February 2014 CE - Advocate Health Care
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Transcript February 2014 CE - Advocate Health Care
The Victim of Drowning
Abused Drugs
February 2014 CE
Condell Medical Center
EMS System
Site Code: 107200E-1214
Prepared by: Sharon Hopkins, RN, BSN, EMT-P
Rev 2.14.14
1
Objectives
Upon successful completion of this module,
the EMS provider will be able to:
Discuss definition and circumstances of
drowning
Describe the pathophysiology of drowning.
Discuss the assessment and management of the
patient who has drowned.
List the categories of drugs most commonly
abused.
2
Objectives cont’d
Describe the effects on the body systems
based on the drug abused.
Describe complications noted with abuse of
drugs
Discuss assessment and management of the
patient who has abused drugs.
Describe the benefits of using capnography in
patient assessment and in defining patient
care.
Actively participate in interpretation of
waveform capnography.
3
Objectives cont’d
Actively
participate in review of
selected Region X SOP’s.
Actively participate in case scenario
discussion.
Review responsibilities of the preceptor
role.
Successfully complete the post quiz with a
score of 80% or better.
4
Definition and Stats of Drowning
Drowning is death by suffocation due to
immersion in water
Death is within 24 hours of the incident
Near-drowning is survival past the 24 hour mark
from the incident
Most likely victims are young and healthy
3 likely victims
Toddlers
Adolescents
Elderly
Recovery dependent on prompt rescue and
resuscitation
5
Background on Drowning
Toddlers
drown in bathtubs, toilets, and
buckets/pails of water
Adolescents drown usually around larger
bodies of water
Males proportionately higher than females
areas – swimming pools most
likely site
Alcohol involved in approximately 40% of
drownings
Coastal
75% involves use in and around boats
6
Drowning
Usually
occurs silently and quickly
Often within reach of a rescuer
Near-by help do not recognize the situation
Image
of person drowning is a movie
concept
Most people struggle to breathe and have no
breath left to yell for help
Most people do not wave their arms around;
they use their arms to try to lift themselves out
of the water
Most people drowning look like they’re “playing”
7
8
Potential Outcomes of Drowning
Death
Morbidity
Development of disability or injury related to
the incident
Survival
with no morbidity
Most
surviving children found within 2
minutes; most who die are found beyond
10 minutes
9
Definitions
Morbidity
Development of disability or injury related to
the incident
State of being diseased or unhealthy related
to the incident
Mortality
Incidence of death in a population
10
Pathophysiology of Drowning
An
injury to primarily the pulmonary
system and central nervous system (CNS)
Prognosis is linked/related to
Submersion time which directly affects the
severity and duration of hypoxia
Temperature of the water
11
Pathophysiology of Drowning
Victim
initially holds their breath
Involuntary laryngospasm triggered by
water in the airway
Victim unable to breath causing decreased
oxygen levels and increased levels of
carbon dioxide
With decreasing O2 levels, laryngospasm
releases, victim gasps, hyperventilates,
and possibly aspirates waterhypoxemia
12
Pathophysiology cont’d
Water
enters lungs only if actively inhaled;
water does not passively infuse into lungs
Developing hypoxemia and acidosis
contribute the most to morbidity and
mortality
13
Older Term: Dry Drowning
– airway closes due to laryngeal
spasms from the presence of water
Definition
Hypoxemia is cause of death
Includes
10-20% of submersions
Sequence involves
Laryngospasm
Hypoxia
Loss of consciousness
14
Older Term: Wet Drowning
Definition – water inhaled which interferes with
respirations which affects cardiovascular system
Water is aspirated
Surfactant is diluted
Chemical covering alveoli that keeps them open
facilitating respirations
Decrease in transfer of gases
