Transcript Chapter 18 Figures

Integrating Concepts in Biology
PowerPoint Slides for Chapter 18:
Emergent Properties at the Organismal Level
by
A. Malcolm Campbell, Laurie J. Heyer, and
Chris Paradise
Title Page
Quaking Aspen Trees
Opening Figure
Autograft and Allograft Differences
Fig. 18.1
Test Skin Grafts and Pregnancy
Fig. 18.2
Extended Grafting and Pregnancy Tests
Fig. 18.3
T Cells Cause Rejection
Fig. 18.4
MHC I Present Self Peptides
Fig. 18.5
MHC I & Peptides Cause Rejection
Fig. 18.6
T Cell Receptors Bind Presented Self
Fig. 18.7
Table 18.1
Table 18.1
Maternal and Fetal Cell Interface
Fig. 18.8
MHC IG Present on Fetal Cells
Fig. 18.9
MHC IG Responsible for Tolerance
Fig. 18.10
Interleukin-10 Suppresses T Cells
Fig. 18.11
MHC IG Allele & Gene Structure
Fig. 18.12
MHC IG Allele & Gene Structure
Fig. 18.13
MHC IG Allele & Gene Structure
Fig. 18.14
Table 18.2 Distribution of genetic markers in four representative fungal samples
from a forest in Michigan. Each numbered sample was cultured to isolate the four
haploid mating types (a, b, c, and d). RFLP and RAPD numbers are arbitrary
designations for particular genetic loci, numbers in RFLP rows are arbitrary
designations for alleles, and + or 0 indicates a RAPD fragment was present or
absent, respectively.
Table 18.2
Table 18.3 Estimates of growth and size of large A. gallica individual fungus.
Table 18.3
MHC IG Allele & Gene Structure
Fig. 18.15
MHC IG Allele & Gene Structure
Fig. 18.16
Table 18.4 Genetic analysis of quaking aspen stand in Utah. Sample size was 209.
Table 18.4
MHC IG Allele & Gene Structure
Fig. 18.17
MHC IG Allele & Gene Structure
Fig. 18.18
MHC IG Allele & Gene Structure
Fig. 18.19
MHC IG Allele & Gene Structure
Fig. 18.20
Table 18.5 A sampling of expressions in animals studied by Charles Darwin, to
which he ascribed various emotions, some of which are shown.
Table 18.5
MHC IG Allele & Gene Structure
Fig. 18.21
MHC IG Allele & Gene Structure
Fig. 18.22
Table 18.6 Results from experiments investigating adrenalin in cats.
Table 18.6
MHC IG Allele & Gene Structure
Fig. 18.23
Aging Is Universal
Fig. 18.24
Interactions Affect Fecundity and Mortality
Fig. 18.25
Humans Exhibit Programmed Senescence
Fig. 18.26
Testing Disposable Soma Theory
Fig. 18.27
Table 18.7
Comparison of island and mainland opossum traits. A p-value of
<0.05 indicates significant differences in the two populations.
Table 18.7
Ethics for End of Life Issues
Fig. ELSI 18.1
Table 18.8
Comparison of physical traits for Drosophila fruit flies subjected to
high or low adult mortality. Total refers to the average value for each
trait using the total population indicated by n.
Table 18.8
Disposable Soma Theory in Plants
Fig. 18.28
Biological Source of Senescence
Fig. 18.29
Aging and Senescence in Bacteria
newest poles
Fig. 18.30a
Aging and Senescence in Bacteria
b)
Fig. 18.30b & c
c)
Table 18.9
Gene activity for E. coli cells placed in fresh media after a couple
hours (young cells) or 16 days (old cells) of stationary phase
existence.
Table 18.9
Oxidative Damage and Aging
a)
b)
Fig. 18.31
c)
Table 18.10
Proposed molecular events that can contribute to physical aging
and senescence
Table 18.10