Transcript Assessment
Nursing Care & Interventions for the
Client with Disorders of the Thyroid
Gland
Keith Rischer RN, MA, CEN
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Today’s Objectives…
Compare and contrast pathophysiology &
manifestations of thyroid/parathyroid gland
dysfunction.
Identify, nursing priorities, and client education
associated with thyroid/parathyroid gland dysfunction.
Interpret abnormal laboratory test indicators of
thyroid/parathyroid gland dysfunction.
Analyze assessment to determine nursing diagnoses
and formulate a plan of care for clients with
thyroid/parathyroid gland dysfunction.
Describe the mechanism of action, side effects and
nursing interventions of pharmological management
with thyroid/parathyroid gland dysfunction.
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Thyroid Glands:Patho
Thyroid gland
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Thyroxin (T3)
Triiodothyronine (T4)
Functions
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•
•
Controls metabolism
of all cells
Regulate protein,
CHO, fat metabolism
Exert
chronotropic/inotropic
cardiac effects
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Hyperthyroidism:Causes
Graves disease
Goiter
T3 Thyrotoxicosis
Thyroid cancer
Tumors in body
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Hyperthroidism: Assessment
chart 67-1 p.1482
Early
• Visual changes
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Blurred
Double vision
Photophobia
Heat intolerance/diaphoresis
Weakness, fatigue
Other
exopthalmos
Tachycardia or systolic
hypertension
Agitation, tremors, anxiety
Palpitations
Increased libido, amenorhea
Restlessness, confusion,
psychosis
seizures
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Hyperthyroidism:Diagnostic Tests
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Serum thyronine (T4)
Serum Triodothyronine (T3)
Thyroid stimulating hormone (TSH)
low
in Graves
high in secondary (due to pituitary disorder)
•
Thyroid scan
increase
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radioactive iodine uptake
Ultrasound
ECG
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Nursing Diagnostic Priorities
Imbalanced nutrition…less than body requires
•
Hyperthermia r/t increased metabolic rate
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Bedding change frequently (diaphoresis)
Sponge baths
Cool environment
Fatigue r/t sleep deprivation
•
High in calories, proteins, and carbohydrates with
supplemental feedings
Encourage rest – fatigue
Keep environment quiet
Deficient knowledge
Exopthalmos
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Thyroid Crisis/Storm
Patho
• Uncontrolled hyperthyroidism
• Excess thyroid hormone release
Physical assessment
• Extreme temperature
• Hypertension
• Tachycardia
Treatment
• Inderal
• Closely monitor VS-rhythm-temp
• Fever reduction
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Hyperthyroidism:Medical Management
Antithyroid medications
• Propylthiouracal (PTU)
•
block synthesis of thyroid hormone
Iodine (SSKI)
reduce vascularity of thyroid gland
Beta blockers
Radioactive iodine therapy
• To ablate thyroid to make the pt become hypothyroid;
Taken orally
Relief of symptoms may take 6-8 weeks
•
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Hyperthyroidism:Surgical Management
Preop
care
Post op care
•ABC’s
Humidified O2
•Support
of neck with movement
& coughing
•Semi-Fowlers position
•Incisional care
Postoperative
complications
•Hemorrhage
•Respiratory
Stridor
Tracheotomy equipment readily
available
•Laryngeal
distress
nerve damage
Hoarseness/weak voice
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Hypothyroidism
Patho
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Decreased metabolism
Myxedema coma
Cellular edema
– Generalized NP edema…eyes, hands, feet, tongue
Causes
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Thyroid surgery/radioactive iodine treatment
Iodide deficiency
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Hypothyroidism: Assessment
Change
•
chart 67-5 p.1488
in sleep habits
more lethargic
Decreased
libido
Generalized weakness
Muscles aches
Cold intolerance
Constipation
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Myxedema Coma
Those at highest risk
• Surgery
• Chemo
• Withdrawal thyroid meds
Assessment
• Respiratory failure
• Hypotension
• Labs
Emergency care
• ABC’s
• Replace fluids
• Administering meds. Steroids, IV glucose, Levothyroxine sodium
(thyroid)
• Monitor Temp. & BP frequently
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Hypothyroidism: Diagnostic Tests
Laboratory
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studies
Serum T3
Serum T4
TSH
high
in primary
Low in secondary
Treat with Lifelong thyroid replacement
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Levothyroxine (Synthroid)
Assess thyroid levels. May start low to avoid
cardiac problems
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Nursing Diagnostic Priorities
1.
2.
3.
4.
5.
Decrease cardiac output
• Assess for bradycardia, dysrhythmias
• O2 if needed
Ineffective Breathing pattern
• care when giving sedation
Disturbed thought processes
• assess lethargy, memory deficit, poor attention span,
difficulty communicating
Constipation
Deficient knowledge
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Hyperparathyroidism
Parathyroid
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glands
Regulate calcium and phosphate balance
Labs
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Hypercalcemia and hypophosphatemia
Causes
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Tumor
Chronic renal failure
Vit. D deficiency
Neck trauma or radiation
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Hyperparathyroidism: Assessment
Bone fractures from demineralization from bones
Recent weight loss
Arthritis
Psychological distress
History of Radiation to neck
GI
• N/V, diarrhea, constipation
Renal stones
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Hyperparathyroidism: Medical Management
Diet
• restrict Calcium…esp milk products
Medications
• Lasix
Increased excretion of calcium
• Phosphates
Inhibits bone resorption and interferes with
calcium absorption
• Calcitonin
Use to decrease skeletal calcium release
Hyperparathyroidectomy
• Same
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Hyperparathyroidism:
Nursing Interventions
Hydration
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(strict I & O)
IV saline in large amounts and lasix to excrete calcium
Assess for Congestive heart failure R/T fluid overload
Cardiac monitoring
Serum Calcium levels need to be done frequently
Educate client to report N/V, palpations, numbness
Care to reduce fractures – lift gently
•
Ambulation helps prevent demineralization
Observe for renal calculi
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