Transcript Slide 1
Hypoperfusion and Shock
Hypoperfusion
Common problem
Extent makes resuscitation
difficult
Shock due to hypoperfusion
Start fluid resuscitation as soon
as possible
© 2009 NAEMT
Overview
Describe differences between
compensated and uncompensated
shock
Review differences of distributive,
non-distributive and obstructive shock
Explore pathophysiology for different
etiologies of shock
Discuss interventions for early and late
shock
© 2009 NAEMT
Physiology
BP = Cardiac Output x Systemic Resistance
Cardiac Output = Stroke Volume x Heart Rate
After-load = Resistance to blood being ejected
Pre-load = Blood returned to heart
Starling’s Law = Amount of cardiac
muscle stretch
LifeART
NHTSA
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Shock Compensation
Children vs. Adults
Children
Increased heart
rate
Vasoconstriction
Prolonged
compensation
Rapid
decompensation
Adults
Increased stroke
volume
Vasoconstriction
Tachycardia
Slow, but
sustained
compensation
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Categories of Shock
Non-Distributive
Hypovolemic
Hemorrhagic
Metabolic
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Categories of Shock
Distributive
Anaphylaxis
Septic
Neurogenic
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Categories of Shock
Obstructive
Pulmonary embolus
Tension pneumothorax
Cardiac
tamponade
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Etiologies of Hypoperfusion
(Common)
Emesis and diarrhea
Osmotic diuresis from diabetes
Internal or external blood loss
Plasma loss from sepsis or
anaphylaxis
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Etiologies of Hypoperfusion
(Uncommon)
Spinal cord injury
Cardiac failure
Medications required to
restore perfusion
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Severity of Hypoperfusion
Compensated
Volume
Compensated
Decompensated
Signs are due
to inadequate
tissue perfusion
Compensated
shock is
reversible with
fluids
Time
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Severity of Hypoperfusion
Compensated Shock Signs
Compensated
Decompensated
Volume
Pulse
Breathing
Blood
Pressure
AVPU
Altered Mental Status
Time
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Severity of Hypoperfusion
Compensated Shock Signs
Compensated
Decompensated
Weak or absent
peripheral
pulses, weak
central pulses
Volume
Weak
peripheral
pulses, strong
central pulses
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Time
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Severity of Hypoperfusion
Dehydration Testing
Hypovolemic patient’s
skin will “tent”
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Severity of Hypoperfusion
Decompensated Shock
Compensated
Decompensated
Volume
Inadequate
tissue perfusion
to all organs
Body is unable
to continue
compensation
Time
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Severity of Hypoperfusion
Decompensated Shock Signs
Compensated
Decompensated
Volume
Pulse
Breathing
Blood
Pressure
AVPU
V
Altered Mental Status
P
U
Weak or absent peripheral pulses, weak central pulses
Time
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Severity of Hypoperfusion
Decompensated Shock Signs
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Assessment
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Scene Survey
Hazards to you, your partner,
the patient and bystanders
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First Impression
Pediatric Assessment Triangle
Compensated or
decompensated
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First Impression
General Appearance
Observe interactions
Not sick - attentive to environment,
focus on familiar people and
objects, alert for threats
Good brain function requires
adequate oxygenation, ventilation,
cerebral perfusion
Sick - does not care you are
present or recognize parents
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First Impression
General Appearance
Muscle tone
Spontaneous movements
Skin color
Other signs of distress
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First Impression
Work of Breathing
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First Impression
Circulation to the Skin
Skin color, capillary refill,
distal vs. central pulses
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First Impression
Sick
Rapid Initial Assessment
Not Sick
Yes
Significant MOI?
No
Appropriate Interventions
Relationship
Transport Priority
Involve Family
Transport Method
Detailed History
Transport Destination
Focused Physical Exam
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Initial Assessment
Airway
Loss of airway
may occur in
decompensated
shock
Identify and treat life
threats
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Initial Assessment
Breathing
Assess for chest trauma
Abnormal sounds
Rate effort and volume
Administer O2 and
treat cause
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Initial Assessment
Circulation
Compensated
Weak peripheral pulses,
strong central pulses
Decompensated
Weak or absent peripheral
pulses, weak central pulses
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Initial Assessment
Circulation Management – Intravenous
Fluid bolus if any signs of shock
Early recognition of hypoperfusion
and fluid resuscitation are key
Select a large bore catheter
Location close to central circulation
Two IVs may be needed
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Initial Assessment
Circulation Management – Intraosseous
Can be used on any age
child
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Intraosseous Space
Blood Flow
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Anatomy
Neonate Leg Cross Section
Skin
Intraosseous
Catheter
Tibia
Lateral
Compartment
Fibula
Subcutaneous
Fat
Anterior
Compartment
Posterior
Compartment
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Other Issues
IO Insertion
Depth based on patient size and weight
Gently insert catheter
Advance catheter slowly
Feel needle drop into medullary space
Frequently monitor insertion site and
extremity
Need hands-on training
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IO Insertion
Anatomical Landmarks
Patella
Tibial
Tuberosity
Medial
Tibia
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IO Insertion
Unable to Palpate Tibial Tuberosity
Finger Width
Finger Width
Often difficult or impossible
to palpate
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IO Insertion
Able to Palpate Tibial Tuberoisty
Finger Width
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Anatomy
Neonate Leg Cross Section
Traditional IO
Catheter
Tibia
Fibula
Left Leg
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Anatomy
11 y.o. Tibia Cross Section
Tibia
Fibula
Left Leg
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Pain
Somatic and Visceral
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Initial Assessment
Circulation Management – Crystalloids
20 mL/kg, < 20 minutes
Reassess patient after
each fluid bolus
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Initial Assessment
Never Administer D5W
D5W can lead to
hyperglycemia
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Initial Assessment
Circulation Management – Medications
Sepsis
Pressers and
antibiotics
Cardiogenic
Shock
Pressers, furosemide,
morphine and
antiarrhythmics
Anaphylaxis
Epinephrine,
diphenhydramine,
Solu-Medrol
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Initial Assessment
Circulation Management – Medications
Use medications after fluid
boluses
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Transport Decision
Rapid transport for
pediatric shock patients
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Focused History
Questions to Determine Type of Shock
Bleeding
Vomiting
Diarrhea
Fluid intake /
urine output
Fever
Anaphylaxis
signs
FEMA Photo Library / Andrea Boomer
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Head to Toe Physical Exam
Done En Route
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Ongoing Assessment
Done Frequently
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Summary
Recognition and rapid intervention
are keys to treatment
Pulse quality and level of
consciousness are key indicators
Obtain IV or IO access if shock
treatment is needed
Deliver crystalloid fluids at 20
mL/kg
© 2009 NAEMT