Energy systems and fitness
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Transcript Energy systems and fitness
LESSON ONE
AMINISTRATION
COMPONENTS OF FITNESS
AEROBIC FITNESS
VO2 MAX
ENERGY SYSTEMS
ONSET OF BLOOD LACTIC ACID (OBLA)
COMPONENTS OF
FITNESS
CARDIORESPIRATORY
ENDURANCE
MUSCULAR
STRENGTH
MUSCULAR
ENDURANCE
BODY
COMPOSITION
FLEXIBILITY
AEROBIC ACTIVITY
USES LARGE MUSCLE GROUPS
RHYTHMIC
CONTINUOUS/INTERVAL
RAISES HR TO TRAINING LEVEL
GREATER THAN 20 MINUTES
OXYGEN
CONSUMPTION
VO2 MAX:
THE MAX AMOUNT OF O2 THAT
THE BODY IS ABLE TO UTILIZE PER MINUTE OF
PHYSICAL ACTIVITY
VO2 MAX IS CONSIDERED THE SINGLE, BEST
INDICATOR OF A PERSONS LEVEL OF AEROBIC
FITNESS
VO2 MAX OCCURS WHERE A FURTHER
INCREASE IN WORK IS NOT ACCOMPANIED BY
AN ADDITIONAL INCREASE IN O2
CONSUMPTION
STROKE VOLUME
sv
VOLUME
OF BLOOD
PUMPED PER BEAT
CARDIAC OUTPUT
VOLUME
OF BLOOD
PUMPED PER MINUTE
CO = HR X SV
FICK PRINCIPLE
THE AMOUNT OF OXYGEN USED IS A
FUNCTION OF HOW MUCH BLOOD IS
SENT TO MUSCLE AND HOW MUCH O2
IS IN THE BLOOD
(A-V) O2 DIFFERENCE
FICK EQUATION:
VO2 = CARDIAC
OUTPUT
X
(A-V) 02
DIFFERENCE
GAS TRANSPORT
AND
EXCHANGE
DIFFUSION
OXYGEN
HEMOGLOBIN
CARBON
DIOXIDE
CARBON
MONOXIDE
CHANGES IN OXYGEN
TRANSPORT CAPACITY
CAUSES:
LOSS
OF BLOOD
LACK
OF OXYGEN IN AIR
BLOCKADE
ANEMIA
OF Hb
SOURCES OF ENERGY
CARBOHYDRATE
FAT
PROTEIN
ATP
ADENOSINE
TRIPHOSPHATE
ENERGY SYSTEMS
PHOSPHAGEN (ATP + CP)
ANAEROBIC GLYCOLYSIS
AEROBIC RESPIRATION
–AEROBIC GLYCOLYSIS
–BETA OXIDATION
ATP + CP
CREATINE PHOSPHATE (CP):
HIGH ENERGY COMPOUND
RAPID ENERGY PRODUCTION
OCCURS WITHOUT OXYGEN
IMMEDIATE ENERGY RESERVE
30 SECONDS HIGH INTENSITY
ANAEROBIC
GLYCOLYSIS
SPLITTING OF GLUCOSE
WITHOUT OXYGEN
BRIDGES THE GAP BEGINNING OF EXERCISE
PRODUCES HIGH LACTIC
ACID CONCENTRATIONS
2-3 MINUTES OF INTENSE
EXERCISE
AEROBIC GLYCOLYSIS
BEGINS WITH GLUCOSE
FORMS PYRUVIC ACID
IN THE PRESENCE OF O2 PYRUVIC
ACID IS METABOLIZED INTO CO2
AND H2O
KREBS CYCLE - CHEMICAL
BREAKDOWN OF PYRUVIC ACID
BETA OXIDATION
FAT METABOLISM
REQUIRES OXYGEN
LONG DURATION EXERCISE
LOWER INTENSITY
SUBSTRATE
UTILIZATION
DEPENDENT ON 3 FACTORS:
INTENSITY/DURATION
STATE
DIET
OF TRAINING
O.B.L.A
LACTIC ACID ACCUMULATION
EXPRESSED AS A PERCENTAGE
OF MAXIMAL O2 UPTAKE
NORMALLY OCCURS BETWEEN
55% AND 65% OF VO2 MAX
OCCURS AT 80% VO2 MAX IN
HIGHLY TRAINED ATHLETES
REMOVAL OF LACTIC
ACID
OXIDATION TO CO2 AND H2O
LOSS IN URINE AND SWEAT
CONVERSION TO GLUCOSE OR
GYCLOGEN
OBLA “FACTS”
INTENSITY
DEPENDENT
CAN
BE TRAINED
CAN
BE POSTPONED
LESSON TWO
HEART RATE RESPONSE
HEART RATE MONITORS
MAX HEART RATE/RECOVERY RUN
HEART RATE
RESPONSE
HEART RATE AND OXYGEN UPTAKE
HAVE A LINEAR RELATIONSHIP
HEART RATE IS CONSIDERED A GOOD
WAY TO ESTIMATE EXERCISE INTENSITY.
