triage
Sorting of patients based upon the severity of
their injuries
Primary triage
Secondary triage
Triage Tags
Alerts care providers to patient priority
Prevents re-triage of the same patient
Serves as a tracking system
The METTAG
Treatment
Red treatment unit
Yellow treatment unit
Green treatment unit
Triage
Priority 1
(RED)
Triage
Priority 2
(Yellow)
Triage
Priority 3
(Green)
OB / GYN
OB / GYN
Labor
Bloody Show
Crowning
Predelivery Emergencies
Labor
Stage One
(Dilation)
Stage Two
(Expulsion)
Stage Three
(Placental Stage)
Management of a Patient
in Labor
Transport the patient in labor unless delivery is
imminent.
Maternal urge to push or the presence of
crowning indicates imminent delivery.
Delivery at the scene or in the ambulance will be
necessary.
Field Delivery
Set up delivery area.
Give oxygen to mother and
start
Drape mother with toweling
from OB kit.
Monitor fetal heart rate.
As head crowns, apply gentle
pressure.
Suction the mouth and then
the nose.
Clamp and cut the cord.
Dry the infant and keep it
warm.
Deliver the placenta and
save for transport with the
mother.
OB / GYN ( Normal Delivery)
OB / GYN ( Normal Delivery)
OB / GYN ( Normal Delivery)
OB / GYN ( Normal Delivery)
OB / GYN ( Normal Delivery)
OB / GYN ( Normal Delivery)
Apgar Scoring
OB / GYN ( Normal Delivery)
Care of Newborn
OB / GYN
(Resuscitation)
HR Less than 100
OB / GYN
(Resuscitation)
HR less than 80
OB / GYN
(Resuscitation)
HR less than 60
Neonatal Resuscitation
If the infant’s respirations are below 30 per minute
and tactile stimulation does not increase rate to
normal range, assist ventilations using bag valve
mask with high-flow oxygen.
If the heart rate is below 80 and does not respond to
ventilations, initiate chest compressions.
Transport to a facility with neonatal intensive care
capabilities.
Causes of Bleeding
During Pregnancy
Abortion
Ectopic pregnancy
Placenta previa
Abruptio placentae
Abortion
Termination of pregnancy before the 20th
week of gestation.
Different classifications.
Signs and symptoms include cramping,
abdominal pain, backache, and vaginal
bleeding.
Treat for shock.
Provide emotional support.
Ectopic Pregnancy
Assume that any female of childbearing age
with lower abdominal pain is experiencing an
ectopic pregnancy.
Ectopic pregnancy is life-threatening.
Transport the patient immediately.
Placenta Previa
Usually presents with
painless bleeding.
Never attempt vaginal
exam.
Treat for shock.
Transport
immediately—
treatment is delivery
by
c-section.
Abruptio Placentae
Signs and symptoms
vary.
Classified as partial,
severe, or complete.
Life-threatening.
Treat for shock, fluid
resuscitation.
Transport left lateral
recumbent position.
Abnormal Delivery Situations
OB / GYN (Abnormal
Deliveries)
Breech
Breech Presentation
The buttocks or both feet present first.
If the infant starts to breath with its face
pressed against the vaginal wall, form a “V”
and push the vaginal wall away from infant’s
face. Continue during transport.
OB / GYN (Abnormal
Deliveries)
Prolapsed Cord
Prolapsed Cord
The umbilical cord precedes the fetal presenting part.
Elevate the hips, administer oxygen, and keep warm.
If the umbilical cord is seen in the vagina, insert two
gloved fingers to raise the fetus off the cord. Do not
push cord back.
Wrap cord in sterile moist towel.
Transport immediately; do not attempt delivery.
OB / GYN (Abnormal
Deliveries)
Limb Presentation
Limb Presentation
With limb presentation, place the
mother in knee–chest position,
administer oxygen, and transport
immediately. Do not attempt delivery.
Other Abnormal Presentations
Whenever an abnormal presentation or position of
the fetus makes normal delivery impossible, reassure
the mother.
Administer oxygen.
