EMS Equipment Review
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Transcript EMS Equipment Review
EMS Equipment
Review
MARCH 2015 CE
CONDELL MEDICAL CENTER EMS
SYSTEM CE
IDPH SITE CODE #107200E-1215
PREPARED BY: SHARON HOPKINS, RN, BSN, EMT-P
1
Objectives
2
Upon successful completion of this module, the EMS
provider will be able to:
List indications for use of a variety of EMS equipment
used in the field.
Manage a group of peers in setting up and applying
a variety of equipment used in the field.
Evaluate the effectiveness of application of a variety
of EMS equipment in a practical setting.
Objectives cont’d
3
Actively participate in review of selected Region X
SOP’s as related to the topics presented.
Actively participate in review of the process of
transmission of 12 lead EKG’s using department
specific equipment.
Actively participate in reviewing the operation of your
department monitor/defibrillator, pacing capacity,
synchronized cardioversion
and defibrillation at the paramedic level.
Objectives cont’d
Actively participate in HARE/Saeger traction
application.
Successfully complete the post quiz with a score
of 80% or better.
4
Equipment and Patient Interventions 5
There
comes responsibility when using equipment in
the delivery of patient care. You need to:
recognize
what the problem is to know what to do
be able to distinguish what the appropriate intervention(s)
is/are
understand how to properly apply and use the equipment
chosen
recognize when the intervention is working as well as not
accomplishing the goal
know what documentation must be done with each piece
of equipment used in patient care
be knowledgeable regarding the cleaning and returning
to service for each piece of equipment
Capnography Background
A continuous, non-invasive monitoring tool
Measures level of CO2 at end of exhalation
Quantitative
results provides a number
Assesses respiratory status thru-out respiratory cycle
Provides current, at the moment, breath-to-breath
information on patient status
Results measured as mmHg of CO2
Normal
35 – 45 mmHg
6
Capnography Information
Numeric value provides end tidal (end of
breath) CO2 level
Waveform is a picture representation of the CO2
value exhaled with each breath
Airway status reflected in:
ETCO2 value (mmHg)
Waveform picture
Respiratory rate
7
Definitions
Ventilation
Process
of breathing; eliminating CO2 from body
Respiration
Exchange
Oxygenation
Getting
of gasses at alveoli level
O2 to tissues; measured by pulse oximetry
Diffusion
Process
by which gas moves between alveoli and
pulmonary capillaries (gases move from area of high
concentration to areas of low concentrations)
8
Capnography Usefulness
Provides information on how effectively the
body is:
Producing CO2 (metabolism)
Transporting CO2 (perfusion)
Exhaling CO2 (ventilations)
Goal – attain/maintain CO2 levels 35 – 45 mmHg
9
Capnography Usefulness cont’d
10
Confirms and monitors advanced airway placement
Indicates effectiveness of chest compressions
Blood
must circulate through lungs to off-load CO2 for it
to be exhaled
Levels
expected to minimally be >10mmHg during CPR
Indicates return of spontaneous circulation (ROSC)
Sudden,
sustained rise in levels toward 35-45 mmHg
Allows early interventions to be started
Capnography Usefulness cont’d
11
Monitor asthma & COPD conditions and response to
bronchodilator therapy
Detect increased respiratory depression and hypoventilation
Tiring
accessory muscles
Neuromuscular
disease effect on respiratory center
Change
in level of consciousness – alcohol/drug overdose,
head trauma, sedation/analgesia
Seizure
activity &/or post ictal period
Capnography Waveform
A-B – respiratory baseline
B-C expiratory upslope
C-D expiratory plateau
D – end of exhalation
point
of measurement
D-E – inspiratory downslope
12
Capnography Waveforms
Hypoventilation
CO2
retained so
values
Hyperventilation
CO2
eliminated
so values
13
Capnography Waveforms
Asthma attack or COPD