Atelectasis develops
Ventilation perfusion mismatch occurs
Blood pumped thru deoxygenated lungs causing a
decreased level of O2 in circulating blood volume
15
Mammalian Diving Reflex
Sudden exposure to cold water (<200C (680F))
Breathing stopped - apnea
Bradycardia
Vasoconstriction of nonessential vascular beds
• Blood shunted to heart and brain
Metabolic processes slowed down
Noted especially in young children
Patient may appear dead
Patient cannot be pronounced if cold & dead; must be
warm & dead
16
Pathophysiology of
Fresh Water Drowning
Fresh
water moves across alveolarcapillary membrane into microcirculation
Electrolyte abnormality can occur
Fresh
water exposure
Surfactant destroyed
Alveoli collapse
Ventilation-perfusion mismatch occurs
Pneumonia
rare consequence but more
likely in stagnant warm water and fresh
water
17
Pathophysiology of
Salt Water Drowning
Salt
water increases osmotic gradient
Fluid drawn into alveoli
Surfactant washed out
Membrane between alveoli and capillaries
damaged
Ventilation-perfusion mismatch
Lungs
non-compliant and become filled
with fluid
18
Complications of Submersion
– major complication
Water and bacteria enter the lungs
Hypoxemia
Development of pneumonia
Development of ARDS
• Lung condition causing leaky pulmonary capillaries
and development of hypoxemia
Hypothermia
if in cold water
Cervical spine injury complicates the
capability of the victim to self-rescue
19
Pulmonary Injuries from Drowning
Contaminated
foreign material
contaminates the pulmonary system
Particulate matter present
Bacteria has a portal of entry
Vomitus increases risk of aspiration
Pulmonary system exposed to chemical
irritants
Surfactant destroyed/washed away
20
Obtaining a History
Age
Underlying medical problems
History drug/alcohol use/abuse
Length of time submerged/unattended prior to
finding
Potential trauma with drowning (i.e.: dive)
Change in level of consciousness
Change in behavior since removal from water
Prior or since any seizure activity or chest pain?
Vomiting
21
Assessment of the Patient Who
Has Drowned
“CAB”
approach if found unresponsive
“ABC” approach if alive
Vital signs
Neurological assessment
AVPU, GCS, PMS/SMV/CMS, pupils
Lung
sounds
Cardiac monitoring
Consideration spinal immobilization/spinal
motion restriction
22
Management of the Patient Who
Has Drowned
Typically
based on findings during the
assessment phase
Closely monitor airway and apply
precautions against aspiration
Rapid transport
All victims of drowning regardless of
presentation should be encouraged to be
evaluated by a physician
• Some problems are not immediately obvious
23
Region X SOP
Adult Near Drowning
Adult
Routine Medical Care or Adult
Routine Trauma Care
Spinal precautions
Consider CPAP if condition indicates
Determine patient stability
Stable? Treat signs and symptoms
Patient alert
Skin warm and dry
Systolic B/P >90 mmHg
24
Adult Near Drowning SOP cont’d
Unstable?
Altered mental status
Systolic B/P <90 mmHg
Secure airway
Assess for hypothermia
If normothermic treat dysrhythmias per
protocol
If hypothermic follow hypothermic protocol
25
Adult Hypothermic Protocol
Adult
Routine Medical Care
Frostbite
Move to warm environment
Rapidly rewarm frozen areas with warm water
(if available)
OR
Use hot packs wrapped in towel
Handle skin like a burn
• Protect with light, dry sterile dressing
• Elevate and immobilize
• Do not let surfaces rub together
Manage pain appropriately
26
Adult Hypothermic SOP cont’d
Systemic
Avoid rough handling and excess activity
Apply heat packs (as available) to axilla,
groin, neck, thorax
Assess
hypothermia
pulse
Present? – Continue assessment
Absent?
• Withdrawal of Resuscitative Efforts policy does not
apply to these patients
27
Adult Hypothermic SOP cont’d
Pulse absent
Universal Adult Emergency Cardiac Care (CPR)
Evaluate flexion of extremities
Flexible?
• Follow cardiac protocol
• Extend time between medications to the max
• Repeat defibrillation as core temp rises
Not flexible?
