Transcript S 2
CARDIOVASCULAR
EXAMINATION
I.U. Cerrahpaşa Medical Faculty
Department of Pediatrics
Division of Pediatric Cardiology
Prof. Dr. Ayşe Güler EROĞLU
HISTORY
Sweating
Exercise intolerance
Common respiratory tract infections
Growth retardation
Feeding difficulties
Palpitation
Dyspne
Cyanosis
Chest pain
Syncope
HISTORY
Medical history
Ilnesses
Medications
Prenatal history
Mother’s ilnesses (diabetes mellitus, lupus)
Mother’s medications
Natal history
Prematurity
Birth weight
Family history
Congenital heart diseases
Sudden death
PHYSICAL EXAMINATION
INSPECTION
General appearance
Chromosomal, hereditary, nonhereditary
syndroms
Pallor
Cyanosis
Clubbing
Neck vein distension
Left precordial bulge
PALPATION
Pulses
Volume
Rate
Rhythm
Character
Chest
Apical impulse
In newborn and infants 4. intercostal space/midclavicular line
In older children and adults 5. intercostal space/midclavicular line
Precordial activity
Thrills
VOLUME OF PULSES
Increase in pulse volume
Fever, anemia, exercise and thyrotoxicosis
Weak pulses
Low cardiac output (left heart obstructive lesions:
aortic valve atresia or stenosis)
Bounding pulses
Patent ductus arteriosus, aortic regurgitation,
large systemic arteriovenous fistula
Differences in pulse volume between
extremities
Coarctation of the aorta
OSCULTATION
Heart rate and rhythm
Heart sounds
Other sounds
Murmurs
HEART SOUNDS
First heart sound (S1): The S1 is associated with
closure of the atrioventricular valves (mitral and
tricuspid) It corresponds to the beginning of
systole.
Abnormally wide splitting: right bundle branch
block, Ebstein’s anomaly
Increased S1: Fever, anemia, excitement,
thyrotoxicosis, short PR interval, mitral
stenosis
Decreased S1: long PR interval and mitral
regurgitation
Second heart sound (S2): The S2 is associated with
closure of semilunar valves (aortic and
pulmonary). It corresponds to the beginning of
diastole. In every normal child, the s2 is split
during inspiration and single during expiration
(normal splitting of the S2).
HEART SOUNDS
Widely split S2
Right ventricle volume overload: ASD, partial
anomalous pulmonary venous return)
Right ventricle pressure overload: pulmonary
stenosis
Delay in electrical activation of right ventricle:
right bundle branch block
Early aortic valve closure: mitral regurgitation
Narrowly split S2
Pulmonary hypertension
Aortic stenosis
Paradoxically split S2
Severe aortic stenosis
Left bundle branch block
HEART SOUNDS
Single S2
Only one semilunar valve is present: aortic or
pulmonary
atresia,
persistent
truncus
arteriosus
P2 is not audible: transposition of the great
arteries, tetralogy of Fallot, severe pulmonary
stenosis
Aortic closure is delayed: severe aortic
stenosis
P2 occurs early: pulmonary hypertension
P2 increases in pulmonary hypertension and
decreases in severe pulmonary stenosis,
tetralogy of Fallot and tricuspid stenosis
HEART SOUNDS
Third heart sound (S3): The S3 is a low-frequency
sound in early diastole and is related to rapid
filling of the ventricle.
It is commonly heard in normal children and
young adults.
A loud S3 is abnormal and is audible in large
shunt VSD, congestive heart failure.
Fourth heart sound (S4): The S4 is a lowfrequency of late diastole and is rare in infants
and children.
It is always pathologic.
It is seen in conditions with decreased
ventricular compliance.
OTHER SOUNDS
Ejection clic: It follows the S1 very closely,
therefore it sounds like a splitting of the S1
Valvular aortic and pulmonary stenosis, dilated
great arteries
Midsystolic click with or without late systolic
murmur
Mitral or tricuspid valve prolapse
Opening snup: It occurs earlier than the S3 during
diastole
Mitral or tricuspid stenosis
Pericardial friction rub (frotman)
Pericarditis
Pericardial knock
Constrictive pericarditis
CHARACTERISTICS OF HEART MURMURS
Timing
Radiation
Location
Murmur
Intensity
Quality
TIMING OF HEART MURMURS
Murmurs
Systolic
Ejection
(Diamond
Crescendodecrescendo)
Regurgitant
(Holosistolic
Pansistolic)
Continuous
Early
Late
Diastolic
Early
Middiastolic
Late
Sistolic ejektion murmurs
(Diamond shaped,
crescendo-decrescendo)
Aortic stenosis
Pulmonary stenosis
Increased flow in aorta
Increased flow in pulmonary artery
Sistolic regurgitant murmurs
(Holosistolic, pansistolic)
Ventricular septal defect
Mitral regurgitation
Tricuspid regurgitation
Late sistolic murmurs
Mitral valve prolapse
Tricuspid valve prolapse
Early diastolic murmurs
(Decrescendo)
Aortic regurgitation
Pulmonary regurgitation
Middiastolic murmurs
(Flow murmurs)
Increased flow across the atrioventricular
valves in patients with ASD, VSD, PDA
Late diastolic murmurs
(Presistolik)
Mitral valve stenosis
Tricuspid valve stenosis
Continuous murmurs
Arterial
PDA
Coronary artery
fistula
Pulmonary AV fistula
Sistemic AV fistula
Venous
Venous hum
LOCATION OF HEART MURMURS
Pulmonary
Aorta
Tricuspid
Mitral
Aortic
area:
right
parasternal 2. intercostal
space
Pulmonary
area:
left
parasternal 2. intercostal
space
Tricuspid
area:
left
parasternal
4.-5.
intercostal space
Mitral area (cardiac apex):
5.-6.intercostal
space/
midclavicular line
Mezocardiyak area (second
aortic area, Erb): left
parasternal 3.-4. intercostal
space
INTENSITY OF HEART MURMURS
Graded from 1 to 6.
Grade 1: Barely audible.
Grade 2: Soft, but easily audible.
Grade 3: Moderately loud, but no accompanied
with a thrill.
Grade 4: Louder and associated with a thrill.
Grade 5: Audible with the stethescope barely on
the chest.
Grade 6: Audible with the stethoscope off the
chest.