Difficulty
upslope
exhaling evidenced by slow, gradual
14
Capnography Waveforms
Apnea or loss of
advanced airway
- flat line
15
ETCO2 Detector
End tidal (end of breath) CO2 detector
Qualitative device
16
Indicates
presence/absence of detectable CO2 exhaled
via pH sensitive paper
Does
not provide specific measurement of numeric value
Color
Able
May
scale estimates CO2 level
to change as detected levels change
take up to 6 breaths to wash enough CO2 out for
proper measurement
ETCO2 cont’d
17
Gastric content or acidic drug contact on pH
paper can affect accuracy of values detected
When perfusion decreased
(shock, arrest) ETCO2 reflects
change in pulmonary blood flow
CO2 level
Does
not reflect ventilation status
and
Altered CO2 Levels
CO2 level
Shock,
cardiac arrest, pulmonary embolism,
bronchospasm, complete airway obstruction
CO2 level
Hypoventilation,
hyperthermia
respiratory depression,
18
CO2 Influence on Circulation
19
CO2 in blood (hypoventilation)
Cerebral
vasodilation increase in intracranial
pressure (ICP) due to increased blood flow to
the brain
CO2 in blood (hyperventilation)
Cerebral
vasoconstriction decrease in fresh
blood flow to brain; decrease in levels of
adequate oxygen and glucose negatively
affect function of brain
ETCO2 Result Interpretation
Yellow – yes, CO2 is being detected in exhaled
breath
Tan – poor perfusion or ventilation status
First
evaluate placement of airway device
Continue
to trouble shoot
Blue or purple – no CO2 being detected
First
evaluate placement of airway device
Continue
to trouble shoot
20
Trouble Shooting Advanced Airway
Placement – “DOPE”
D – displacement of tube (i.e.: into esophagus)
Chest
rise and fall?
Gastric
sounds?
Bilateral
breath sounds?
O – obstruction
P – pneumothorax
E – equipment failure
Faulty
cuff
21
Esophageal Detector Device - EDD
22
A modified bulb syringe
Simple means of evaluating for missed endotracheal
intubation
Squeeze bulb, attach to end of endotracheal tube
Bulb re-expands = tube in trachea
Bulb does not re-expand or does so slowly – collapsing
sides of esophagus onto tube preventing air from filling
EDD – consider esophageal placement
EDD cont’d
23
Need to interrupt ventilations to use device
Evaluate results of technique used with results of all other
steps of confirmation – could be extenuating reason why
you get false negatives
Defibrillators
Electrical capacitor that stores energy
Biphasic defibrillators provide waveforms that
use less DC energy than monophasic machines
Energy
flows in one direction and then reverses
Therefore, possible decrease in tissue damage
Survival rates increase if early CPR provided with
prompt defibrillation attempt as soon as possible
after collapse
24
Defibrillation
Early defibrillation critical to survival from sudden
cardiac arrest
Most
frequent initial rhythm in arrest is VF
Treatment
for VF is defib (defibrillation)
Probability
of successful defibrillation diminishes
over time
VF
deteriorates to asystole over time
Check with your vendor to know your biphasic
device’s recommended energy settings
25
Ventricular Fibrillation as Presenting
Rhythm
Best chance of survival in public
Early activation of EMS
CPR
initiated very soon after collapse
Early application of AED or other defibrillation attempt
Current passes though fibrillating heart to depolarize
heart cells to allow them to uniformly repolarize
Allows
dominant pacemaker (SA node) to take over
electrical control
Goal
– resume organized electrical activity
26
Influences on Success of Defibrillation
Time from onset of VF – shorter time survival
Condition of myocardium
Less
success in presence of hypoxia, acidosis,
hypothermia, electrolyte imbalance, drug toxicity
Pad size
Larger
pads felt to be more effective and cause
less myocardial damage; should not overlap
Ideal
size for adults10-13 cm (4 -5 inches)
Ideal
size for peds 4.5 cm (roughly 3 inches)
27
Influences cont’d
Pad / skin interface
Need
to the resistance