• Follow cardiac protocol
• Limit shocks to 1
• Withhold IV medications
28
Pediatric Near Drowning SOP
Routine
Pediatric Care
Spinal Precautions
Oxygenate at 100%
Determine stability
Stable? Treat signs and symptoms
• Awake, alert, normal respirations
Unstable? Abnormal respirations, altered
mental status
• Assess for gag reflex
29
Pediatric Near Drowning SOP
cont’d
If
gag reflex absent
Intubate and assist ventilations via advanced
airway with BVM
• 1 breath every 6 - 8 seconds
If
gag reflex present
Assist ventilations via BVM
• 1 breath every 3 – 5 seconds
Assess
for hypothermia
Normothermic – treat dysrhythmias
Hypothermic – refer to hypothermic protocol
• Same as adult
30
Prognosis of Drowning Victim
Often
poor
Related to multiple factors
Patients worse off
Initial presentation full arrest
Those remaining comatose
Those with fixed and dilated pupils
Those in respiratory arrest
Duration
of hypoxemia impacts potential
for survival and for body system insult
31
Prognosis cont’d
Morbidity and death primarily from
Laryngospasm
Pulmonary insult
• Resulting hypoxemia and acidosis
Effects on the brain and other organ systems from
hypoxemia and acidosis
Secondary death risk from development of
ARDS
Patients presenting awake and conscious have
best chance of full recovery
32
Patients with Altered Mental Status
Consider etiology – AEIOU-TIPPS
Trauma/temperature
Epilepsy
Infection
Insulin
Psychogenic
Overdose/opiates Poisoning
Uremia
Shock / seizure /
stroke / shunt
Alcohol
33
Drugs Potentially Abused
– depresses CNS;
depresses ventilations, creates feeling of
euphoria
Opioids/Narcotics
Synthetic – Tramadol, Propoxyphene
(Darvon), Fentanyl
Semisynthetic – dextromethorphan,
hydrocodone (Vicodin®, Lortab®), oxycodone
(oxycontin, Percodan®, Percocet)
Natural – codeine, morphine, paregoric,
heroin
34
Opioids/Narcotics cont’d
Long
term effects
Restlessness
Muscle and bone pain
Insomnia
Vomiting
Cold flashes
35
Narcotic - Heroin
Harvested from opium poppy
3-6 months from planting to harvesting
Snorted, injected, smoked
Constricted pupils
Droopy, watery eyes
Dry mouth
Nausea/vomiting
Slow slurred speech
Mental clouding
Loss of coordination
pulse
pain sensitivity
appetite
sexual drive
36
Heroin cont’d
Long
term effects
Physical and psychological dependence
Tolerance
Mood swings
Seizures
Coma
37
Definitions
Physical dependence
Psychological dependence
Perceived “need” or “craving” for a drug
Can last a long time even after use stops
Addiction
Changes in the body after repeated use of drug that
requires continued administration of drug to prevent
withdrawal symptoms
Compulsive drug-seeking behavior; loss of control in
use of drug; drug most important thing for user
Physical tolerance
Shortened duration and intensity of effects creating
need for increasingly larger doses to attain desired
effect
38
Drugs Potentially Abused
Stimulants
Chemical substances (natural or synthetic)
that affect the CNS and accelerates activities
Caffeine
Nicotine
Adrenalin
Cocaine
Amphetamines
39
Stimulant - Cocaine
Euphoria then depression
Dilated pupils
Nasal tissue irritation, perforated septum
Tooth decay – anesthetic effect on gums
Vasoconstriction at point of injection
Increased heart rate
Severe chest pain
AMI – most common in 18-45 year-old;
Coronary artery spasm and platelet activation
contribute to coronary occlusion; may have no
evidence noted on angiogram inspection
40
Stimulant – Cocaine cont’d
Long
term effects
Strong psychological dependence
Physical tolerance
Eating disorders
Impotence
Seizures
Strokes
41
Stimulant - Cocaine
Smoked
Effects quicker/stronger
Onset 7 seconds; duration 15 minutes
Short period of high
Injected
Highly water soluble – dissolves easily in
water
Onset 15-30 seconds; duration 15-20 minutes
42
Stimulant - Cocaine
Crack cocaine
Process of converting cocaine hydrochloride (HCl)
back to cocaine base for smoking
Ingredients added to cocaine, heated, then cooled,
