teixeira---cardiac-arrhythmia-in-children
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Cardiac arrhythmias in children:
emphasis on SVT
Definition, identification and management
Otto H. P. Teixeira, MD, FRCPC, FACC
Conduction system of the heart
What is an
arrhythmia?
An arrhythmia is an abnormal heart rhythm
• Sinus arrhythmia
• Premature ventricular beats (PVCs)
• Premature atrial beats (PACs)
What is an
Arrhythmia?
• An arrhythmia is an
abnormal heart rhythm
What is an
Arrhythmia?
• An arrhythmia is an
abnormal heart rhythm
• Bradycardia is when the
heart rate is too
slow
What is an
Arrhythmia?
• An arrhythmia is an
abnormal heart rhythm
• Bradycardia is when the
heart rate is too
slow
• Tachycardia is when the
heart rate is too
fast for the age
Normal heart rates in children
Nelson Textbook of Pediatrics 19th ed.
Elsevier/Saunders 2011: 1613-16
AGE
Premature
0-3 mo
3-6 mo
6-12 mo
1-3 yr
3-6 yr
6-12 yr
12+yr
HEART RATE
120-170*
100-150*
90-120
80-120
70-110
65-110
60-95
55-85
Cardiac arrhythmia in children
What's "too slow" ?
• newborn: less than 80 beats a minute.
• a trained teenage athlete: 50 beats a minute
Cardiac arrhythmia in children
Symptoms of bradycardia
•
•
•
•
•
Fatigue
Dizziness
Lightheadedness
Fainting or near-fainting spells
In extreme cases, cardiac arrest occur
Supraventricular tachycardia (SVT)
All forms of tachycardia except VT *
* Van Hare G. Supraventricular tachycardia. In: Kliegman, Stanton, St. Geme,
Schor, Behrman, eds. Nelson Textbook of Pediatrics 19th ed.
Elsevier/Saunders 2011: 1613-16
SVT in children
Most common form of tachycardia in
children:
1 : 250-1000 (Gillette & Garson)
SVT in children
Most common form of tachycardia in
children:
1 : 250-1000 (Gillette & Garson)
Narrow QRS complexes often with no
discernible “P” wave
HR: 150-300 bpm and unvarying
Mechanisms of Arrhythmias
Classifying tachycardia:
frequency
Common:
AV reentry
Less common:
atrial
Uncommon:
AV node reentry
Rare:
other
SVT in children
Re-entrant:
with accessory pathway
without pathway
SVT in children
Re-entrant:
with accessory pathway
without pathway
Ectopic or automaticity: AET, JET
AV re-entrant tachycardias
Reentry: most common etiology in children
AVRT, AVNRT: age dependent
Narrow QRS: usual
1:1 AV conduction: usual
Sudden onset and cessation
SVT in children
Etiology
Reentry:
AVRT: most common in infants
Accessory connection anywhere
in the AV ring - WPW, Mahaim
tracts, others
AVNRT: adolescents and adults
AV peri-nodal area: dual AV node
“slow-fast” AV node tracts
SVT in children
HR 215, occasional “P wave” seen
SVT in children
HR 187 bpm: P waves not discernible
AV re-entrant tachycardias
• WPW: 1 : 1000
• 25 % of SVT
orthodromic: narrow QRS during
tachycardia
antidromic: broad QRS
(indistinguishable from
VT)
WPW
AV re-entrant tachycardias
Orthodromic tachycardia
using an accessory
pathway in WPW
Activation: AV node, HisPurkinje system, ventricle,
accessory pathway, atria. P
wave closely follows the
QRS PR > RP
AV re-entrant tachycardias
Antidromic tachycardia:
antegrade conduction may lead
to atrial fibrillation and VF:
risk of SCD
AV nodal re-entrant tachycardia:
AVNRT
Dual pathways within the AV node
“slow-fast” AV nodal tracts
More common in adolescents and adults
May cause syncope
May respond to beta blockers and
ablation*
* Teixeira OH, Bilaji, Gillette PC. RF catheter ablation of
atrioventricular nodal reentrant tachycardia in children. PACE 1994;
17:1621-1626.
Management: SVT in children
Evaluate
General: IV, O2, capillary filling, BP
12-lead ECG (may double speed):
REENTRY?
Stable?
1. Vagal maneuvers: infant - modified diving
reflex (ice to face); older - gag, head
immersion in water, Valsalva
Management (cont’d)
2. Adenosine:
0.1 mg/kg mx 6 mg
ECG rhythm should be running
Management (cont’d)
• Adenosine:
0.1 mg/kg mx 6mg. ECG rhythm
should be running
f a i l e d!?
• Check tracing: reentry? check IV
Double dose to mx. 12 mg
Management (cont’d)
3. Consider synchronized DC cardioversion:
0.5-2 Joules/kg
failed!?
• Check: equipment, paddles, position,
coupling gel, contact
• Repeat: double dose
4. Overdrive pacing: PM, esophageal lead
5. Other drugs:
• Amiodarone:
5 mg/kg IV over 20-60 min
• Procainamide:
15 mg/kg IV over 30-60 min
Question
• One month-old male with narrow QRS heart
rate of 320 bpm and tachypnea, poor
peripheral pulse. Your treatment of choice
includes all, EXCEPT:
• 1. Vagal maneuvers, ie, ice to face.
Question
• One month-old male with narrow QRS heart
rate of 320 bpm and tachypnea, poor
peripheral pulse. Your treatment of choice
includes all, EXCEPT:
• 1. Vagal maneuvers, ie, ice to face.