Greater
the resistance the less energy delivered to
the heart and the greater the heat production at
the skin surface
Pad contact
Max
contact with skin; no air bubbles breaking
contact; no pads touching or overlapping
Avoiding placement of pads over bone
Bone
is poor conductor of electricity
28
Pad Placement
Operator Choice
Anterior /posterior
1
pad over apex of heart, under
left breast
1
pad under left scapula in line
with anterior pad
Anterior/anterior (apex)
Anterior
pad on right upper sternum just
below clavicle
Apex
pad below left nipple in anterior axillary line over
apex of heart
29
Pad Placement cont’d
DO NOT place pads
Over
sternum – bone poor conductor of
electricity
Over
pacemaker or AICD – deflects energy;
could damage the implanted device
Place
Over
at least one inch away from device
topical medication patches – deflects
energy
30
Defibrillation
Indications
VF,
pulseless VT
Contraindications
Failure
to demonstrate one of the above rhythms
Asystole
– defibrillation places a patient into
asystole for the dominant pacemaker to take
over
PEA
– electrical activity not a problem; needs
mechanical response fixed
31
Defibrillation
Equipment
Monitor/defibrillator
Defibrillating
Example:
pads
PadPro
Defibrillation/pacing/cardioversion/monitoring
electrodes
Most
come with conductive gel already applied in
center of pad
32
Defibrillation Safety
CPR is performed just until the defibrillator is ready
Confirm O2 not blowing across patient’s chest wall –
hold away from the patient when not using the BVM
Physically look all around (“nose to toes”)
Clearly yell out “all clear”
Deliver energy
Immediately resume CPR
33
Return of Spontaneous Circulation
ROSC
After 2 minutes of resumed CPR, evaluate the
rhythm
If an organized rhythm is viewed on the monitor,
THEN check for a pulse
If
no pulse, rhythm is PEA
Resume
CPR
Adult
1 and 2 man CPR 30:2
Infant
and child 1 man CPR 30:2
Infant
and child 2 man CPR 15:2
34
Indications to Activate Cooling
Protocol Post ROSC
Presumed cardiac arrest
NOT
indicated for respiratory or traumatic arrest
Remains unconscious and unresponsive
ROSC present at least 5 minutes
Systolic B/P >90 with or without pressor agent use
(i.e.: Dopamine)
Airway has been secured
35
ROSC Contraindications
Major head trauma or traumatic arrest
Recent major surgery within past 14 days
Systemic infection (i.e.: septic shock)
Coma from other causes
Active bleeding
Isolated respiratory arrest
Hypothermia (34o C/93.2o F) already present
36
Induction of ROSC
Place ice paks in the axilla, neck and groin
Areas
where blood vessels tend to be superficial
Place ice pak over IV site
If patient begins to shiver, contact Medical
Control
Anticipate
Shivering
order for Valium to stop the shivering
will generate heat and therefore
increase body temperature
37
Vasopressor - Dopamine
Stimulates alpha, beta, and dopaminergic
receptors based on dose provided
Starting dose 5mcg/kg/min IVPB up to 20
mcg/kg/min
Take patient’s weight and drop last number
Minus
Left
Ex:
2 from number left
with rate to run IVPB in drops per minute
150 pounds; drop “0”
15
– 2 = 13 drops per minute
38
Dopamine cont’d
Dopaminergic effects at 2 mcg/kg/min
Renal
vasodilation to improve blood flow to kidneys
Keep
kidneys working, the body keeps working
Beta effects 5 – 10 mcg/kg/min
Increases
strength of myocardial contraction –
squeeze more blood out of ventricles
Alpha effects at >20 mcg/kg/min
Severe
vasoconstriction that diminishes blood flow to
all tissues
39
AED (Automated External Defibrillator) 40
Function
AED’s will
Analyze rhythms
Deliver a shock if indicated
Ventricular
fibrillation (VF)
Monomorphic
and polymorphic VT if rate and R
wave morphology exceed preset values
Will not deliver a synchronized shock
Can indicate loose electrodes / poor electrode
contact
AED Use in Pediatrics
41
Pediatric attenuator used to deliver lower energy doses to
children (built into cables with peds pads)
1-8 year old