then filtered to collect crystals
Free basing
Process of converting cocaine HCl back to cocaine
base for smoking
Onset 8 - 10 seconds
Effects 5 - 10 times more intense than snorting
• Theory that freebasing removes contaminants
making a purer product
Requires adding a solvent in process which risk of
explosion and fire
43
Stimulant - Methamphetamine
Powder, liquid, tablets
Injected, inhaled, smoked, oral
Smoked – in bloodstream 5-10 seconds
High lasts 80
More potent than amphetamine
Physical & psychological dependency
Tolerance develops quickly
Users volatile, paranoid, unreliable
Intense tremendous energy; impulsive
44
Meth cont’d
attention span
Intense itching – “bugs”
Tremors
Euphoria
Rapid speech
Hallucinations
Violence
Stages: Rush up to 30 min High 4-160 Binge-3-15
days; ingest more to continue high Tweaking (end of
binge)– psychotic state; paranoia, depression,
aggression Crash – body shuts down, deep sleep 1-3
days meth hangover – lasts 2-14 days; deteriorated
state, exhausted; starts over to feel better withdrawal
– painful & difficult; lasts 30-90 days; depressed,
energy, no pleasure, crave the drug
45
Drugs Potentially
Abused
Bath salts – outlawed in most States
Strong stimulant that creates aggression &
hallucinations
So named due to resemblance to bath salts
Labeled “not for human consumption” to avoid much
oversight
Synthetic similar to amphetamines
Unable to identify exact chemical composition so
treatment can be hampered/ difficult
3 most common chemicals: mephedrone,
pyrovalerone, methylenedioxyprovalerone (MPDV)
46
Bath Salts cont’d
Snorted,
injected, ingested with food/drink
Strongly addictive; triggers intense cravings
Agitation
Paranoia
Hallucinations
Chest pain
heart rate
High B/P
Kidney pain
Muscle tension
temp or chills
Nausea
Confusion
47
May overheat and tear off clothes
Bath Salts cont’d
Pt
often reports they thought they were
going to die; heavy drug users swear they
will never use this again
Paranoia aggressive, uncontrollable
attacks on others, self-destructive with
complete disassociation with reality
Probably won’t respond to requests
Pepper spray and tasers not likely to be
effective
Effects last 3-4 hours or longer
48
Drugs Potentially Abused
Depressants
Tranquilizers – used medically for anxiety,
depression, sedation, seizures, anesthesia,
sleep control, as antipsychotics
• Anti-anxiety – Rohypnol, Valium, Xanax
Barbiturates – seconal, Phenobarbital
Non-barbiturate – haldol, Quaaludes
49
Depressants – Rohypnol - Roofies
Intended as pre-med for anesthesia and
treatment for insomnia
Left no taste, odor, or visible effect in drinks
(changed in 1977 to be visible in drinks)
Onset 20-30 min; duration 8-120; detectable 720
Impairs judgment
inhibitions
Blackout/amnesia
Dizziness
Drunk like behavior
Gait ataxia
Slurring & stuttering
pulse, B/P
50
Common Over-The-Counter (OTC)
Products
ASA and
Tylenol commonly added to
many over-the- counter products
Inadvertent overdosing can be common
51
Drugs Commonly Abused
– Tylenol®
Most common poisoning
Suppository, tablet, liquid, drops, caplets
Max dose 4000 mg/day (8 extra strength;
12 regular)
>7000 mg/day could be severe OD
Acetaminophen
(14 extra strength; 22 regular)
Symptoms
start approximately 120 post
ingestion
52
Acetaminophen – Tylenol® cont’d
1st 24 hours
Abdominal pain
Sweating
Pale, tired
24 – 72 hours
Pain RUQ
Dark colored urine
72 - 96 hours
•appetite
•Nausea//vomiting/diarrhea
•Jaundice
• urine production
Blood in urine
•Hungry, shaky, weak & tired
Blurred vision
•Fever
Tachypnea, tachycardia •Confusion, coma
53
Acetaminophen – Tylenol® cont’d
Good
recovery if treated within 8 hours of
overdose
Improvement within 7 days
Liver
failure and death in few days without
rapid treatment
Increased risk
Alcoholic intake >3/day
Smoker
Known liver disease
54
OTC Drugs Commonly Abused
acid – Aspirin
Availability makes it a common source of
unintentional and suicidal ingestion