DETERMINING TRAINING
HEART RATE
ESTIMATE MAXIMUM HR (MHR)
220 - AGE = MHR
DETERMINE HEART RATE RESERVE
HRR = MHR - RESTING HEART RATE
TRAINING HEART RATE (THR)
(____% X HRR) + RHR = THR
RATE OF PERCEIVED
EXERTION
6
7 VERY VERY LIGHT
8
9 VERY LIGHT
10
11 LIGHT
12
13 SOMEWHAT HEAVY
14
15 HEAVY
16
17 VERY HEAVY
18
19 VERY VERY HEAVY
20
LESSON THREE
FACTORS AFFECTING AEROBIC
EXERCISE
SHORT TERM RESPONSES
LONG TERM ADAPTATIONS
FACTORS AFFECTING
AEROBIC EXERCISE
MODE OF EXERCISE
HEREDITY
POTENTIAL FOR FITNESS
GENDER
AGE
BODY COMP0SITION
LEVEL OF ACTIVITY
EXERCISE PRESCRIPTION
FITT PRINCIPLE
FREQUENCY
3 - 5 TIMES PER WEEK
INTENSITY
60%-90% OF MHR
12-14 RPE SCALE
50%-80% VO2 MAX
TIME
20-30 MINUTES
TYPE
LARGE MUSCLE GROUPS
RHYTHMIC
CONTINUOUS
SHORT TERM RESPONSES
TO AEROBIC EXERCISE
INCREASE IN PULMONARY
VENTILATION (RESPIRATION)
INCREASE IN HEART RATE
INCREASE IN STROKE VOLUME
INCREASE IN CARDIAC OUTPUT
INCREASE IN MUSCLE TEMPERATURE
INCREASE IN BLOOD FLOW TO HEART
LONG TERM ADAPTATIONS TO
AEROBIC EXERCISE
DECREASE
IN RHR
LOWER HR AT A GIVEN WORKLOAD
DECREASE IN HR RECOVERY TIME
INCREASE NUMBER AND SIZE OF
FUNCTIONAL CAPILLARIES
INCREASE IN CARDIAC OUTPUT
INCREASE IN STROKE VOLUME
INCREASE IN THE NUMBER AND SIZE
OF MITACHONDRIA
INCREASE IN THE ABILITY TO MOBILIZE
AND UTILIZE FAT
DECREASE IN BLOOD LIPIDS
DECREASE IN RESTING BLOOD
PRESSURE
LONG TERM ADAPTATIONS TO
AEROBIC EXERCISE
MUSCULOSKELATAL BENEFITS
INCREASED MUSCULAR STRENGTH
INCREASED CAPILLARY DENSITY IN MUSCLE
STRENGTHENING OF TENDONS, LIGAMENTS,
AND JOINTS
COUNTERACTS OSTEOPOROSIS
MISCELLANEOUS BENEFITS
ENHANCED TOLERANCE TO HEAT
RELEASE OF ENDORPHINES “RUNNERS HIGH”
LESSON FOUR
AEROBIC FITNESS ASSESSMENT
EXERCISE PRESCRIPTION
AEROBIC FITNESS
ASSESSMENT
VO2 MAX:
THE MAX AMOUNT OF O2 THAT
THE BODY IS ABLE TO UTILIZE PER MINUTE OF
PHYSICAL ACTIVITY
VO2 MAX IS CONSIDERED THE SINGLE, BEST
INDICATOR OF A PERSONS LEVEL OF AEROBIC
FITNESS
VO2 MAX OCCURS WHERE A FURTHER
INCREASE IN WORK IS NOT ACCOMPANIED BY
AN ADDITIONAL INCREASE IN O2
CONSUMPTION
EXPRESSING VO2
ABSOLUTE
VO2/VO2 MAX:
L/min
RELATIVE
VO2/VO2 MAX:
ml O2/Kg/min
ASSESSING VO2 MAX
MAXIMAL TEST: REQUIRES A
PERSON TO EXERCISE TO
EXHAUSTION
SUBMAXIMAL TEST:MEASURES AN
INDIVIDUALS RESPONSE AT
SUBMAXIMAL EXERCISE.