Use pediatric pads if available
No attenuator (peds pads)available, use standard AED pads
< 1 year old
Manual defibrillator preferred
If no manual defibrillator, use peds pads with attenuator
No peds pads, use AED pads available
AED Use With CPR
Do NOT interrupt CPR to apply pads
Apply
pads while CPR in progress
Do not touch patient during analysis phase
Can provide compressions during charging phase
No O2 flow across patient body during defibrillation
attempt
Call and look “ALL CLEAR” prior to each defibrillation
attempt
Immediately resume CPR
42
Transition From AED To Defibrillator
43
Upon
arrival at scene, if AED ready to discharge,
utilize AED
Do
not interrupt operation of device
During
2 minutes of CPR, can switch from AED use
to monitor/defibrillator
Immediately
resume CPR after delivery of each
defibrillation attempt regardless of equipment used
Synchronized Cardioversion
44
A controlled form of defibrillation using a lower energy
level that interrupts underlying reentrant pathway
Used with organized rhythms and in presence of a pulse
Monitor interprets QRS cycle and energy delivered
during R wave
Less
vulnerable area of QRS
Downslope
Minimal
of T wave is relative refractory area
stimulant could generate rhythm into VF
Indications Synchronized Cardioversion
Unstable tachyarrhythmias
SVT
Rapid
atrial fibrillation or flutter
Hazard
of breaking loose a blood clot in the atria
and resulting in a stroke
Ventricular
tachycardia
Note: polymorphic VT NOT likely to respond to
synchronized cardioversion – no defined R wave
45
Synchronized Cardioversion Procedure
Apply pads
Anterior/anterior
or anterior/posterior position
Sedate if possible
This
is a painful procedure!
Versed
2 mg IVP/IO; repeated every 2 minutes;
max 10 mg (desired effect – sedation!)
Consider pain management
Fentanyl
1 mcg/kg IVP/IN/IO; may repeat in 5
minutes to max of 200 mcg total dose
46
Sync Procedure cont’d
Activate “sync” button
Verify
47
R wave is being flagged/identified
Choose energy setting starting at the lowest watt setting
100j,
200j, 300j, 360j
Verify O2 not blowing across chest wall
Look (nose to toes) and call “ALL CLEAR”
Press and hold sync buttons until energy discharged
Momentary
delay waiting to identify the R wave
Sync Procedure cont’d
If synchronized cardioversion needs to be
repeated, need to reset the “sync” button
Safety
that machine will default to defibrillation
mode after every discharge of energy
If VF occurs, verify sync mode is off and defibrillate
patient without delay
48
Transcutaneous Pacemaker - TCP
Electrical cardiac pacing across the skin
TCP is a painful non-invasive procedure so sedation
will most likely be necessary
Indications
Symptomatic
bradycardia
Hypotensive
Hypoperfusing
Evaluate
level of consciousness and B/P for most
reliable indicators of patient condition/stability
49
TCP Procedure
Apply pads
(-)
(+)
over apex of heart, anterior chest wall
mid upper back below left scapula
Set desired heart rate (80)
Confirm sensitivity at auto/demand
Begin mA current at 0
Turn pacer on
50
TCP Procedure
cont’d
Slowly increase output until ventricular capture
Spike
followed by widened QRS
Reassess vital signs and pain level
Document settings – mA and rate
Reassess need for sedation and analgesia
Valium
2 mg IVP/IO over 2 minutes; repeat every
2 minutes until max of 10 mg total dose
Fentanyl
1 mcg/kg IVP/IO/IN ; can repeat dose in
5 minutes with max total of 200 mcg
51
Critical Thinking Skill and TCP
In setting of acute MI, consider contacting
Medical Control
May
want to decrease heart rate of TCP just
enough to maintain perfusion
Want to avoid increasing the work load on the
heart by automatically selecting 80 as the heart
rate
Increasing
work load on heart may increase the
size of the infarction
52
What would you do…
53
You applied the TCP for a symptomatic bradycardia
You had a paced rhythm
You notice the following rhythm strip change – what is the
rhythm and what would you do?