Acetylsalicylic
Found in multiple OTC products
Tablet,
capsules, liquid, topical
Affects multiple systems:
Central nervous
Pulmonary
Metabolic
• Cardiovascular
•Hepatic
•Renal
55
Acetylsalicylic acid – Aspirin
Earliest
Nausea & vomiting (common)
Diaphoresis
•Tinnitus
Vertigo
•Tachycardia
Hyperventilation (common)
As
signs & symptoms
toxicity progresses
Agitation
Hallucinations
Lethargy
•Delirium
•Convulsions
•Stupor
56
Acetylsalicylic acid – Aspirin
– indicates severe toxicity
especially in young children
Patient becomes severely hypoxic
Dehydration due to vomiting, increased
respiratory rate, hyperthermia
GI hemorrhage more likely in chronic
intoxication
Field care is supportive
Hyperthermia
57
Acetylsalicylic acid – Aspirin
Sample Ingestion for 166# / 75 kg Adult
<150
mg/kg – no toxicity
11,250 mg – 35 adult ASA
150-2300
mg/kg – mild to moderate
toxicity
22,500 mg – 69 adult ASA
301
– 500 mg/kg – serious toxicity
37,500 mg – 116 adult ASA
>500
mg/kg – potentially lethal toxicity
>37,500mg
58
Product Potentially Abused
glycol – Antifreeze
Colorless, odorless, sweet tasting liquid
120 ml (4 oz) could be fatal to average
sized man
1st symptom similar to drinking alcohol
(ethanol)
Within few hours
Ethylene
Nausea & vomiting, convulsions, stupor, coma
59
Ethylene glycol – Antifreeze
Overdose
Damage to brain, lungs, liver, kidneys
Metabolic acidosis
Shock
Organ failure
Death
Outcome
depends on time treatment
started
60
Drugs Accidentally Ingested
Nicotine
ingestion
1 full cigarette, 3 butts, one piece of nicotine
chewing gum swallowed can be toxic to a
toddler
Product
can be tempting smelling like
mint, vanilla, and cherry
Patient needs a medical evaluation
61
Nicotine Ingestion
Mild
poisoning
Vomiting
Lethargy
Severe
•Sweating
•Tremors
poisoning
Confusion
Paralysis
Seizures
Field
care is supportive
62
Paraphernalia Expected in
Environment
Syringes,
needles
Scales
Baggies
Pipes
for smoking
Coffee filters
Glass vials
Spoons
Hemostats
63
Paraphernalia cont’d
– prevents teeth grinding
Vicks inhaler – opens nasal passages
allowing for bigger “hit” snorted
Glow sticks – to be amused by light show
when waving stick around
Pacifiers
64
Complications of Drug Abuse
Destruction
of nasal septum due to
snorting cocaine
65
Complications of Drug Abuse
Abscess at injection site and infection from
intense scratching of
imaginary “bugs”
“Meth mouth”
Enamel eroded away
due to corrosive
chemicals in product
66
Complications of Drug Abuse
Brain
changes from use of ecstasy
Left side is a normal
brain scan
Right side is 3 weeks
since last use of drug
67
Complications of Drug Abuse
indicates use of glucose – the energy
source for brain function
Decreased red = decreased glucose use =
decreased brain activity
Red
68
Assessment of Patients Abusing
Drugs
Having
high index of suspicion important
Assess thoroughly to identify the product
to determine measures to control it
Rescuer risk
Exposure risk during hands-on assessment
Disease transmission – hepatitis C, HIV
Injury due to violent patient
69
Assessment of the Patient With
Abusive Behavior with Drugs
Start
with ABC’s
CAB if patient is in arrest
Frequently
monitor for changing vital signs
Frequently monitor for change in
respiratory status
Usually perform routine medical
assessments and possible trauma
assessments
70
Management of Patients Abusing
Drugs
Protect
rescuer safety first then patient
Most field care is supportive and based on
signs and symptoms evident
Consider use of law enforcement support
for violent patients
Violent patient may need sedation as soon
as possible
Agitation and increased activity could worsen
the patient condition
71
Adult Altered Mental Status SOP
Consider
etiology
Diabetes
Drug Overdose
Poisoning
Alcohol related
Stroke
Adult
Routine Medical Care
Immobilize C-spine as indicated
72
Adult Altered Mental Status SOP
Obtain
blood glucose level and record
If <60 administer D50% - 50 ml IVP/IO OR
Glucagon 1 mg IM/IN