• 2. Adenosine 0.1-0.2 mg/Kg IV push
Question
• One month-old male with narrow QRS heart
rate of 320 bpm and tachypnea, poor
peripheral pulse. Your treatment of choice
includes all, EXCEPT:
• 1. Vagal maneuvers, ie, ice to face.
• 2. Adenosine 0.1-0.2 mg/Kg IV push
• 3. Verapamil 0.1 mg/Kg IV push
Question
• One month-old male with narrow QRS heart
rate of 320 bpm and tachypnea, poor
peripheral pulse. Your treatment of choice
includes all, EXCEPT:
• 1. Vagal maneuvers, ie, ice to face.
• 2. Adenosine 0.1-0.2 mg/Kg IV push
• 3. Verapamil 0.1 mg/Kg IV push
• 4. Synchronized DC cardioversion: 0.5-1.0 J/Kg
SVT in children
Etiology
Automaticity:
atrial ectopic tachycardia
(AET)
junctional ectopic
tachycardia (JET)
Ectopic or automaticity: AET, JET
AET
Atrial automatic focus: incessant , may be
associated with cardiac dysfunction
Insidious onset with “warm-up” and
“cool-down”
Abnormal “P” wave axis and PR interval
Rate variation
May have periods of sinus rhythm during
sleep: P changes
The P wave morphology depends on the position of the focus
May appear like sinus tachycardia: RP interval is longer than
the PR.
The P waves may be blocked (not shown).
AET
AET
• Rarely needs emergency treatment
• Adenosine may work and helps diagnosis:
P waves revealed during adenosine induced AV block
Inappropriate sinus tachycardia (Bauernfeind R et al An
Intern Med 1979; 91:702-710)
Chaotic or multifocal AET
•
•
•
•
•
•
> 3 ectopic Ps
Frequent blocked Ps
Varying PR
Occurs mostly in infants < 1 yr
Difficult single drug management
May improve spont by 3 yr of age
Chaotic or multifocal AET
•
•
•
•
•
•
> 3 ectopic Ps
Frequent blocked Ps
Varying PR
Occurs mostly in infants < 1 yr
Difficult drug management
May improve spont by 3 yr of age
JET
• Uncommon; often post-op
• EKG: narrow QRS with V rate faster than A,
and AV dissociation
• Diff. to distinguish from VT: ”Why?”
• Rates 180-240 bpm
• Congenital: rare may be seen pre-natally: HR
370 bpm
JET
First-degree AV block is
typical and 2nd-degree AVB
is common. The tachycardia
and degree of AV block are
influenced by the autonomic
tone
Lead II of a ECG from a patient
with postoperative JET. “Ps”
are marked with blue lines
and QRS complexes are in red
JET
• Difficult management: post-op
• several drugs may have to be tried in stepwise
fashion: amiodarone, propafenone, sotalol
Question
In atrial ectopic tachycardia (AET):
• 1. The etiology is automaticity
Question
• In atrial ectopic tachycardia (AET):
• 1. The etiology is automaticity
• 2. Warm-up and cool-down may be seen
Question
•
•
•
•
In atrial ectopic tachycardia (AET):
1. The etiology is automaticity
2. Warm-up and cool-down may be seen
3. P wave is present
Question
•
•
•
•
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In atrial ectopic tachycardia (AET):
1. The etiology is automaticity
2. Warm-up and cool-down may be seen
3. P wave is present
4. Blocked Ps may be seen
Question
•
•
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•
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In atrial ectopic tachycardia (AET):
1. The etiology is automaticity
2. Warm-up and cool-down may be seen
3. P wave is present
4. Blocked Ps may be seen
5. All of the above
Atrial flutter
Reentry:
• Intra-atrial reentry: note flutter waves
(“saw-tooth”), variable AV
conduction
Atrial flutter
Atrial flutter: management
• 1. Synchronized DC cardioversion: choice
treatment
• 2. Chronic flutter: risk of embolism >
anticoagulartion
• Digoxin, B-blockers, Ca blockers: delay AV
node conduction
• Other drugs used to maintain sinus rhythm:
procainamide, propafenene, amiodarone,
sotalol
Atrial flutter: management
Atrial flutter: unstable
1. Synchronized DC cardioversion: half doses
2. Calcium channel blockers:
Diltiazem: 0.25-0.35 mg/kg over 2-15
min (may repeat)
Drip: 0.5-0.15 mg/kg/h
3. Overdrive pacing: PM, esophageal lead
4. Other drugs:
procainamide, amiodarone, sotalol
Atrial flutter: management
• Neonatal flutter: digoxin for 1 yr after which
arrhythmia may not recur
Atrial fibrillation
• Uncommon
• Often associarted with atrial problem:
enlargement
• Seen in older children with atrial enlargement
or, rheumatic valve stenosis or surgery
Atrial fibrillation: management
• Rate control: Ca blockers
• No digoxin if there is WPW
• Acute:
Procainamide, amiodarone, DC cardioversion
Anticoagulation may be necesssary
Conclusions
SVT is the most common tachyarrhythmia in
children
The etiology is primarily reentry
In older children it is usually well tolerated
In infants it may be a life-threatening
emergency
GOOD LUCK !
Nodal rhythm
Note AV dissociation with “Ps” appearing from
time to time
Nodal bradycardia: HR 37bpm
3- year-old girl with hyperthyroidism (Pediatr 1997;
100.2.e11). Flutter 1:1 conduction. Note negative
flutter waves in V1-V3 and II, III, aVF
ATRIAL FLUTTER