Failure to capture
Reassess patient; increase mA; consider need for CPR
Defibrillation During Pacing Mode
Check your device for specifics
When in the pacing mode and the need to
defibrillate occurs, for some models, you may
have to turn off the pacing mode
If pacing must be resumed, reset all levels
54
12 Lead EKG’s
55
A graphic recording of electrical activity in the heart
Must evaluate the pulse to determine mechanical
response
Single lead (i.e.: lead II) evaluates cardiac rhythms
12 lead views can diagnose an acute MI
Early interpretation of 12 lead EKG early diagnosing
early reperfusion & restoring blood flow to ischemic
tissues
Acute MI
Death of portion of heart muscle from
prolonged deprivation of oxygenated blood
Heart’s demand exceeds supply of oxygen over
extended period of time
Often associated with atherosclerosis process
Location and size of infarct depends on vessel
involved and site of obstruction
Left
ventricle most common site
56
Evolution of Acute MI
Ischemia – initial lack of oxygen
ST
Injury to myocardial tissue
ST
depression can be reversible
elevation can be reversible
Death/infarction
Necrotic
tissue can lead to scar formation
Irreversible
Can
process
leave a positive Q wave marker in leads
affected
57
58
AMI Process
Ring of ischemic tissue surrounds infarcted
myocardium
Collateral circulation may develop
Ischemic area often site of arrhythmia
development
59
Complications of AMI
60
Arrhythmia most common
VF most lethal
Most
common cause of sudden death within one hour of
onset of signs and symptoms
Destruction of myocardial muscle mass can lead to CHF
due to impairment of pumping capability
Cardiogenic shock may develop if heart function is
inefficient and inadequate
Ventricular aneurysm can develop due to damaged
wall of heart – can rupture causing instant death
Patient Assessment
Pain most common chief complaint
Lasts
Not
more than 30 minutes
relieved by rest or NTG
Tired and weak most often complaint in elderly,
long standing diabetic and women
Determine responses to OPQRST assessment
Activity
at onset, provocation/palliation
(worsens/improves), quality in their words,
radiation, severity on 0 -10 scale, time of onset
61
EMS Action
Apply monitor
Examine underlying rhythm – document rhythm
Obtain 12 lead EKG
Evaluate
for ST segment elevation
If
elevation, in what group of leads?
If
depressed, look for reciprocal elevation
Watch for development of arrhythmias
62
Proper Placement EKG Chest Leads
63
Groups of Acute MI by Leads
64
Identifying Groups of ST Elevation
65
Why Aspirin???
66
Inhibits platelets from aggregating/collecting at site of
plaque rupture inside vessel wall
Decreases morbidity and mortality rate
Chewed to increase breakdown and absorption time of
medication
Patients on daily aspirin already have elevated and
acceptable blood levels of aspirin – don’t have to
supplement a dose if absolutely sure they took one today
Always better to give full dose than to risk skipping any
dose (just in case of skipped dose)
12 Lead EKG Procedure
Obtain rhythm strip
Interpret, report and document rhythm
Obtain 12 lead EKG
Identified
with patient age, sex, department
name in preparation for transmission
Review for ST elevation pattern
Report to Medical Control what you see, then
read word for word interpretation on 12 lead
EKG printout
67
12 Lead EKG Documentation
Interpret the rhythm strip and document on
patient care run report
Document presence or absence of ST elevation
If
elevation, report and document in which leads