If
not alert, respirations decreased or
narcotic overdose suspected
Narcan 2 mg IN/IVP/IO every 5 minutes as
needed to achieve desired effect
Improvement in ventilatory status
Max total 10mg
73
Adult Altered Mental Status SOP
Note:
Attempt to identify substance(s) involved
Transport with patient any containers of
medications found at scene if not a safety risk
Consider use of restraints prior to
administration of Narcan
74
Pediatric Altered Mental Status
Same
as Adult SOP except
IV fluid challenge listed
• 20 ml/kg
Remember frequent reassessment to note how
patient is handling the fluid challenge
Medication dosing based on pediatric weight
75
Narcan® (Naloxone)
Narcotic/opioid
antagonist
Can reverse respiratory depression
induced by exposure to narcotics
Morphine
Hydromorphine
Oxycodone
Demoral
Heroin
Paregoric
Dilaudid
Codeine
Percodan
Fentanyl
Darvon
Methadone
Talwin
Nubain
76
Narcan® (Naloxone) cont’d
Onset
within 2 minutes
Reversal effects dependent on amount of
narcotic taken
Be prepared to repeat dosage as needed to
max 10 mg IN/IVP/IO
Use
cautiously in patients with cardiac
irritability and narcotic addiction
May cause nausea, vomiting, withdrawal
symptoms, seizures
77
Opioid Withdrawal
Tachycardia
Hypertension
Anxiety
Dilated
Rare but possible:
seizures
stroke
dysrhythmia
pupils
Sweating
Vomiting/diarrhea
Rhinorrhea – runny nose
Piloerection - goose bumps
Yawning
78
Region X SOP – Behavioral
Emergencies
Establish
scene & personal safety
Call law enforcement as appropriate
Determine & document if patient threat to
self/others/unable to care for self
Attempt to verbally calm patient
Restrain as necessary and document
Reason, type, time, response
79
Region X SOP - Behavioral
Emergencies
Consider
medical etiology
Hypoxia
Substance abuse/overdose
Excited delirium/Hyperthermia
Neurological disease (CVA, bleed)
Metabolic problem (hypoglycemia)
Adult/Pediatric
Routine Medical Care
If pediatric patient, contact Medical
Control for med orders
80
Region X SOP - Behavioral
Emergencies
Note:
All ED’s in Region X are able to accept
patients with behavioral issues
All patients must be medically screened prior
to admission to psych unit
Bring
to ED any containers found at scene
that do not pose a risk
Contact Medical Control in all instances
where a refusal of transport is being
considered
81
Region X SOP - Behavioral
Emergencies
For
Versed 2 mg IN
May repeat 2 mg IN every 2 minutes
Titrate to desired effect
Max up to 10mg
For
severe anxiety/agitation
additional sedation if required
Valium 5 mg IVP over 2 minutes
Repeat as needed to max total 10 mg
Or Valium 10 mg IM
82
Capnography – A Useful Tool
Carbon
dioxide (CO2) is a trace gas
Produced as a by-product in the body and
delivered to the lungs
Detectable in exhaled air
Measured as a partial pressure in mmHg
PETCO2
In
normal conditions, PETCO2 35-40mmHg
Correlates with cardiac output
83
ETCO2 vs PETCO2
ETCO2
detectors
Colormetric qualitative devices
• Indicates presence/absence of CO2
exhaled at end of the breath
CO2 is present or not
PETCO2
Quantitative device
• Indicates a precise level of
measurable CO2 exhaled
at the end of the breath
84
Capnography Usefulness in Patient
Assessment
Monitors quality of CPR
Can optimize the compression depth and rate
If PETCO2 drops below 10mmHg, evaluate to improve
CPR technique
Indicates correct placement of endotracheal
tube
Can detect return of spontaneous circulation
(ROSC)
If PETCO2 abruptly increases toward the norm of 3540mmHg, reassess the patient for ROSC (pulse)
85
Capnography in Patient
Assessment
What is your interpretation?
Normal waveform; CO2 exhaled at levels of 3545mmHg
Is intervention required?
No
86
Capnography in Patient
Assessment
What is your interpretation?
Hyperventilation – blowing off CO2 therefore CO2
Is intervention required?
Talk patient through slowing down their rate
If bagging, slow down your rate – you are
hyperventilating the patient!