VO2 MAX IS PREDICTED FROM
EXERCISE HEART RATE FOR THE
GIVEN SUBMAXIMAL EXERCISE
ASSESSING VO2 MAX
LAB
FIELD
MAX
SUBMAX
BENEFITS OF TESTING
ASSESSES STRENGTHS AND
WEAKNESSES
PROVIDES BASELINE DATA
PROVIDES FEEDBACK
PROVIDES HEALTH
ASSESSMENT
PROVIDES UNDERSTANDING
TESTING WILL NOT
PREDICT FUTURE “GOLD
MEDALISTS”
SIMULATE ACTUAL PHYSICAL
DEMANDS OF SOME SPORTS
2 MILE RUN TEST
MEN:
VO2MAX = 99.7 - (3.35 X TIME)
WOMEN:
VO2MAX = 72.9 - (1.77 X TIME)
PRINCIPLES OF
EXERCISE
OVERLOAD
PROGRESSION
RECOVERY
SPECIFICITY
REGULARITY
VARIETY
BALANCE
3 PHASES OF AN AEROBIC
EXERCISE PROGRAM
PREPARATORY
PHASE
USUALLY LASTS 4-6 WEEKS
CONDITIONING
PHASE
USUALLY
LASTS 12-20 WEEKS
INTENSITY 70 - 80 HRR
DURATION/FREQUENCY INCREASED
BY 10% PER WEEK
MAINTENANCE
PHASE
BEGINS 6 MONTHS AFTER STARTING
PROGRAM
CONTINUES FOR A LIFETIME
PHYSIOLOGICAL
EFFECTS OF A WARM UP
INCREASE IN BODY TEMPERATURE
INCREASE IN BLOOD FLOW TO
MUSCLES AND HEART
DECREASED MUSCLE CONTRACTION
AND REFLEX TIME
DECREASES CHEMICAL REACTION
TIME
PHYSIOLOGICAL EFFECTS OF
A COOL DOWN
DECREASED LACTIC ACID
LEVELS
PREVENTS BLOOD POOLING
STRETCHING WARM MUSCLES
IMPROVES FLEXIBILITY
DECREASED CHANCES OF
CARDIAC IRREGULARITIES
EXERCISE ADHERENCE
FUN
CROSS-TRAINING
REGULARITY
PROPER
EQUIPMENT
TRAINING PARTNER
SET GOALS
AVOID OVERTRAINING
KEEP A LOG
ASSESSMENTS
MONITOR HEALTH
LESSON FIVE
FATIGUE
DELAYED ONSET OF MUSCLE
SORENESS (DOMS)
CONTRAINDICATIONS
ENVIRONMENTAL
CONSIDERATIONS
STADIUM STAIR CLIMBING
MECHANISMS OF FATIGUE
POSSIBLE CAUSES:
DECREASED ATP SUPPLY
INCREASE IN LACTIC ACID
DEHYDRATION
MOTIVATION
HYPERTHERMIA
DEPLETION OF GLYCOGEN
ELECTROLYTE IMBALANCE
POSSIBLE CAUSES OF
MUSCLE SORENESS
ACUTE: LACK OF BLOOD FLOW (O2)
AND GENERAL FATIGUE OF MUSCLES
DELAYED ONSET OF MUSCLE
SORENESS (D.O.M.S.):
12
HOURS AFTER EXERCISE
MAY LAST 2-4 DAYS
TEARS TO MUSCLE AND
CONNECTIVE TISSUE
INCREASE FLUID RETENTION
MUSCLE SPASMS
NOT A SPRAIN OR INJURY
PREVENTION: WARM UP, START
SLOW, COOL DOWN PROPERLY
CONTRAINDICATIONS
OF EXERCISE
RECENT HEART ATTACK
UNSTABLE ANGINA PECTORIS
ABNORMAL HEART ACTIVITY
PAIN IN CHEST, ARMS FOLLOWING ACTIVITY
DIZZINESS, LIGHT HEADINESS
LACK OF COORDINATION, CONFUSION, COLD
SWEATING
ILLNESS, PARTICULARLY VIRAL INFECTIONS
SYMPTOMS OF
OVERTRAINING
MUSCLE SORENESS
HEADACHES, SORE THROAT,
MILD COLD
IRRITABILITY
INSOMNIA
LACK OF INTEREST IN DAILY
ACTIVITIES
LOSS OF APPETITE
SUDDEN DROP IN WEIGHT
CONSTIPATION OR DIARRHEA
SKIN ERUPTIONS
ABNORMALLY HIGH RHR
RISK FACTORS
AGE
HEREDITY
GENDER
SMOKING
OBESITY
LACK OF EXERCISE
HIGH CHOLESTEROL
HIGH BLOOD PRESSURE
ABNORMAL EKG
STRESS
DIABETES