Provide copy of rhythm strip and 12 lead EKG to
ED secretary for placement on patient’s medical
record
68
CPAP
Continuous positive airway pressure
Effective therapy for acute CHF –
pulmonary edema
Can avert the need for intubation and
mechanical ventilation if applied early enough
Maintains constant pressure within the airway
and through-out the respiratory cycle
Keeps alveoli open and expanded
Increases surface space for diffusion of gases
69
CPAP cont’d
Buys
time for other therapies
(i.e.: medications) to work
Precaution
Too
much pressure can
inhibit ventricular filling
decreasing cardiac output
B/P
can drop
70
CPAP Indications
Stable pulmonary edema
Alert;
systolic B/P >90mmHg
COPD with wheezing
First
contact Medical Control for orders
For unstable pulmonary edema (altered mental
status, systolic B/P <90 mmHg), contact Medical
Control to discuss use of CPAP
Reminder: all therapies used in pulmonary
edema have potential to drop the B/P
71
CPAP Contraindications
Respiratory arrest or apnea
Pneumothorax or trauma to chest wall
Tracheostomy present
Can’t
get tight fit over trach stoma
Actively vomiting
72
CPAP Procedure
Sit patient upright
Assess and obtain baseline vital signs
Begin O2 via non-rebreather mask while setting
up equipment
Administer first dose NTG
Used
as venodilator to decrease blood return to
heart (decreases pre-load)
73
CPAP Flow Safe II Procedure
Assemble CPAP Flow Safe II
Attach
proximal end of O2 tubing with
manometer to port in mask
Attach
distal end of tubing to O2 source
Secure
face mask snugly to patient’s face using
head harness
Adjust
O2 flow – 13-14 lpm for 10 cm H2O
Continue administration of medications
74
CPAP Procedure cont’d
75
Lasix 40 mg IVP (80mg if on med at home) as a diuretic
If systolic B/P remains >90 mmHg
Morphine
2 mg IVP slowly over 2 minutes
May
repeat 2 mg every 2 minutes as needed to max of
10 mg
Used
to decrease anxiety and for benefit of vasodilation
If patient shows deterioration during CPAP treatment,
remove CPAP, consider intubation, inform Medical
Control
CPAP Tidbits
Be prepared to coach patient through first few
minutes of CPAP use until positive effects begin
Patient
is already frightened
Patient
may feel suffocated with the mask on
Exhaling
against the resistance is tough at first
76
HARE and Saeger Traction
Indicated for isolated mid-femur fractures
Reduces
muscle spasm and therefore pain level
Reduces
risk of bones overriding
Contraindications
Open
Do
Hip,
fracture
not want to draw contamination into the wound
knee, or pelvic fractures
Increased
risk of nervous or vascular complications
77
Preparing for Traction Application
Assess motor/sensory/circulation before and
after splinting
Can
you move this/can you feel that?
Mark
pulses once found – easier to find the site on
reassessment
Compare
to uninjured side
Apply manual traction until mechanical traction
in place
78
HARE Application
Measure and adjust splint
Support distal end of splint on backboard
Apply distal ankle hitch while maintaining
manual traction
Position traction under injured extremity
Secure proximal end to groin area
Apply hook to ankle hitch
Replace manual traction with mechanical traction
79
HARE Traction
80
Adjust straps avoiding over the knee and over the injured site
Saeger Traction Application
Support leg and maintain gentle traction
Use uninjured leg to measure and adjust splint length
Place splint inside injure leg; padded bar snug against
pelvis in groin (watch pressure areas!!!)