87
Capnography in Patient
Assessment
What is your interpretation?
Hypoventilation, CO2 levels, is present possibly due
to CNS depression, narcotic overdose or heavy
sedation
Is intervention required?
Yes, assist the patient’s ventilations
88
Capnography in Patient
Assessment
What is your interpretation?
“Shark fin” appearance indicating obstruction during
exhalation
Seen in asthma, COPD, airway obstruction
Is intervention required?
Yes, bronchodilators or suctioning may be required
89
Capnography in Patient
Assessment
Pt is intubated. What is your interpretation?
Flattening waveform indicates esophageal intubation;
no measurement of exhaled CO2
Is intervention required?
Yes, extubate, hyperventilate, and reintubate
90
Case Scenario Discussion
Review
the following cases
Determine your general impression
Determine appropriate course of action
Discuss anything unique to the type of call
encountered
91
Case Scenario #1
EMS
called to the scene for a 2 year-old
found floating in the bathtub
They were only out of sight “a few seconds”
Pediatric assessment triangle
Limp, flaccid, no activity
No respiratory movement noted
Cyanotic
Consider
c-spine precautions
92
Case Scenario #1 cont’d
CAB
Circulation-airway-breathing
5-10 second pulse check- pulse rapid, regular,
weak
Respirations absent
What
assessment
intervention is required now?
Support ventilations via BVM
• 1 breath every 3-5 seconds (12-20/minute) in peds
patient
• Auscultate breath sounds
93
Case Scenario #1 cont’d
Assess
for possible trauma
Place on the monitor
Interpretation?
• Sinus tachycardia
94
Case Scenario #1 cont’d
What
is the rate of ventilation via BVM?
1 breath every 3- 5 seconds (12 - 20/minute)
What
is the rate of ventilations via an
advanced airway (ie: ETT or King airway)?
One breath every 6-8 seconds (8 – 10 / minute)
How
is ETT placement confirmed?
Direct visualization, bilateral rise & fall of chest,
5 point auscultation, ETCO2 yellow, waveform
capnography
• PETCO2 35 – 45 mmHg noted in perfusing patient
95
Case Scenario #2
EMS
called to a private residence for a 32
year-old found unresponsive.
Last seen 4-6 hours ago
Found lying on the floor pale, dry, vomitus
evident
Pulse present but weak
Respirations 4/minute with gasping
Start thinking causes – AEIOU-TIPS
Hx: Valium, Tylenol, ETOH overdose today
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Case Scenario #2
What
720 – 1240 mg/kg
150#
constitutes an overdose of Valium?
= 68 kg
68kg x 720mg = 49,090 mg
• Or 9,818 - 5 mg tablets
68kg x 1240 mg = 84,320 mg
• Or 16,864 – 5 mg tablets
Hard
to OD on valium alone
Mixing Valium with alcohol becomes the
hazard
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Case Scenario #2 cont’d
Assessments/interventions
Clear airway
• Suction, positioning, document presence of
vomitus – aspiration likely
Support ventilations
• BVM 1 breath every 5-6 seconds in adults
Prepare to protect airway
Suctioning
Positioning
Advanced airway
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Case Scenario #2 cont’d
IV
access
If unable to establish peripheral, then what?
• IO – pre-tibial site initial preferred site
How do you confirm IO placement?
• Needle stands up
• Flushes easily and without infiltration
• Fluid flows with pressure bag
Obtain
blood glucose level
72
99
Case Scenario #2 cont’d
Administer
2mg IN/IVP/IO every 5 minutes; max 10 mg
What
Narcan
is the desired effect of Narcan?
Improve ventilations
Don’t necessarily need patient fully awake
Consider application of restraints prior to
administration of Narcan
If
the patient is tubed, what is ventilation
rate?
1 breath every 6-8 seconds (all ages)
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Case Scenario #2 cont’d
After
IO insertion and fluid infusion begun,
patient becoming more restless and
moving leg around; pulse rate up; facial
grimacing present
What might be the problem?
Pain due to infusion via IO route
Intervention?
Slow down fluid rate; Lidocaine 50 mg over 60
seconds, wait 60 seconds then begin infusion
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Case Scenario #3
EMS
called for adult found by friends
floating in the lake; unknown submersion
time
0-0-0
Assessment/treatment required?