EXERCISING IN THE
COLD
DRESS IN LAYERS
AVOID PROFUSE SWEATING
AVOID WIND CHILL FACTOR
DRINK PLENTY OF WATER
____________________________
HYPOTHERMIA
FROSTBITE
MAJOR FORMS OF
HEAT ILLNESS
HEAT CRAMPS: MUSCLE PAIN AND
SPASM
HEAT EXHAUSTION: WEAK, RAPID
PULSE, LOW BLOOD PRESSURE,
HEADACHE, AND DIZZINESS
HEAT STROKE: CEASATION OF
SWEATING, DRY, HOT SKIN AND VERY
HIGH BODY TEMPERATURE - THE MOST
SERIOUS AND COMPLEX HEAT
PROBLEM
FACTORS TO MODIFY
HEAT TOLERANCE
GRADUALLY ACCLIMATIZE
DRINK PLENTY OF WATER
BEFORE DURING AND AFTER
ALLOW FOR EVAPORATION LIMITED IN HUMID WEATHER
REPLACE ELECTOROLYTES
LOST THROUGH SWEATING
AVOID EXERCISE DURING
HOTTEST TIME OF DAY
EFFECTS OF
DEHYDRATION
REDUCED CIRCULATORY
CAPACITY
REDUCED MAX CARDIAC
OUTPUT
ALTERED AUTONOMIC NERVOUS
CONTROL OF SWEAT GLANDS
REDUCED WORK CAPACITY
FLUID REPLACEMENT
WITH CARBOHYDRATE
INCREASE
IN BLOOD
GLUCOSE
INCREASE
IN GLUCOSE
UTILIZATION BY MUSCLE
INCREASE
IN ENDURANCE
BENEFITS OF FLUID
REPACEMENT
IMPROVED REGULATION OF BODY
TEMPERATURE
INCREASED SWEAT RATE
DECREASED BODY CORE TEMP
INCREASED PLASMA VOLUME
INCREASED STROKE VOLUME
INCREASED PERFORMANCE
LESSON SIX
RUNNING BASICS
DIFFERENT RUNNING
MODALITIES/TRAINING METHODS
INTERVAL RUNNING
RUNNING
MODALITIES AND
TRAINING METHODS
LONG SLOW DISTANCE
FARTLEK “SPEED PLAY”
FAST, CONTINUOUS
INDIAN RUNS
INTERVALS
INTERVAL TRAINING
DEFINITION: SERIES OF REPEATED BOUTS
OF EXERCISE ALTERNATED WITH PERIODS
OF RELIEF
VARIABLES
(TO EMPHASIZE A PARTICULAR ENERGY SYSTEM)
RATE AND DISTANCE OF WORK
(BASED ON PERFORMANCE TIME)
NUMBER OF REPETITIONS
RELIEF TIME BETWEEN INTERVALS
FREQUENCY OF TRAINING PER WEEK
BENEFITS OF
INTERVAL TRAINING
ALLOWS FOR QUALITY, HIGH
INTENSITY WORK
APPLIES PRINCIPLE OF
SPECIFICITY
CAN STRESS ALL ENERGY
SYSTEMS
TRAINS NERVOUS SYSTEM
FORMULA FOR
DETERMINING INTERVAL
PACE (4 -8 X 400M)
MILE TIME = 8:00 MINUTE
400 METER = 2:00 MINUTE
SUBTRACT 1-4 SECONDS :
= 1:56-1:59
LESSON 14
NUTRITION BASICS
SUBSTRATE
CARBOHYDRATES
FATS
PROTEINS
NUTRITION
PROTEIN: 12% OF DAILY CALORIC
INTAKE
FAT: NOT TO EXCEED 30% OF DAILY
CALORIC INTAKE
CARBOHYDRATE: AT LEAST 50%-60%,
CONSIDER 70%-75% OF DAILY CALORIC
INTAKE
MAJOR SOURCE OF ENERGY
SPARES BREAKDOWN OF PROTEIN
METABOLIC PRIMER FOR FAT
IMPORTANT FOR ATHLETIC PERFORMANCE
RELEASED WITHIN MUSCLE 3 TIMES AS FAST
AS FAT
GENERAL GUIDELINES
FOR A HEALTHFUL DIET
EAT A VARIETY OF FOODS
AVOID FATS AND CHOLESTEROL
EAT FOODS HIGH IN STARCH AND
FIBER
AVOID EXCESSIVE SUGAR AND
SODIUM
MAINTAIN ADEQUATE CALCIUM
INTAKE
MAINTAIN RECOMMENDED BODY
WEIGHT
USE ALCOHOL IN MODERATION
DRINK PLENTY OF WATER