Transport immediately.
Do not attempt field delivery in these
circumstances.
Other Delivery Complications
OB / GYN (Abnormal
Deliveries)
Multiple Births
Multiple Births
Follow normal guidelines, but have additional
personnel and equipment.
In twin births, labor starts earlier and babies
are smaller.
Prevent hypothermia.
OB / GYN (Abnormal
Deliveries)
Meconium
Meconium Staining
Fetus passes feces into the amniotic fluid.
If meconium is thick, suction the
hypopharynx and trachea using an
endotracheal tube until all meconium has
been cleared from the airway.
Maternal Complications of
Labor and Delivery
Postpartum Hemorrhage
Defined as a loss of more than
500 cc of blood following delivery.
Treat for shock as necessary.
Follow protocols if applying antishock
trousers.
Uterine Rupture
Tearing, or rupture, of the uterus.
Patient complains of severe abdominal pain and will
often be in shock. Abdomen is often tender and
rigid.
Fetal heart tones are absent.
Treat for shock.
Give high-flow oxygen.
Transport patient rapidly.
Infants and Children
Airway
Maneuvers
FBAO
Adjuncts
Infants and Children
Trauma
Common Causes
Types
Shock
Causes
Assessment
Treatment
Anatomical and physiological considerations
in the infant and child.
a. In the supine position, an infant’s or child’s
larger head tips forward, causing airway
obstruction.
b. Placing padding under the patient’s back and
shoulders will bring the airway to a neutral or
slightly extended position.
General Approach to
Pediatric Assessment
Basic Considerations
Much of the initial patient assessment can be
done during visual examination of the scene.
Involve the caregiver or parent as much as
possible.
Allow to stay with child during treatment and
transport.
Scene Size-Up
Conduct a quick scene size-up.
Take BSI precautions.
Look for clues to mechanism of injury or nature
of illness.
Allow child time to adjust to you before
approaching.
Speak softly, simply, at eye level.
Suctioning
Decrease suction pressure to less than 100
mm/Hg in infants.
Avoid excessive suctioning time—less than 15
seconds per attempt.
Avoid stimulation of the vagus nerve.
Check the pulse frequently.
Inserting an oropharyngeal airway in a
child with the use of a tongue blade.
Ventilation
Avoid excessive bag pressure and volume.
Obtain chest rise and fall.
Allow time for exhalation.
Flow-restricted, oxygen-powered devices are
contraindicated.
Do not use BVMs with pop-off valves.
Apply cricoid pressure.
Avoid hyperextension of the neck.
Circulation
Two problems lead to cardiopulmonary
arrest in children:
Shock
Respiratory failure
Signs and symptoms of shock
(hypoperfusion) in a child.
Respiratory Emergencies
Infections
Upper airway distress
Croup
Epiglottitis
Lower airway distress
Asthma
Bronchiolitis
a. Croup and
b. Epiglottitis
Positioning of the child with epiglottitis.
Often there will be excessive drooling.
The child with epiglottitis should be
administered humidified oxygen and
transported in a comfortable position.
Poisoning and Toxic Exposure
Accidental poisoning is a common childhood
emergency.
Leading cause of preventable death in
children.
Medical Emergencies
Seizures
Trauma Emergencies
Falls
Motor vehicle crashes
Car vs. pedestrian injuries
Drowning and near drowning
Penetrating injuries
Burns
Physical abuse
Falls are the most common cause of
injury in young children.
A deploying airbag can propel a child safety seat
back into the vehicle’s seat, seriously injuring
the child secured in it.
Medical Emergencies
SIDS
Sudden Infant Death Syndrome
(SIDS)
SIDS is the sudden death of an
infant during the first year of life
from an illness of unknown etiology.
Child Abuse and
Neglect
The stigmata of child abuse.
Infants and Children with
Special Needs
Common home-care devices
Tracheostomy tubes
Apnea monitors
Home artificial ventilators
Central intravenous lines
Gastric feeding and gastrostomy tubes
Shunts
Medical Emergencies
Meningitis
Summary
Questions ?