Spinal immobilization
Immediate CPR – Perform CAB assessment
Place patient on monitor
102
Case Scenario #3
What
VF
What
is the rhythm?
do you do?
Administer 1st shock and finish assessing
patient to determine further course of action
103
Case Scenario #3 cont’d
Decision
Is patient normothermic or hypothermic?
• If normothermic, treat dysrhythmia
• If hypothermic, determine if extremities flexible
If hypothermic, place warm packs
• Axilla, groin, neck, thorax
• If extremities can be flexed, administer medications
but extend time between to longest available
Repeat defibrillation as core temp rises
• If extremities cannot be flexed, then hold meds
Limit defibrillation to 1 attempt
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Case Scenario #3 cont’d
Cannot
withdraw resuscitative efforts until
patient warmed
Adult CPR ratios?
1 & 2 man CPR – 30:2
If pulse present but not breathing, support
ventilations 1 every 5 - 6 seconds via BVM
• 1 every 6-8 seconds if via advance airway
If no pulse and advanced airway placed,
ventilate 1 every 6 - 8 seconds
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Case Scenario #4
EMS
called for 18 year old running thru
streets naked and screaming
PD on scene
What kind of restraints do you have?
Verbal de-escalation
Manual control
Soft restraints
Handcuffs only used by PD
• If handcuffs used, PD must accompany EMS in the
ambulance to the hospital
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Case Scenario #4
Consider
etiology
Hypoxia
Substance abuse / OD
Excited delirium / hyperthermia
Neuro disease ( i.e.; CVA, intracerebral bleed)
Metabolic problems (i.e.: hypoglycemia)
If
peds patient contact Medical Control for
medication orders
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Case Scenario #4 cont’d
For
If
severe anxiety / agitation
Versed 2 mg IN
Can repeat every 2 min until a max of 10 mg
additional sedation is required
Valium 5 mg IVP over 2 minutes
Repeat as needed
Max total dose 10 mg
Or give Valium 10mg IM
108
Reminder: Preceptor Role
Your
role is to nurture, guide, grow the
student in their role
Provide constant feedback and support
Student expected to increase their
knowledge and skill level over time
increasing their independence
Dynamic process changing over time
Student can only improve with appropriate
direction and guidance
109
Review of Ventilation Rates
Inadequate
breathing with pulse
Infant & child rescue breaths via BVM
• 1 every 3-5 seconds (12 - 20 / minute)
Adult
• 1 every 5 -6 seconds (10 – 12 / minute)
Ventilations
with advanced airway in place
Infant, child, adult
• 1 breath every 6 -8 seconds (8 – 10 / minute)
• If during CPR, asynchronous with chest
compressions
110
Hazards of Hyperventilation
As
respiratory rates increase above
recommended, the patient will be
hyperventilating
Hyperventilation blows off CO2 decreasing
levels
Vessels reflexively vasoconstrict
Impeded blood flow decreases cerebral
perfusion
Cell
death becomes secondary insults
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Bibliography
Aehlert, B. ECG’s Made Easy. 4th Edition. Mosby Jems.
2011.
Bledsoe, B., Porter, R., Cherry, R. Paramedic Care
Principles & Practices, 4th edition. Brady. 2013.
Region X SOP’s; IDPH Approved January 6, 2012.
2010 American Heart Association Guidelines for CPR
and ECC
http://www.sgna.org/issues/sedationfactsorg/medications
.aspx
http://www.tustinca.org/weblink8/0/doc/218449/Page1.as
px
http://www.who.int/water_sanitation_health/diseases/dro
wning/en/
112
Bibliography
http://emedicine.medscape.com/article/772753-overview
www.illinoispoisoncenter.org
Poison Control – 1-800-222-1222
Abcpoolsafety.org
Swimforlife.com
http://www.nlm.nih.gov/medlineplus/ency/article/000774.
htm
http://www.dentalgentlecare.com/drug_use_&_oral_clues
.htm
http://www.drugfreeworld.org/drugfacts/crystalm
eth/the-stages-of-the-meth-experience.html
http://www.huffingtonpost.com/2013/11/07/bath-saltszombies-video_n_4234691.html
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