Attach strap to thigh
Attach padded hitch to foot and ankle
Extend splint until correct tension obtained
Apply elastic straps to secure leg to splint
81
Saeger Splint
Do not place straps over fracture
site
Release manual
traction
Reassess distal pulse,
motor, and sensory
82
Pain Control With Use of Traction
Fentanyl 1 mcg/kg IVP/IN/IO
May
repeat same dose in 5 minutes
Max
total dose of 200 mcg
As a CNS depressant, watch the respiratory status
If respiratory depression occurs, begin to support
ventilations via BVM
1
Breath every 5 – 6 seconds
Document
10 -12 breaths per minute assisted
Narcan 2 mg IVP/IN/IO can be used to reverse
respiratory depression due to opioid use
83
Cleaning of Traction Splints
Rinse off gross contaminant
Wet down all surfaces with Cavicide wipes
Let device air dry
Confirm all straps are accounted for and
repackage device in preparation for next
patient
84
Combat Application Tourniquet CAT
Indications
Uncontrollable
hemorrhage when
usual means have failed
Contraindications
Non-compressable
site
Equipment
Tourniquet
with attached rod
85
CAT - Procedure
Apply tourniquet proximal to bleeding site as
distal as possible; preferably over bare skin
Pull band very tight and securely fasten band
back on itself
Twist rod until bright red bleeding has stopped
Or
until distal pulses are eliminated
Place rod inside clip; locking into place
Secure straps over clip holding rod
86
CAT – Potential Problems
Inability to control bleeding
Continue
Prepare
Apply
with direct pressure
to apply a second CAT
QuikClot dressing if available
Must
be applied directly over wound site for
impregnated material to be effective
87
CAT – Documentation Pearls
Reason CAT was applied
Time and site of CAT application
Results post intervention
Consideration of administration of pain medication
Fentanyl
1 mcg/kg IVP/IN/IO
May
repeat in 5 minutes, same dose
Max
200 mcg total dosing
88
Midazolam
Versed
Potent, rapid onset, short acting benzodiazepine
Onset
3-5 minutes
Duration
20-30 minutes
Used as sedative and hypnotic
Has amnesic properties and reduces anxiety
Amnesia
of recent past (antegrade) useful to
inhibit unpleasant reminders of procedures
Low toxicity and high rate of effectiveness
89
Indications for
Versed Per
Region X SOP’s
Sedation prior to synchronized cardioversion
Useful to maintain sedation post drug assisted
intubation procedure
Suppresses seizure activity
IN
90
route allows safer delivery method
Decreases severe anxiety and apprehension
Precautions With
Versed
Crosses placental barrier – could cause
respiratory depression in newly born infant
Elderly more sensitive to effects; metabolize med
more slowly
Toxicity increases when mixed with CNS
depressants (alcohol, opioids like Fentanyl,
tricyclic antidepressants)
Toxicity may be higher in patients with COPD
91
Side Effects of
Versed
Respiratory depression
Drowsiness
Hypotension
When administering, have a BVM readily available
Be prepared to assist respirations
1
breath every 5 – 6 seconds
Document
10 – 12 breaths per minute assisted
92
Fentanyl
Synthetic opiate analgesic for pain control
Shorter acting than morphine
Onset immediate when administered IVP
Peak effect 3 5 minutes
Lasts 30 – 60 minutes
Does not affect blood pressure like Morphine
does
93
Dosing For Fentanyl per Region X SOP’s
Adult
1
mcg/kg IN/IVP/IO
May
repeat same dose in 5 minutes
Max
total dose 200 mcg
Pediatrics
0.5
mcg/kg IVP/IN/IO
May
repeat same dose in 5 minutes
Max
total dose 200 mcg
94
Precautions With Fentanyl
Crosses the placental barrier – could cause
respiratory depression in newly born infant
Monitor respiratory rate, SpO2 levels, and level of
consciousness
Have BVM available to counteract potential
respiratory depression
1
breath every 5 – 6 seconds
Document
10 – 12 respirations per minute
95
Cleaning of Equipment –
After Every Patient Use
96
In general, each piece of equipment in contact with a
patient MUST be cleaned between each patient use
Gross contaminant must be removed
Surfaces need to remain wet and allowed to air dry
All cables need to be wiped down (i.e.: EKG, B/P, pulse ox)
Cables
drag across contaminated surfaces A LOT!!!
B/P cuffs need to be wiped down
Pulse ox sensors need to be cleaned following manufacturer
recommendations
Department Review of Equipment
Review set up of capnography monitoring
Review operation of monitor/defibrillator for
defibrillation, synchronized cardioversion, and
TCP
Review procedures for transmission of 12 lead
EKG to receiving hospital
In teams, apply the HARE or Saeger traction
device to a peer
97
Bibliography
98
Bledsoe, B., Porter, R., Cherry, R. Paramedic Care Principles &
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