Transcript Chapter 20
CHAPTER 20
Cholinergic Drugs
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Cholinergic Drugs
Drugs that stimulate the parasympathetic
nervous system (PSNS)
The PSNS is the opposing system to
the SNS
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Cholinergic Drugs (cont’d)
Also known as cholinergic agonists or
parasympathomimetics
Mimic effects of the PSNS neurotransmitter
acetylcholine (ACh)
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Cholinergic Receptors
Two types, determined by:
Location
Action once stimulated
Nicotinic receptors
Muscarinic receptors
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Nicotinic Receptors
Located in the ganglia of both the
PSNS and SNS
Named nicotinic because they can be
stimulated by the alkaloid nicotine
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Muscarinic Receptors
Located postsynaptically in the effector
organs of the PSNS
Smooth muscle
Cardiac muscle
Glands
Named muscarinic because they can be
stimulated by the alkaloid muscarine
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Mechanism of Action
Direct-acting cholinergic agonists
Bind to cholinergic receptors, activating them
Indirect-acting cholinergic agonists
Inhibit the enzyme acetylcholinesterase, which
breaks down ACh
Results in more ACh available at the receptors
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Indirect-Acting
(Cholinesterase Inhibitors)
Reversible
Bind to cholinesterase for a period of
minutes to hours
Irreversible
Bind to cholinesterase and form a permanent
covalent bond
The body must make new cholinesterase to break
these bonds
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Drug Effects
Effects seen when PSNS is stimulated
The PSNS is the “rest and digest” system
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Drug Effects (cont’d)
SLUDGE acronym
Salivation
Lacrimation
Urinary incontinence
Diarrhea
Gastrointestinal cramps
Emesis
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Drug Effects (cont’d)
Stimulate intestine and bladder
Increased gastric secretions
Increased gastrointestinal motility
Increased urinary frequency
Stimulate pupils
Constriction (miosis)
Reduced intraocular pressure
Increased salivation and sweating
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Drug Effects (cont’d)
Cardiovascular effects
Decreased heart rate
Vasodilation
Respiratory effects
Bronchial constriction, narrowed airways
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Drug Effects (cont’d)
At recommended doses, cholinergics
primarily affect muscarinic receptors
At high doses, cholinergics stimulate nicotinic
receptors
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Drug Effects (cont’d)
Desired effects are from muscarinic receptor
stimulation
Many undesirable effects are caused by
stimulation of nicotinic receptors
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Indications
Direct-acting drugs
Reduce intraocular pressure
Useful for glaucoma and intraocular surgery
• Acetylcholine
• Carbachol
• Pilocarpine
Topical application because of poor oral
absorption
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Indications (cont’d)
Direct-acting drug—bethanechol
Increases tone and motility of bladder and GI tract
Relaxes sphincters in bladder and GI tract,
allowing them to empty
Helpful for postsurgical atony of the bladder
and GI tract
Oral dose or SC injection
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Indications (cont’d)
Indirect-acting drugs
Cause skeletal muscle contractions
Used for diagnosis and treatment of
myasthenia gravis
Used to reverse neuromuscular blocking drugs
Used to reverse anticholinergic poisoning
(antidote)
• Examples: physostigmine, pyridostigmine
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Indications (cont’d)
Indirect-acting drug—cevimeline (Evoxac)
Used to treat xerostomia (dry mouth) resulting
from Sjögren’s syndrome
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Indications (cont’d)
Indirect-acting anticholinesterase drugs
Used for treatment of mild to moderate
Alzheimer’s disease
• donepezil (Aricept)
• tacrine (Cognex)
• galantamine (Razadyne)
• rivastigmine (Exelon)
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Indications (cont’d)
memantine (Namenda)
Not a cholinergic drug
Also used in the treatment of Alzheimer’s disease
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Adverse Effects
Adverse effects are a result of overstimulation of
the PSNS
Cardiovascular
CNS
Bradycardia, hypotension, conduction abnormalities
(AV block and cardiac arrest)
Headache, dizziness, convulsions
Gastrointestinal
Abdominal cramps, increased secretions, nausea,
vomiting
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Adverse Effects (cont’d)
Respiratory
Increased bronchial secretions, bronchospasms
Other
Lacrimation, sweating, salivation, loss of binocular
accommodation, miosis
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Interactions
Anticholinergics, antihistamines,
sympathomimetics
Antagonize cholinergic drugs, resulting in
decreased responses
Other cholinergic drugs
Additive effects
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Herbal Products: Gingko
Common uses
Organic brain syndrome
Vertigo
Tinnitus
May cause GI upset, headache, bleeding
Potential interactions
Aspirin
NSAIDs
Anticoagulants
Anticonvulsants
Others
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Nursing Implications
Note that these drugs will stimulate the PSNS
and mimic the action of ACh
Assess for allergies, presence of GI or GU
obstructions, asthma, peptic ulcer disease,
or coronary artery disease
Perform baseline assessment of vital signs
and systems overview
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Nursing Implications (cont’d)
Medications should be taken as ordered
and not abruptly stopped
Doses should be spread evenly apart
to optimize the effects of the medication
Overdosing can cause life-threatening
problems. Patients should not adjust dosages
unless directed by their physician
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Nursing Implications (cont’d)
Encourage patients with myasthenia gravis to
take medication 30 minutes before eating to
help improve chewing and swallowing
When cholinergic drugs are prescribed for
Alzheimer’s disease, be honest with
caregivers and patients that the drugs are for
management of symptoms (not a cure)
Therapeutic effects of anti-Alzheimer’s drugs
may not occur for up to 6 weeks
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Nursing Implications (cont’d)
Atropine is the antidote for cholinergics, and it
should be available in the patient’s room for
immediate use if needed
Patients should notify their physician if they
experience muscle weakness, abdominal
cramps, diarrhea, or difficulty breathing
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Nursing Implications (cont’d)
Monitor for therapeutic effects
Alleviated signs and symptoms of myasthenia
gravis
In postoperative patients with decreased GI
peristalsis, monitor for:
• Increased bowel sounds
• Passage of flatus
• Occurrence of bowel movements
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Nursing Implications (cont’d)
Monitor for therapeutic effects
In patients with urinary retention/hypotonic
bladder, urination should occur within 60 minutes
of bethanechol administration
Also monitor for adverse effects
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